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84篇 您的检索式:作者名="SCALTRITI M"
    题名 作者 年代 出处 被引量
1The epidermal growth factor receptor pathway:a model for targeted therapy显示文摘Scaltriti M Baselga J 2006Clin Cancer Res2006,12,:1
2Nuclear translocation of a clusterin isoform is associated with induction of anioikis in SV40-immortalized human prostate epithelial cell显示文摘Caccamo AE Scaltriti M Caporali A 0,,:1
3Expression of p95 HER-2,a truncated form of the HER-2 receptor,and response to anti-HER-2 therapies in breast cancer显示文摘Scaltriti M Rojo F Ocana A 0,,08:1
4Clinical benefit of lapatinib-based therapy in patients with human epidermal growth factor receptor 2-positive breast tumors coexpressing the truncated p95HER-2 receptor显示文摘Scaltriti M Chandarlapaty S Prudkin L 0,,:1
5Loss of HER-2 amplification following trastuzumab-based neoadjuvant systemic therapy and survival outcomes显示文摘Mittendorf EA Wu Y Scaltriti M 2009Clin Cancer Res2009,15,23:1
6Phosphatidylinositol 3- kinase hyperactivation results in lapatinib resistance that is reversed by the mTOR/phosphatidylinositol 3-kinase inhibitor NVP-BEZ235 显示文摘Eichhorn P J Gili M Scaltriti M 2008Cancer Res2008,68,92:1
7Biosynthesis of tumorigenic HER2 C - terminal fragments by alternative initiation of translation 显示文摘Anido J Scaltriti M Bech Serra J J 2006Embo J2006,25,13:1
8Cyclin E amplification/over expression is a mechanism of trastuzumab resistance in HER2 + breast cancer patients显示文摘Scaltriti M Eichhom PJ Cortés J 0,,:1
9Intracellular clusterin induces G2-M phase arrest and cell death in PC-3 prostate cancer cells显示文摘SCALTRITI M SANTAMARIA A PACIUCCI R 2004Cancer Res2004,64,17:1
10Lapatinib, a HER2 tyrosine kinase inhibitor, induces stabilization and accumulation of HER2 and potentiates trastuzumab- dependent cell cytotoxicity 显示文摘Scaltriti M Verma C Guzman M 2009Oncogene2009,28,6:1
11NVPBEZ235,a dual PI3K/mTOR inhibitor prevents PI3K signaling and inhibits the growth of cancer cells with activating PI3K mutations显示文摘SERRRA V MARMAN B SCALTRITI M 2008Cancer Res2008,68,19:1
12Cyclin E amplification/overexpression is a mechanism of trastuzumab resistance in HER-2 + breast cancer patients显示文摘Scaltriti M Eichhom PJ Cortés J 0,,:1
13Cyclin E amplification/overexpression is a mechanism of trastuzumab resistance in HER2+ breast cancer patients显示文摘SCALTRITI M EICHHORN PJ CORT(E)S J 0,,:1
14The epidermal growth factor receptor pathway:a model for targeted therapy显示文摘Scaltriti M Baselga J 2006Clin Cancer Res2006,12,18:1
15Synergy between trastuzumab and pertuzumab for human epidermal growth factor 2 ( Her2 ) from colocalization : an in silico based mech- anism显示文摘Fuentes G Scaltriti M Baselga J 2011Breast Cancer Res2011,13,3:1
16The epidermal growth factor receptor path- way:a model for targeted therapy显示文摘Scaltriti M Baselga J 2006Clin Cancer Res2006,12,:1
17PI3K inhibition results in enhanced HER signaling and acquired ERK dependency in HER2-overexpressing breast cancer显示文摘Serra V Scaltriti M Prudkin L 2011Oncogene2011,30,22:1
18NVP- BEZ235, a dual PBK/mTOR inhibitor, prevents PI3K signaling and inhibits the growth of cancer cells with activating PI3K mutations 显示文摘SERRA V MARKMAN B SCALTRITI M 2008Cancer Res2008,68,19:1
19NVP-BEZ235,a dual PI3K/mTOR inhibitor,prevents PI3K signaling and inhibits the growth of cancer cells with activating PI3K mutations显示文摘Serra V Markman B Scaltriti M 2008Cancer Res2008,68,:1
20Phosphatidylinositol 3-kinase hyperactivation results in lapatinib resistance that is reversed by the mTOR/phosphatidylinositol 3-kinase inhibitor NVP-BEZ235显示文摘Eichhorn PJ Gili M Scaltriti M 2008Cancer Res2008,68,:1
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