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| 1 | Talin and kindlin: the one-two punch in integrin activation显示文摘抽象合适的房间房间和房间矩阵接触由 integrin 粘附受体调停了为开发,有免疫力的反应, hemostasis 并且创伤愈合是重要的。Integrins 通行证 trans 膜为细胞外的 ligands 和细胞内部的发信号分子通过他们的调整亲密关系双向地发信号。由 integrins 的如此的双向发信号被经常被连接在之中的 conformational 变化启用细胞外, transmembrane 和细胞质的领域。这里,我们考察在有 talin 类脂化合物和 kindlin 类脂化合物相互作用的合作, talin-integrin 和 kindlin-integrin 相互作用怎么调整 integrin 亲密关系并且在这些区域的进步怎么帮助我们理解 integrin 相关的疾病。 | Feng Ye Adam K. Snider Mark H. Ginsberg | 2014 | Frontiers of Medicine2014，8，1： | 7 |
| 2 | 残余应力导致三层聚烯烃管道防腐层剥离与开裂显示文摘这是美国运输部发起的研究项目,一个管道防腐层专家团队参与了本研究项目,评价了三层聚烯烃管道外防腐涂层(3LPO)的完整性。研究表明,三层聚烯烃管道外防腐涂层(3LPO)存在剥离和面层开裂两大完整性问题。过去几年里,据文献报道,三层聚烯烃管道外防腐涂层(3LPO)的熔结环氧粉末(FBE)底漆与钢管界面上发生多起防腐层剥离事故,以及聚丙烯(PP)面层发生开裂事故。这些防腐层事故引起人们对使用三层聚烯烃管道外防腐涂层(3LPO)的关注。一般来讲,三层聚烯烃管道外防腐涂层(3LPO)采用比较厚的聚烯烃面层增强防腐层抗机械损伤和防止水渗透的能力。但是,聚烯烃的热膨胀系数比钢材高得多,结果在防腐层系统里产生比较高的残余热应力。因为残余应力高,造成防腐层剥离,尤其在管端截短防腐层和任何防腐层的边上,因为这些是高应力集中部位。特别是假如钢管表面预处理不当,就无法保证防腐层持久达到很强的粘合强度。如果熔结环氧粉末(FBE)底漆配方选择不当,发生热氧化降解,也导致防腐层过早失效。如果使用温度很高,聚丙烯也会因为热氧化降解而变脆。在残余应力下,这样脆性的聚丙烯面层就会开裂。本文分析了三层聚烯烃管道外防腐涂层(3LPO)中的残余应力,并且探讨了残余应力对三层聚烯烃管道外防腐涂层(3LPO)剥离和聚丙烯(PP)面层开裂机理的影响。 | B.T.A.Cheng S Hung-jue J Han D Wong A Kehr M Mallozzi F Aguirre H Pham W Snider 王向农(译) | 2011 | 防腐保温技术2011，19，3： | 3 |
| 3 | Three-dimensional simulation of liquid injection,film formation and transport,in fluidized beds显示文摘Liquid injection,and film formation and transport in dense-phase gas-solids fluidized beds are numerically simulated in three dimensions using a collisional exchange model that is based on the mechanism that collisions cause transfer of liquid mass,momentum,and energy between particles. In the model,each of the particles is represented by a solid core and a liquid film surrounding the core. The model is incorporated in the framework of the commercial code Barracuda? developed by CPFD Software. The commercial software is an advanced CFD-based computational tool where the particles are treated as discrete entities,calculated by the MP-PIC method,and tracked using the Lagrangian method. Details of the collisional liquid transfer model have been previously presented in O'Rourke,Zhao,and Snider (2009); this paper presents new capabilities and proof-testing of the collision model and a new method to better quantify the penetration length. Example calculations of a fluidized bed without liquid injection show the expected effect of collisions on the reduction of granular temperature (fluctuational kinetic energy) of the bed. When applied to liquid injection into a dense-phase fluidized bed under different conditions,the model predicts liquid penetration lengths comparable to the experiments. In addition,the simulation reveals for the first time the dynamic mixing of the liquid droplets with the bed particles and the transient distribution of the droplets inside the bed. | Paul Zhao Peter J. O'Rourke Dale Snider | 2009 | Particuology2009，7，5： | 2 |
| 4 | 飞机结构件加工的可行性技术分析显示文摘有关刀具和技术的非传统性选择究竟能达到什么样的程度?对此曾经做过一项研究试验,通过这项试验,可以使一台加工中心更有效地铣削加工复杂的零件。 | Jay Snider Ⅱ Richard Guiler | 2007 | 现代制造2007，，3： | 2 |
| 5 | ^(99m)TC-Methylene diphosphonate uptake at injury site correlates with osteoblast differentiation and mineralization during bone healing in mice显示文摘99m Tc-Methylene diphosphonate(99m Tc-MDP) is widely used in clinical settings to detect bone abnormalities.However, the mechanism of99 m Tc-MDP uptake in bone is not well elucidated. In this study, we utilized a mouse tibia injury model, single-photon emission computed tomography(gamma scintigraphy or SPECT),ex vivo micro-computed tomography, and histology to monitor99 m Tc-MDP uptake in injury sites during skeletal healing. In an ex vivo culture system, calvarial cells were differentiated into osteoblasts with osteogenic medium, pulsed with99 m Tc-MDP at different time points, and quantitated for99 m Tc-MDP uptake with a gamma counter. We demonstrated that99 m Tc-MDP uptake in the injury sites corresponded to osteoblast generation in those sites throughout the healing process. The99 m Tc-MDP uptake within the injury sites peaked on day 7 post-injury, while the injury sites were occupied by mature osteoblasts also starting from day 7.99 m Tc-MDP uptake started to decrease 14 days post-surgery, when we observed the highest level of bony tissue in the injury sites. We also found that99 m Tc-MDP uptake was associated with osteoblast maturation and mineralization in vitro. This study provides direct and biological evidence for99 m Tc-MDP uptake in osteoblasts during bone healing in vivo and in vitro. | Zhendong A Zhong Anderson Peck Shihong Li Jeff Van Oss John Snider Casey J Droscha Tingtung A Chang Bart O Williams | 2015 | Bone Research2015，3，2： | 2 |
| 6 | Comparison of modified Thrombelastograph and Plateletworks whole blood assays to optical p latelet aggregation for monitoring reversal of clop idogrel inhibition in elective surgery patients 显示文摘 | Craft RM Chavez JJ Snider CC | 2005 | J Lab Clin Med2005，145，6： | 1 |
| 7 | Koch Global: epidemiology of tuberculosis:Morbidity and mortality of a worldwide epidemic显示文摘 | DE Snider A | 1995 | JAMA1995，273，： | 1 |
| 8 | Transgenic expression of granulocyte-macrophage colonystimulating factor induces the differentiation and activation of a novel dendritic cell population in the lung显示文摘 | Wang J Snider DP Hewlett BR | 2000 | Blood2000，95，7： | 1 |
| 9 | Reliability of Tuffier's line as an anatomic landmark 显示文摘 | Snider KT Kribs JW Snider EJ | 2008 | Spine2008，33，6： | 1 |
| 10 | Procalcitonin assay in sys- temic inflammation, infection, and sepsis: clinical utility and limitations显示文摘 | Becker K L Snider R Nylen E S | 2008 | Crit Care Med2008，36，3： | 1 |
| 11 | Procalcitonin assay in systemic inflammation,infection, and sepsis: clinical utility and limitations 显示文摘 | Becker KL Snider R Nylen ES | 2008 | Crit Care Med2008，36，： | 1 |
| 12 | Transplantation of embryonic stem cells overexpressing Bcl-2 promotes functional recovery after transient cerebral ischemia显示文摘 | Wei L Cui L Snider BJ | | 0，，1： | 1 |
| 13 | Anemia and hematoma volume in acute intracerebral hemorrhag显示文摘 | Kumar M A Rost N S Snider R W | 2009 | Crit Care Med2009，37，5： | 1 |
| 14 | Procalcitonin in sepsis and systemic inflammation:a harmful biomarker and a therapeutic target显示文摘 | Becker KL Snider R Nylen ES | 2010 | Br J Pharmacol2010，159，2： | 1 |
| 15 | Significance of increased peripheral dopamine release for the human stress response and hypertension 显示文摘 | Snider SR Kuchel O Dopamine: an important neurohormone of the sympathoadrenal system | 1983 | Endocr Rev1983，4，3： | 1 |
| 16 | Pmcalcitonin in sepsis and sys - temic inflammation: a harmful biomarker and a therapeutic target 显示文摘 | Becker KL Snider R Nylon ES | 2010 | Br J Pharmae2010，159，2： | 1 |
| 17 | Neurotrophic molecular显示文摘 | Snider W D Johnson E M | 1989 | Ann Neuro1989，26，1： | 1 |
| 18 | Neurotrophic molecules显示文摘 | Snider W D Johnson E M | 1989 | Annal Neurol1989，26，2： | 1 |
| 19 | Mortality is increased by procalcitonin and decreased by an antiserum reactive to procalcitonin in experimental sepsis显示文摘 | Nylen ES Whang KT Snider RH Jr | 1998 | Crit Care Med1998，26，6： | 1 |
| 20 | Procalcitonin and its component peptides in systemic inflammation:immunochenmical characteriza- tion显示文摘 | Snider RH Nylen ES Becker KL | 1997 | J Investid Med1997，45，： | 1 |
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