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26篇 您的检索式:作者名="Ueberham U"
    题名 作者 年代 出处 被引量
1Enhanced matrix degradation after withdrawal of TGF - betal triggers hepatocytes from apoptosis to proliferation and regeneration显示文摘Arendt E Ueberham U Bittner R 2005Cell Prolif2005,38,5:1
2Conditional tetracycline- regulated expression of TGF-β1 in liver of transgenic mice leads to reversible intermediary 显示文摘Ueberham E Low R Ueberham U 2003Fibrosis Hepatology2003,37,5:1
3The regulation of amyloid precursor protein metabolism by cholinergic mechanisms and neurophin receptor signal显示文摘Rossner S Ueberham U SchliebsR 1998Prog Neurobiot1998,56,5:1
4Conditional tetracycline-regulated expression of TGF-beta1 in liver of transgenic mice leads to reversible intermediary fibrosis显示文摘UEBERHAM E LOW R UEBERHAM U 2003Hepatology2003,37,5:1
5Conditional tetracycline-regulated expression of TGF-betal in liver of transgenie mice leads to reversible intermediary fibrosis显示文摘UEBERHAM E LOW R UEBERHAM U 2003Hepatology2003,37,5:1
6Different dendriteand dendritic spine alterations in basal and apical arborr ii mutanthuman amyloid precursor protein transgenic mice 显示文摘Alp6r A Ueberham U Briickner M K 2006Brain Res2006,1099,1:1
7The regulation of amylold precursor protein metabolism by cholinergic mechanisms and neurotrophin receptor signaling 显示文摘Rossner S Ueberham U Schliebs R 1998Prog Neurobiol1998,56,50:1
8The expression of cell cycle proteins in neurons and its relevance for Alzheimer's disease显示文摘Ueberham U Arendt T 2005Curr Drug Targets CNS Neurol Disord (S1568- 007X)2005,4,3:1
9Cyclin C expression is involved in the pathogenesis of Alzheimer's disease显示文摘Ueberham U Hessel A Arendt T 2003Neurobiol Aging2003,24,3:1
10The regulation of amyloid metabolism by cholinergic mechanisms and neurotrophin receptor signaling 显示文摘Rossner S Ueberham U 1998Prog Neurobiol1998,56,5:1
11The regulation of amyloid precursor protein metabolism by cholinergic mechanisms and neurophin receptor signal显示文摘Rossner S Ueberham U Schliebs R 1998Prog Neurobiol1998,56,5:1
12Conditional tetracycline - regulated expression of TGF - beta 1 in the liver of transgenic mice leads to reversible intermediary fibrosis显示文摘Ueberham E Low R Ueberham U 2003Hepatology2003,37,5:1
13Enhanced matrix degradation after withdrawal of TGF-beta1 triggers hepatocytes from apoptosis to proliferation and regeneration显示文摘Arendt E Ueberham U Bittner R 2005Cell Prolif2005,38,5:1
14Conditional tetracycline-regulated expression of TGF-β1 in liver of transgenic mice leads to reversible intermediary fibrosis 显示文摘Ueberham E Low R Ueberham U 2003Hepatology2003,37,5:1
15Condi tional tetracycline-regulated expression of TGF betal in liver of transgenic miceleads to reversi ble intermediary fibrosis 显示文摘Ueberham E Low R Ueberham U 2003Hepatology2003,37,5:1
16Conditional tetracycline - regulated expression of TGF- β1 in liver of transgenic mice leads to reversibleintermediary fibrosis显示文摘Ueberham E Low R Ueberham U 2003Hepatology2003,37,:1
17p75 and TrkA receptor signaling independently regulate amyloid precursor protein mRNA expression, isoform composition, and protein secretion in PC12 cells显示文摘Rossner S Ueberham U Schliebs R 1998J Neurochem1998,71,2:1
18Conditional tetracycline-regulated expression of TGF-beta1 in liver of transgenic mice leads to reversible intermediary fibrosis显示文摘Ueberham E Low R Ueberham U 2003Hepatology2003,37,5:1
19Cyclin C expression is involved in the pathogenesis of Alzheimer's disease显示文摘Ueberham U Hessel A Arendt T 2003Neurebiol Aging2003,24,3:1
20Conditional tetracycline-regulated expression of TGF-beta1 in liver of transgenic mice leads to reversible intermediary fibrosis显示文摘UEBERHAM E LOW R UEBERHAM U 2003Hepatology2003,37,5:1
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