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| 1 | Predictors of multiple injuries in individual distance runners:A retrospective study of 75,401 entrants in 4 annual races-SAFER XX显示文摘Background There are limited data on factors that predict an increased risk of multiple injuries among distance runners.The objective of this study was to determine risk factors that are predictive of individual runners with a high annual multiple injury risk(MIR).Methods A retrospective,cross-sectional study at 4 annual(2012-2015)Two Oceans 21.1 km and 56.0 km races in South Africa with 75,401 consenting race entrants.Running-related injury data were collected retrospectively through an online pre-race medical screening questionnaire.The average number of injuries for each runner every year was calculated by taking a runner's race entry history and injury history into account and categorizing entrants into 4 MIR categories(high,intermediate,low,and very low(reference)).Multiple logistic regression modeling(odds ratios)was used to determine whether the following factors were predictive of a high MIR(average>1 injury/year):demographics,training and racing,chronic-disease history(composite chronic disease score(CCDS)),and history of allergies.Results Of all entrants,9.2%reported at least 1 injury,and 0.4%of entrants were in the high MIR category;the incidence rate was 2.5 injuries per 10 runner-years(95%confidence interval(95%CI):2.4-2.7).Significant factors predictive of runners in the high MIR category were:running for>20 years:OR=2.0(95%CI:1.3-3.1;p=0.0010);a higher CCDS:OR=2.2(95%CI:2.0-2.4;p<0.0001);and a history of allergies:OR=2.8(95%CI:2.0-3.8;p<0.0001).Conclusion Runners who have been running recreationally for>20 years and those with multiple chronic diseases or a history of allergies were at higher risk of multiple running-related injuries.This high-risk group can be targeted for further study and possible injury-prevention interventions. | Sonja Swanevelder Nicola Sewry Martin Schwellnus Esme Jordaan | 2022 | Journal of Sport and Health Science2022，11，3： | 3 |
| 2 | Late onset muscular dystrophy with cerebral white matter changes to partial merosin deficiency显示文摘 | Tan E Topaloglu H Sewry C | 1997 | Neuromuscular Disorders1997，7，2： | 1 |
| 3 | Defining alpha-skeletaland alpha-cardiac actin expression in human heart andskeletal muscle explains the absence of cardiac involvementin ACTA1 nemaline myopathy显示文摘 | Ilkovski B Clement S Sewry C | | 0，，12： | 1 |
| 4 | Defining alpha-skeletaland alpha -cardiac actin expression in human heart and skeletalmuscle explains the absence of cardiac involvement in ACTA1nemaline myopathy 显示文摘 | llkovski B Clement S Sewry C | 2005 | Neuromuscul Disord2005，15，12： | 1 |
| 5 | Molecular mechanisms and phenotypic vari-ation in RYR1-related congenital myopathies显示文摘 | Zhou H Jungbluth H Sewry CA | 2007 | Brain2007，130，8： | 1 |
| 6 | Deletions in the 5' region of dystrophin and resulting phenotypes显示文摘 | Muntoni F Gobbi P Sewry C | 1994 | J Med Genet1994，31，： | 1 |
| 7 | The Emery-Dreifuss muscular dystrophy protein,emerin,is a nuclear membrance protein显示文摘 | Manial S Nguyen M Sewry CA | 1996 | Hum Mol Genet1996，5，： | 1 |
| 8 | Mutation in Emery-Dreifuss muscular dystrophy and their effects on emerin protein expression显示文摘 | Manial S Recan D Sewry CA | 1998 | Hum Mol Gene1998，7，： | 1 |
| 9 | Clinical, histological and genetic characterization of reducing body myopathy caused by mutations in FHL1 显示文摘 | Sehessl J Taratuto AL Sewry C | 2009 | Brain2009，132，： | 1 |
| 10 | Expression of dystrophin-associated glycoproteins and utrophin in carriers of Duchenne muscular dystrophy显示文摘 | Sewry C A Matsumura K Campbell K P | 1994 | Neuromuscular Disorders1994，4，5： | 1 |
| 11 | The Emery-Dreifuss muscular dystrophy protein, emerin, is a nuclear memberane protein显示文摘 | Manilal S Nguyen TM Sewry CA | 1996 | Hum Mol Genet1996，5，6： | 1 |
| 12 | Nesprins,but not sun proteins,switch isoforms at the nuclear envelope during muscle development显示文摘 | Randles KN Lam le T Sewry CA | 2010 | Dev Dyn2010，239，3： | 1 |
| 13 | Defining alpha-skeletal and alpha-cardiac actin expression in human heart and skeletal muscle explains the absence of cardiac involvement in ACTA1 nemaline myopathy显示文摘 | Ilkovski B Clement S Sewry C | | 0，，12： | 1 |
| 14 | Variable clinical phenotype in merosin-deficient congenital muscular dystrophy associated with differential immunolabelling of two fragments of the laminin alpha 2 chain显示文摘 | Sewry CA Naom I D' Alessandro M | 1997 | Neuromuscular Disorders1997，7，： | 1 |
| 15 | Mutations in the laminin alpha2-chain gene in two children with early-onset muscular dystrophy显示文摘 | Naom I D' Alessandro M Sewry CA | 2000 | Brain2000，123，： | 1 |
| 16 | Dermatomyositis,polymyositis, and coxsackie-B-virus infection 显示文摘 | Bowles NE Dubowitz V Sewry CA | 1987 | J Rheuma-tol1987，3，： | 1 |
| 17 | Molecular mechanisms and phenotypic variation in RYRI-related congenital myopathies 显示文摘 | Zhou H Jungbluth H Sewry CA Feng L Bertini E Bushby | 2007 | Brain2007，130，8： | 1 |
| 18 | Dermatomyositis polymyositis and coxsackie-B-Virus infection显示文摘 | Bowles NE Dubowitz V Sewry CA | 1987 | Lancet1987，1，： | 1 |
| 19 | Infantile onsetmyofibrillar myopathy due to recessive CRYAB mutations 显示文摘 | FORREST KM AL SARRAJ S SEWRY C | 2011 | Neuromuscul Disord2011，21，1： | 1 |
| 20 | Magnetic resonance imaging of muscle in nemaline myopathy显示文摘 | Jungbluth H Sewry CA Counsell S | 2004 | Neuromuscul Disord2004，14，： | 1 |
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