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85篇 您的检索式:作者名="SAMUDIO I"
    题名 作者 年代 出处 被引量
1CXCR4 expression and biologic activity in acute myeloid leukemia are dependent on oxygen partial pressure 显示文摘Fiegl M Samudio I Clise-Dwyer K 2009Blood2009,113,7:1
2resist promotes smooth muscle cell proliferation through activation of extracelluar signal-regulated kinase 1/2 and phosphatidylinositol 3-kinase pathway显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
3Resistin promotes smooth muscle cell proliferation through activation of extracellular signal-regulated kinase phosphstidylinositol 3-kinase pathways 显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
4Estrogen regulation of cyclin D1 gene expression in ZR-75 breast cancer cells involves multiple enhancer elements显示文摘Castro-Rivera E Samudio I Safe S 0,,33:1
5Resistin promotes smooth muscle cell proliferation through activation of extracellular signal -regulated kinase 1/2 and phosphatidylinositol 3-kinase pathways 显示文摘CALABRO P SAMUDIO I WILLERSON J T 2004Circulation2004,110,:1
6Resistin promotes smooth muscle cell proliferation through activation of extracellular signal-regulated kinase 1/2 and phosphatidylinositol 3-kinase pathways显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,:1
7Resistin promotes smooth muscle cell proliferation through activation of extracellular signal- regulated kinasel/2 and phosphatidylinositol kinase path- ways显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
8Targeting the leukemia micro- environment by CXCR4 inhibition overcomes resistance to kinaseinhibitors and chemotherapy in AML显示文摘Zeng Z Shi Y X Samudio I J 2009Blood2009,113,24:1
9CXCR4 expression and biological activity in acute myeloid leukemia are dependent on oxy- gen partial pressure 显示文摘Fiegl M Samudio I Clise-Dwyer K 2009Blood2009,113,7:1
10Resistin promotes smooth muscle cell proliferation through activation of extracellular signal-regulated kinase 1/2 and phosphatidylinositol 3-kinase pathways显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,:1
11CXCR4 ex- pression and biologic activity in acute myeloid leukemia are dependent on oxygen partial pressure显示文摘Fiegl M Samudio I Clise-Dwyer K 2009Blood2009,113,7:1
12Resistin promotes smooth muscle cell proliferation through activation of extracellular signal-regulated kinase 1/2 and phosphatidylinositol 3-kinase pathways显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,:1
13Resistin promotes smooth muscle cell proliferation through activation of extra cellular signal - regulated kinase 1/2 and phosph - atidylinositol 3 - kinase pathways 显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
14Resistin promotes smooth muscle cell proliferation through activation of extracelular signal-regulated kinase 1/2 and phosphatidy linositol 3-kinase pathways 显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
15Regulation of HIF- lasignaling and chemoresistance in acute lymphocytic leuke- mia under hypoxic conditions of the bone marrow microenvi- ronment显示文摘Frolova O Samudio I Benito J 2012Cancer biology therapy2012,13,10:1
16Guggulsterones induce ap- optosis and differentiation in acute myeloid leukemia:identification of isomer-specific antileukemic activities of the pregnadienedione structure 显示文摘Samudio I Konopleva M Safe S 2005Mol Cancer Ther2005,4,12:1
17Small inhibitory RNA duplexes for Sp1 mRNA block basal and estrogen-induced gene expression and cell cycle progressionin MCF-7breast cancer cells显示文摘Abdelrahim M Samudio I Smith R 3rd Burghardt R and Safe S 2002J Bio Chem2002,277,28:1
18Resistin promotes smooth muscle cell proliferation through activation of extracellular signal regulated kinase 1/2 and phospha tidylinositol 3-kinase pathways 显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
19Resistin promotes smooth muscle cell proliferation through activation of extracellular signal-regulated kinase 1/2 and phosphatidylinositol 3-kinase pathways显示文摘Calabro P Samudio I Willerson JT 2004Circulation2004,110,21:1
20Mitochondrial uncoupling and the Warburg effect: molecular basis for the reprogramming of cancer cell metabolism 显示文摘Samudio I Fiegl M Andreeff M 2009Cancer Res2009,69,6:1
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