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| 1 | Quantum network communication: a discrete-time quantum-walk approach显示文摘We study the problem of quantum multi-unicast communication over the butterfly network in a quantum-walk architecture, where multiple arbitrary single-qubit states are transmitted simultaneously between multiple source-sink pairs. Here, by introducing quantum walks, we demonstrate a quantum multiunicast communication scheme over the butterfly network and the inverted crown network, respectively,where the arbitrary single-qubit states can be efficiently transferred with both the probability and the state fidelity one. The presented result concerns only the butterfly network and the inverted crown network, but our techniques can be applied to a more general graph. It paves a way to combine quantum computation and quantum network communication. | Yuguang YANG Jiajie YANG Yihua ZHOU Weimin SHI Xiubo CHEN Jian LI Huijuan ZUO | 2018 | Science China(Information Sciences)2018，61，4： | 5 |
| 2 | p53 deficiency-induced Smad1 upregulation suppresses tumorigenesis and causes chemoresistance in colorectal cancers显示文摘The DNA damage response helps to maintain genome integrity,suppress tumorigenesis,and mediate the effects of radiotherapy and chemotherapy.Our previous studies have shown that Smad1 is upregulated and activated by Atm in DNA damage response,which can further bind to p53 and promote p53 stabilization.Herewe report another aspect of the interplay between p53 and Smad1.Comparison of rectal tumor against paired paraneoplastic specimens and analysis of>500 colorectal tumors revealed that Smad1 was upregulated in tumor samples,which was attributable to p53 defects.Using MEFs as a model,we found that knockdown of the elevated Smad1 in p532/2 MEFs promoted cell proliferation,E1A/Ras-induced cell transformation,and tumorigenesis.Mechanistic studies suggest that elevated Smad1 and momentary activation inhibit cell proliferation by upregulating p57Kip2 and enhancing Atm–Chk2 activation.Surprisingly,elevated Smad1 appears to have a negative effect on chemotherapy,as colorectal tumors,primary cancer cells,and cell lines with Smad1 knockdown all showed an increase in chemosensitivity,which could be attributable to elevated p57Kip2.These findings underscore the significance of Smad1–p53 interaction in tumor suppression and reveal an unexpected role for Smad1 in chemoresistance of colorectal cancers. | Xinsen Ruan Qiao Zuo Hao Jia Jenny Chau Jinlin Lin Junping Ao Xuechun Xia Huijuan Liu Samy LHabib Chuangang Fu Baojie Li | 2015 | Journal of Molecular Cell Biology2015，7，2： | 2 |
| 3 | Packings and coverings of λKv into k-circuits with one chord显示文摘 | Kang Qingde Zhang Yanfang Zuo Huijuan | 2004 | Discrete Math2004，279，： | 1 |
| 4 | Packings and coverings of λKv into k-circuits with one chord显示文摘 | Kang Qingde Zhang Yanfang Zuo Huijuan | 2004 | Discrete Math2004，279，： | 1 |
| 5 | Decompositions ofλKv into k-circuits with one chord显示文摘 | Kang Qingde Zuo Huijuan Zhang Yanfang | 2004 | Australasian Journal ofCombinatorics2004，30，： | 1 |
| 6 | Overexpression of astrocyte-elevated gene-1 is closely correlated with poor prognosis in human non–small cell lung cancer and mediates its metastasis through up-regulation of matrix metalloproteinase-9 expression显示文摘 | Shijun Sun Zunfu Ke Fen Wang Shuhua Li Wenfang Chen Anjia Han Zuo Wang Huijuan Shi Lian-tang Wang Xiaodong Chen | 2012 | Human Pathology2012，，7： | 1 |
| 7 | A dog carrying mutations in AVP-NPII exhibits key features of central diabetes insipidus显示文摘Central diabetes insipidus(CDI)is a rare disease characterized by the excretion of copious amounts of diluted urine(polyuria),excess water intake(polydipsia),and a rise in serum sodium concentration(hypernatremia)(Christ-Crain et al.,2019).The neuropeptide arginine-vasopressin(AVP)is synthesized as a preprohormone along with its carrier protein neurophysin II(NPII)in hypothalamic supraoptic(SON)and paraventricular(PVN)magnocellular neurons,stored in the posterior pituitary,and secreted into the circulation. | Huijuan Xu Haiyang Hao Shirui Wang Xueru Liu Wen Lyu Zhentao Zuo Yan Zhuo Jidong Mi Yong Q.Zhang Rui Tian Huijuan Zhu | 2023 | Journal of Genetics and Genomics2023，50，4： | 0 |
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