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21篇 您的检索式：作者名="STROOPER B D"

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	题名	作者	年代	出处	被引量
1	The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeu- tics显示文摘	Karran E Mercken M Strooper B D	2011	Nat Rev Drug Discov2011，10，：	1
2	Tool support for testing concurrent Java components显示文摘	Long B Hoffman D Strooper P	2003	IEEE Trans Software Eng2003，29，6：	1
3	The disintegrin/ metalloprotease ADAM 10 is essential for Notch signalling but not for-secretase activity in fibroblasts 显示文摘	Hartmann D de Strooper B Serneels L	2002	Hum Mol Genet2002，11，4：	1
4	γ-Secretase inhibitors: still in the running as Alzheimer' s therapeutics 显示文摘	MARJAUS E STROOPER B D	2004	Drug Discovery To- day : Therapeutic Strtegies2004，1，1：	1
5	The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts显示文摘	Hartmann D de Strooper B Serneels L		0，，：	1
6	Tool support for testing concurrent Java components 显示文摘	B Long D Hoffman P Strooper	2003	IEEE Trans on Software Engineering2003，29，6：	1
7	Peroxisome-proliferatoractivated receptor gamma induces a clearance mechanism for the amyloid-beta peptide显示文摘	Camacho IE Serneels L Spittaels K Merchiers P Dominguez D De Strooper B	2004	J Neurosci2004，24，10：	1
8	Peroxisome-proliferator-activated receptor gamma induces a clearance mechanism for the amyloid-beta peptide显示文摘	Camacho IE Serneels L Spittaels K Merchiers P Dominguez D De Strooper B	2004	J Neurosci2004，24，10：	1
9	Presenilins in memory,Alzheimer's disease,and therapy显示文摘	Marjaux E Hartmann D De Strooper B		0，，：	1
10	The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for -secretase activity in fibroblasts显示文摘	Hartmann D de Strooper B Semeels L	2002	Hum Mol Genet2002，11，4：	1
11	The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for - secretase activity in fibroblasts 显示文摘	Hartmann D de Strooper B Semeels L	2002	Hum Mol Genet2002，11，4：	1
12	BACE1 and pre- senilin two unusual aspartyl proteases involved in Alzheimer's disease显示文摘	Dominguez DI Hartmann D De Strooper B	2004	Neurodegener Dis2004，1，45：	1
13	Presenilins in memory, Alzheimer's disease, and therapy显示文摘	Marjaux E Hartmann D De Strooper B	2004	Neuron2004，42，2：	1
14	The disintegrin/ metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts显示文摘	Hartmann D de Strooper B Serneels L	2002	Hum Mol Genet2002，11，21：	1
15	The disintegrin/metallo- protease ADAM 10 is essential for Notch signalling but not for alpha-secre- tase activity in fibroblasts显示文摘	Hartmann D de Strooper B Serneels L	2002	Hum Mol Genet2002，11，：	1
16	The toxic Aβ oligomer and Alzheimer's disease: an emperor in need of clothes 显示文摘	Benilova I Karran E Strooper B D	2012	Nature Neuro- science2012，15，3：	1
17	The disintegrin/metalloproteinase ADAM 10 is essential for Notch signalling but not for - secretase activity in fibroblasts 显示文摘	Hartmann D de Strooper B Serneels L	2002	Hum Mol Genet2002，11，：	1
18	The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts显示文摘	Hartmann D de Strooper B Serneels L	2002	Hum Mol Genet2002，11，：	1
19	The disintegrin/metal- loprotease ADAMlO is essential for Notch signalling but not for al- pha-secretase activity in fibroblasts显示文摘	Hartmann D de Strooper B Semeels L	2002	Hum Mol Genet2002，11，21：	1
20	Novel therapeutic strategies provide the real test for the amyloid hypothesis of Alzheimer's disease显示文摘	DOMINGUEZ D I De STROOPER B	2002	Trends in Pharmacological Sciences2002，23，7：	1

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