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| 1 | Celiac disease:diagnostic criteria in progress显示文摘Until a few years ago,celiac disease(CD)was thought to be a rare food intolerance that was confined to childhood and characterized by severe malabsorption and flat intestinal mucosa.Currently,CD is regarded as an autoimmune disorder that is common in the general population(affecting 1 in 100 individuals),with possible onset at any age and with many possible presentations.The identification of CD is challenging because it can begin not only with diarrhea and weight loss but also with atypical gastrointestinal(constipation and recurrent abdominal pain)and extra-intestinal symptoms(anemia,raised transaminases,osteoporosis,recurrent miscarriages,aphthous stomatitis and associated autoimmune disorders),or it could be completely symptomless.Over the last 20 years,the diagnostic accuracy of serology for CD has progressively increased with the development of highly reliable tests,such as the detection of IgA tissue transglutaminase and antiendomysial and IgG antideamidated gliadin peptide antibodies.The routine use of antibody markers has allowed researchers to discover a very high number of‘borderline’cases,characterized by positive serology and mild intestinal lesions or normal small intestine architecture,which can be classified as potential CD.Therefore,it is evident that the‘old celiac disease’with flat mucosa is only a part of the spectrum of CD.It is possible that serology could identify CD in its early stages,before the appearance of severe intestinal damage.In cases with a positive serology but with mild or absent intestinal lesions,the detection of HLA-DQ2 and HLA-DQ8 can help reinforce or exclude the diagnosis of gluten sensitivity. | U Volta V Villanacci | 2011 | Cellular & Molecular Immunology2011，8，2： | 2 |
| 2 | The role of glial cells and apoptosis of enteric neurons in the neuropathology of intractable slow transit constipation 显示文摘 | Bassotti G Villanacci V Maurer CA | 2006 | Gut2006，55，1： | 1 |
| 3 | Can 'functional'constipation be consid- ered as a form of enteric neuro-gliopathy?显示文摘 | Bassotti G Villanacci V | 2011 | Glia2011，59，3： | 1 |
| 4 | The role of glial cells and apoptosis of enteric neurones in the neuropathology of intractable slow transit constipation显示文摘 | Bassotti G Villanacci V Maurer C A | 2006 | Gut2006，55，1： | 1 |
| 5 | The role of glial cellsand apoptosis of enteric neurones in the neuropathology of intracta-ble slow transit constipation显示文摘 | Bassotti G Villanacci V Maurer C A et a1 | 2006 | Gut2006，55，1： | 1 |
| 6 | Epidermal growth factor receptor overexpression/amplification in adenocarcinomas arising in the gastrointestinal tract 显示文摘 | Rossi E Villanacci V Danesino C | 2012 | Rev Esp Enferm Dig2012，103，12： | 1 |
| 7 | COX-2, CDX2, and CDC2 immunohistochemical assessment for dysplasia-carcinoma progression in Barrett's esophagus 显示文摘 | Villanacci V Rossi E Zambelli C | 2007 | Dig Liver Dis2007，39，4： | 1 |
| 8 | Dilated intercellular a spaces:a major morphological feature of esophagitis 显示文摘 | Ravelli AM Villanacci V Ruzzenenti N | 2006 | J Pediatr Gastroenterol Nutr2006，42，5： | 1 |
| 9 | Enteric neuropathology of the terminal ileum in patients with intractable slow-transit constipation 显示文摘 | Bassotti G Villanacci V Cathomas G | 2006 | Hum Pathol2006，37，10： | 1 |
| 10 | Colonic propulsive and postprandial motor activity in patients with ulcerative colitis in remission显示文摘 | Bassotti G Villanacci V Mazzocchi A | 2006 | Eur J Gastroenterol Hepatol2006，18，： | 1 |
| 11 | HER-2/Neu and Topoisomerase- II-a Expression and Genic Amplification in Pancreatic Adenocarcinoma显示文摘 | Baiocchi GL Villanacci V Rossi E | 2009 | Dig Dis Sci2009，54，： | 1 |
| 12 | Enteric neuropathology of the terminal ileum in pa- tients with intractable slow-transit constipation显示文摘 | BASSOTTI G VILLANACCI V CATHOMAS G | 2006 | Hum Pathol2006，37，： | 1 |
| 13 | Enteric glial cells:new players in gastrointestinal motility? 显示文摘 | Bassotti G Villanacci V Antonelti E | 2007 | Lab In- vest2007，7，7： | 1 |
| 14 | The role of glial cells and apoptosis of enteric neu- rones in the neuropathology of intractable slow transit constipation显示文摘 | BASSOTTI G VILLANACCI V MAURER C A | 2006 | Gut2006，55，： | 1 |
| 15 | Chromosomal study of enteric glial cells and neurons by fluorescence in sltu hybridization in slow transit constipation 显示文摘 | Rossi E Villanacci V Fisogni S | 2007 | Neurogastroenterol Motil2007，19，7： | 1 |
| 16 | COX-2, CDX2, and CDC2 immunohistochemical assessment for dysplasia-carcinoma progression in Barrett's esophagus 显示文摘 | Villanacci V Rossi E Zambelli C | 2007 | Dig Liver Dis2007，39，4： | 1 |
| 17 | The role of glial cells and apoptosis of enterie neurones in the neuropathology of intractable alow transit constipation显示文摘 | Bassotti G Villanacci V Maurer CA | 2006 | Gut2006，55，1： | 1 |
| 18 | Can 'functional' constipation be considered as a form of enteric neuro-gliopathy显示文摘 | Bassotti G Villanacci V | 2011 | Glia2011，59，3： | 1 |
| 19 | The role of glial cells and apoptosis of enteric neurones in the neuropathology of intractable slow transit constipation显示文摘 | Bassotti G Villanacci V Maurer CA | 2006 | Gut2006，55，： | 1 |
| 20 | The role of glial cells and apoptosis of enteric neurones in the neuropathology of intractable slow transit constipation显示文摘 | Bassotti G Villanacci V Maurer CA | 2006 | Gut2006，55，1： | 1 |
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