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14篇 您的检索式:作者名="Sendtner R"
    题名 作者 年代 出处 被引量
1Functional analysis od monocyte subsets insurgical sepsis显示文摘Schinkel C Sendtner R Zimmer S 1998J Trauma1998,44,5:1
2Multiple extracellular signals are required for long-term oligodendrocyte survival 显示文摘garres B Schmid R Sendtner M et al 1993Development1993,118,28:1
3Brain-derived neurotrophic factor prevents the death of motoneurons in newborn rats after nerve section显示文摘Sendtner M Holtmann B Kolbeck R 1992Nature1992,360,6406:1
4Functional analysis of monocyte subsets in surgical sepsis显示文摘Schinkel C Sendtner R Zimmer S 1998J Trauma1998,44,5:1
5Mechanisms of axonal degenera-tion in EAE lessons from CNTF and MHC I knockout mice 显示文摘Linker RA Sendtner M Gold R 2005J Neurol Sci2005,233,12:1
6Funetional analysis of monocyte subsets in suigieal sepsis显示文摘Schinkel C Sendtner R Zimmer S 1998J Trauma1998,44,5:1
7Mechanisms of axonal degeneration in EAE-lessons from CNTF and MHC I knockout mice显示文摘Linker RA Sendtner M Gold R 2005J Neurol Sci2005,233,12:1
8Mechanisms of axonal degeneration in EAE--lessons from CNTF and MHC I knockout mice显示文摘Linker RA Sendtner M Gold R 2005J Neurol Sci2005,233,12:1
9Endogenous ciliary neurotrophic factor is a lesion factor for axotomized motoneuron in adult mice 显示文摘 Gotz R Holtmann B 1997J Neurosci1997,17,18:1
10Brain derived neurotrophic factor prevents the death of motoneurons in new born rats after nerve section显示文摘SENDTNER M HOLTMANN B KOLBECK R 1992Nature1992,360,6406:1
11Mechanisms of axonal degeneration in EAE-lessons from CNTF and MHC I knockout mice显示文摘Linker RA Sendtner M Gold R 0,,1:1
12Ras p21 protein promotes survival and fiber out growth of cultured embryonic neurons显示文摘Borasio G D John J Wittinghofer A Barde Y A Sendtner M Heumann R 1989Neuron1989,,:1
13Endogenous ciliary neurotrophic factor is a lesion factor for axotomized motoneurons in adult mice显示文摘Sendtner M Gotz R Holtmann B 0,,:1
14Cryptic physiological trophic support of motoneurons by LIF revealed by double gene targeting of CNTF and LIF显示文摘Sendtner M Gotz R Holtmann B 0,,:1
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