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78篇 您的检索式：作者名="SHAPIRO GI"

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	题名	作者	年代	出处	被引量
1	Reciprocal P16 inactivation and P16 exepression in primary lung cancer and cell lines显示文摘	Shapiro GI	1995	Cancer Res1995，55，1：	1
2	A phase I study of foretinib, a multi-targeted inhibitor of c-Met and vascular endothelial growth factor receptor 2显示文摘	Eder JP Shapiro GI Appleman LJ	2010	Clin Cancer Res2010，16，13：	1
3	Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines 显示文摘	Shapiro GI Edwards CD Kobzik L	1995	Cancer Res1995，55，：	1
4	Anticancer drug targets: cell cycle and checkpoint control 显示文摘	Shapiro GI Harper JW	1999	J Clin Invest1999，104，：	1
5	Heat shock protein 90 inhibition in lung cancer显示文摘	SHIMAMURA T SHAPIRO GI	2008	J Thorac Oncol2008，3，62：	1
6	P16INK4A partici pates in a G1 arrest checkpoint in response to DNA damag 显示文摘	Shapiro GI Edwards CD Ewen ME	1998	Mol Cell Biol1998，18，：	1
7	Cyclin-dependent kinase pathways as targets for cancer treatment 显示文摘	Shapiro GI	2006	J Clin Oncol2006，24，11：	1
8	Inactivation and p16 INK4 eexpression in primary lung cancers and cell lines显示文摘	Reciprocal Rb	1995	Cancer Res1995，55，3：	1
9	Crizotinib,a small-molecule dual inhibitor of the c-Met and ALK receptor tyrosine kinases显示文摘	Rodig SJ Shapiro GI		0，，12：	1
10	Chemotherapy-induced p53-depend- ent and-independent DNA damage responses are enhanced by poly (ADP-ribose) polymerase (PARP) inhibition in BRCA- proficient cancer eells显示文摘	Johnson N Shapiro GI	2012	Cell Cycle2012，11，3：	1
11	Multiple mechanisms of p16 inactivation in non-small cell lung cancer cell lines显示文摘	Park JE Edwars CD	1995	Cancer Res1995，55，24：	1
12	Reciprocal Rb inactivation and p16ink4 expression in primary lung cancers and cell lines显示文摘	Shapiro GI Edwards CD Kobzik L	1995	Cancer Res1995，55，8：	1
13	Cyclin-dependent kinase pathways as targets for cancer treatment显示文摘	Shapiro GI	2006	J Clin Oncol2006，24，11：	1
14	The physiology of p16( INK4A)- mediated G1 proliferative arrest 显示文摘	Shapiro GI Edwards CD Rollins BJ	2000	Cell Biochem Biophys2000，33，2：	1
15	Cyclin-dependent kinase pathways as targets for cancer treatment显示文摘	Shapiro GI	2006	Journal of Clinical Oncology2006，24，11：	1
16	Cyclin-dependent kinase pathways as targets for cancer treatment显示文摘	Shapiro GI	2006	J Clin Onco12006，24，11：	1
17	Chemotherapy-induced p53-dependent and -independent DNA damage responses are enhanced by poly (ADP ribose) polymerase (PARP) inhibition in BRCA proficient cancer cells显示文摘	Johnson N Shapiro GI	2012	Cell Cycle2012，11，3：	1
18	Cyclin-dependent kinase pathways as targets for cancer treatment显示文摘	Shapiro GI		0，，11：	1
19	A phase I clinical and pharmacokinetic(Pk)trial of the Aurora kinase(AK)inhibitor MK-0457 in Cancer patients显示文摘	Rubin EH Shapiro GI Stein MN	2006	J Clin Oncol2006，24，181：	1
20	P16INK4A participates in a G1 arrest checkpoint in response to DNA damage显示文摘	Shapiro GI Edwards CD Ewen ME	1998	Mol Cell boil1998，18，：	1

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