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9篇 您的检索式：作者名="Biola"

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		题名	作者	年代	出处	被引量
	1	Induction of apoptosis in lymphocytes byglucocorticoids:between physiology and pharmacology显示文摘	Pallardy M,Biola A	1998	CR Seances Soc Biol1998，192，6：	1
	2	Interleukin - 2 inhibits glucocorticoid receptor transcriptional activity through a mechanism involving STATS ( signal transducer and activator of transcription 5 ) but not AP - 1 显示文摘	Biola A Lefebvre P Perrin - Wolff M	2001	Mol Endocrinol2001，15，7：	1
	3	Modeofactionof glucocorticoids显示文摘	Biola A Pallardy M	2000	Preses Med2000，29，4：	1
	4	Time limits and topic assignment for essay tests显示文摘	Biola H.R		0，，：	1
	5	Interleukin-2 inhibits glucocorticoid receptor transcriptional activity through a mechanism involving STAT5(signal transducer and activator of transcription 5)but not Ap-1显示文摘	Biola A Lefebvre P Perrin WM	2001	Mol Endocrinol2001，15，7：	1
	6	Induction of apoptosis in lymphocytes by glucocorticoids: between physiology and pharmacology显示文摘	Pallardy M Biola A	1998	C R Seances Soc Biol Fil1998，192，6：	1
	7	Interleukin-2 inhibits glucocorticoid receptor transcriptional activity through a mechanism involving STAT5 (signal transducer and activator of transcription 5) but not AP-1显示文摘	Armelle Biola Philippe Lefebvre Mallory PW		0，，07：	1
	8	Mode of action of glucocorticoids显示文摘	Biola A Pallardy M	2000	Preses Med2000，29，4：	1
	9	Lineage-Specific Genome Architecture Links Enhancers and Non-coding Disease Variants to Target Gene Promoters显示文摘Long-range interactions between regulatory elements and gene promoters play key roles in transcriptional regulation.The vast majority of interactions are uncharted,constituting a major missing link in understanding genome control.Here,we use promoter capture Hi-C to identify interacting regions of 31,253 promoters in 17 human primary hematopoietic cell types.We show that promoter interactions are highly cell type specific and enriched for links between active promoters and epigenetically marked enhancers.Promoter interactomes reflect lineage relationships of the hematopoietic tree,consistent with dynamic remodeling of nuclear architecture during differentiation.Interacting regions are enriched in genetic variants linked with altered expression of genes they contact,highlighting their functional role.We exploit this rich resource to connect non-coding disease variants to putative target promoters,prioritizing thousands of disease-candidate genes and implicating disease pathways.Our results demonstrate the power of primary cell promoter interactomes to reveal insights into genomic regulatory mechanisms underlying common diseases.	Biola M Javierre	2024	四川生理科学杂志2024，46，4：	0

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