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80篇 您的检索式:作者名="CALVISI DF"
    题名 作者 年代 出处 被引量
1Mechanistic and prognostic significance of aherrant methylation in the molecular pathogenesis of hmnan hepatocellular earcinoma 显示文摘Calvisi DF Ladu S Gorden A 2007J Clin Invest2007,117,9:1
2Activation oil v-Myb a- vian myeloblastosis viral oncogene homolog-like2 (MYBL2)- LIN9 complex contributes to human hepatoearcinogenesis and identifies a subset of hepatocellular carcinoma with mutant p53显示文摘Calvisi DF Simile MM Ladu S 2011Hepatology2011,53,4:1
3Altered methionine metabolism and global DNA methylation in liver cancer: Relationship with genomic instability and prognosis 显示文摘Calvisi DF Simile MM Ladu S 2007lnt J Cancer2007,121,11:1
4Ubiquitous activation of Ras and Jak/Stat pathways in human HCC 显示文摘Calvisi DF Ladu S Gorden A 2006Gastroenterology2006,130,4:1
5Forkhead box M1B is a determi- nant of rat susceptibility to hepatocarcinogenesis and sustains ERK activity in human HCC 显示文摘Calvisi DF Pinna F Ladu S 2009Gut2009,58,5:1
6Activation of beta-catenin provides proliferative and invasive advantages in c-myc/TGF-alpha hepatocarcinogenesis promoted by phenobarbital显示文摘Calvisi DF Ladu S Factor VM 2004Carcinogenesis2004,25,6:1
7Reversal of liver fibrosis in arylhydrocarbon receptor null mice by dietary vitamin A depletion显示文摘 CALVISI DF ELIZONDO G 2004Hepatology2004,39,1:1
8Thymoma viral oneogene homolog/mammalian target of rapamycin activation induces a mod- ule of metabolic changes contributing to growth in insulin - induced hepatocarcinogenesis显示文摘Evert M Calvisi DF Evert K 2012Hepatology2012,55,5:1
9Forkhead box M1B is a determi- nant of rat susceptibility to hepatocarcinogenesis and sustains ERK activity in human HCC 显示文摘Calvisi DF Pinna F Ladu S 2009Gut2009,55,5:1
10Selenoprotein deficiency and high levels of selenium compounds can effectively inhibit hepatocarcinogenesis in transgenic mice 显示文摘Novoselov SV Calvisi DF Labunskyy VM 2005Oncogene2005,24,54:1
11Forkhead box M1B is a de- terminant of rat susceptibility to hepatocarcinogenesis and sustains ERK activity in human HCC 显示文摘Calvisi DF Pinna F Ladu S 2009Gut2009,58,:1
12Activation of beta-catenin during hepatocarcinogenesis in transgenic mouse models: relationship to phenotype and tumor grade 显示文摘Calvisi DF Factor VM Loi R 2001Cancer Res2001,61,:1
13Mechanistic and prognostic significance of aberrant methylation in the mo- lecular pathogenesis of human hepatoeellular carcinmna 显示文摘Calvisi DF Ladu S Gorden A 2007JClinInvest2007,117,9:1
14Altered methionine metabolism and global DNA methylation in liver cancer:relationship with genomic instability and prognosis显示文摘Calvisi DF Simile MM Ladu S 2007Int J Cancer2007,121,11:1
15De novo lipogenesis is re- quired for insulin-mediated hepatocarcinogenesis and contributes to human liver cancer prognosis 显示文摘Calvisi DF Evert K Murtas VD 2011Z Gastroenterol2011,49,:1
16Oncogenic and tumor suppressive roles of polo-like kinases in human hepatocellular carcinoma显示文摘Pellegrino R Calvisi DF Ladu S 0,,03:1
17SCD1 Expression is dispensable for hepatocarcinogenesis induced by AKT and Ras oncogenes in mice显示文摘Li L Wang C Calvisi DF 2013PloS One2013,8,9:1
18Cholangiocarcinomas can originate from hepatocytes in mice 显示文摘Fan B Malato Y Calvisi DF 2012J Clin Invest2012,122,8:1
19Increased lipogenesis,induced by AKT-mTORC1-RPS6 signaling,promotes development of human hepatocellular carcinoma显示文摘Calvisi DF Wang C Ho C 2011Gastroenterology2011,140,3:1
20Activation of beta- catenin provides proliferative and invasive advantages in c-myc/TGF-alpha hepatocarcinogenesis promoted by phenobarbital 显示文摘Calvisi DF Ladu S Factor VM 2004Carcinogenesis2004,25,6:1
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