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| 1 | Update on Anti-Saccharomyces cerevisiae antibodies, anti-nuclear associated anti-neutrophil antibodies and antibodies to exocrine pancreas detected by indirect immunofluorescence as biomarkers in chronic inflammatory bowel diseases: Results of a multicent显示文摘AIM: Anti-Saccharomyces cerevisiae antibodies (ASCA), anti-nuclear associated anti-neutrophil antibodies (NANA) and antibodies to exocrine pancreas (PAB), are serological tools for discriminating Crohn’s disease (CrD) and ulcerative colitis (UC). Like CrD, coeliac disease (CoD) is an inflammatory bowel disease (IBD) associated with (auto) antibodies. Performing a multicenter study we primarily aimed to determine the performance of ASCA, NANA and PAB tests for IBD diagnosis in children and adults, and secondarily to evaluate the prevalence of these markers in CoD. METHODS: Sera of 109 patients with CrD, 78 with UC, 45 with CoD and 50 healthy blood donors were retrospectively included. ASCA, NANA and PAB were detected by indirect immunofluorescence (IIF). RESULTS: ASCA+/NANA- profile displayed a positive predictive value of 94.2% for CrD. Detection of ASCA was correlated with a more severe clinical profile of CrD and treatment of the disease did not influence their serum levels. ASCA positivity was found in 37.9% of active CoD.PAB were found in 36.7% CrD and 13.3% CoD patients and were not correlated with clinical features of CrD, except with an early onset of the disease. Fifteen CrD patients were ASCA negative and PAB positive. CONCLUSION: ASCA and PAB detected by IIF are specific markers for CrD although their presence does not rule out a possible active CoD. The combination of ASCA, NANA and PAB tests improves the sensitivity of immunological markers for CrD. Repeating ASCA, NANA, and PAB testing during the course of CrD has no clinical value. | S Desplat-Jégo C Johanet A Escande J Goetz N Fabien N Olsson E Ballot J Sarles JJ Baudon JC Grimaud M Veyrac P Chamouard RL Humbel | 2007 | World Journal of Gastroenterology2007，13，16： | 24 |
| 2 | Structural style and evolution of a Late Triassic rift basin in the Central High Atlas,Morocco:controls on sediment deposition显示文摘 | Baudon C Fabuel-Perez I Redfern J | | 0，，： | 1 |
| 3 | 关于保留北京鸭种蛋的胶护膜并对其采用整机孵化方式的讨论显示文摘人工孵化不过是一种模仿种蛋在自然环境中孵化过程的繁殖手段。以自然孵化为基准,人工孵化尽可能接近地效仿是关键。环境中孵化过程的繁殖手段。以自然孵化为基准。 | 景若曦(译) 张配配(校) 罗静如(制图表) paul pouvreau stéphane baudon | 2016 | 国外畜牧学(猪与禽)2016，36，7： | 1 |
| 4 | Improved fluorescence excitation-emission matrix regional integration to quantify spectra for fluorescent dissolved organic matter显示文摘 | Zhou J Wang J-J Baudon A | 2013 | Journal of Environmental Quality2013，42，3： | 1 |
| 5 | Infantile diarrhea is still deadly even in developed nations 显示文摘 | Baudon JJ | 2012 | Soins Pediatr Pueric2012，，268： | 1 |
| 6 | Contribution a letude des Buprestides du Laes 显示文摘 | BAUDON A | 1962 | Bulletin de la Socete Royale des Sciences Naturelles du Laos1962，2，： | 1 |
| 7 | Infantile diarrhea is still deadly even in developed na- tions显示文摘 | Baudon JJ | 2012 | Soins Pediatr Pueric2012，268，1： | 1 |
| 8 | Gastroesophageal reflux in infants:myths and realities显示文摘 | Baudon JJ | 2009 | Arch Pediatr2009，16，5： | 1 |
| 9 | Measles elimination efforts and 2008-2011 outbrea,france显示文摘 | Antona D Lévy-Bruhl D Baudon C | | 0，，03： | 1 |
| 10 | The kinematics of reactivation of normal faults using high resolution throw mapping 显示文摘 | Catherine Baudon Joe Cartwright | 2008 | Journal of Structural Geology2008，30，8： | 1 |
| 11 | 3D seismic characterisation of an array of blind normal faults in the Levant Basin, Eastern Mediterranean显示文摘 | Catherine Baudon Joe A Cartwright | 2007 | Journal of Structural Geology2007，30，6： | 1 |
| 12 | Preliminary Data on Legionella Detection in Water Distribution Systems in Cameroon显示文摘 | Marguerite Ndayo Wouafo Ariane Nzouankeu Caroline Kemadjou Guy Joseph Ejenguele Dominique Baudon | 2011 | Journal of Life Sciences2011，5，10： | 0 |
| 13 | 儿童乳糜泻研究结果显示文摘Aims-Determine the proportion of infants whose celiac disease (CD) was confirmed in childhood and evaluate their prognosis in adulthood. Patients and methods-The diagnosis of CD was established between 1971 and 1982 in 84 infants based on intestinal biopsy data; a gluten-free diet was prescribed and the cohort followed prospectively. Results-Thirty-six infants were followed less than 5 years. A second biopsy was performed in 25. Mucosa had healed in 13 and remained atrophic in 12. Three children developed partial villous atrophy between 6 and 12 years of age in spite of the gluten-free diet. Forty-five patients underwent a gluten challenge between 5 and 10 years of age: in 41 histological lesions relapsed, in two mucosa remained normal and clinical and immunological relapse developed in two. Among those 45 patients, 18 were examined after 18 years follow-up: the exclusion diet was resumed in four, overt clinical relapse developed in four and four experienced intermittent gastrointestinal disorders. All biopsies performed during a period of normal diet showed villous atrophy (except in one patient) without correlation with clinical symptoms. Conclusion-The diagnosis of celiac disease in infants was confirmed in nearly all cases in childhood. When they reached adulthood, these patients had few symptoms but their histological lesions persisted. These data are in favor of a lifelong exclusion diet. | Baudon J.-J. Chevalier J. Boccon-GibodL. 张欣 | 2006 | 世界核心医学期刊文摘(胃肠病学分册)2006，2，5： | 0 |
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