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16篇 您的检索式:作者名="Raygada"
    题名 作者 年代 出处 被引量
1Glucagon - like peptide - 1 affects gene transcription and messenger ribonucleic acid stability of components of the insulin secretory system jn RIN 1046 - 38 cells 显示文摘Wang Y Egan JM Raygada M 1995Endocrinology1995,136,:1
2Managing CA-MRSA Infections: Cur- rent and Emerging options显示文摘Raygada JL Levine DP 2009Infections in Medicine2009,26,2:1
3Prepubertal exposure to zearalenone or genistein reduces mammary tu-morigenesis 显示文摘Hilakivi-Clarke L Onojafe I Raygada M 1999British Journal of Cancer1999,80,11:1
4Glucagon-like peptide-1 affects gene transcription and messenger ribonucleic acid stability of components of the insulin secretory system in RIN 1046-38 cells显示文摘Wang Y Egan JM Raygada M 1995Endocrinology1995,136,11:1
5Juvenile xanthogranuloma in a child with previously unsuspeeted neurofibromatosis type 1 and juvenile myelomonoeytie leukemia显示文摘Raygada M Arthur DC Wayne AS 2010Pediatr Blood Cancer2010,54,1:1
6Glucagon-like peptide-1 affects gene transcription and messenger ribonucleic acid stability of components of the insulin secretory system in RIN 1046 - 38 cells 显示文摘Wang Y EganJ M Raygada M 1995Endocrinology1995,136,11:1
7Multidrug efflux pump over expression in Staphylococcus aureus after single and multiple in vitro exposures to bio- cides and dyes显示文摘HUETA A RAYGADA J L MENDIRATFA K 2008Microbiol2008,154,10:1
8Muhidrug efflux pump overexpression in Staphylococcus aureus after single and multiple in vitro exposures to biocides and dyes 显示文摘Huet AA Raygada JL Mendiratta K 2008Microbiol2008,154,10:1
9Prepubertal exposure to zearalenone or genistein reduces mammary tumorigenesis显示文摘Hilakivi Clarke L Onojafe I Raygada M 1999Br J Cancer1999,80,11:1
10Integrative gene network analysis provides novel regulatory relationships, genetic con- tributions and susceptible targets in autism spectrum disor ders显示文摘Lee TL Raygada MJ Rennert OM 2012Gene2012,496,2:1
11Hereditary paragangliomas显示文摘Raygada M Pasini B Stratakis CA 0,,:1
12Glucagon-like peptide-1 af- fects gene transcription and messenger ribonucleic acid stability of components of the insulin secretory system in RIN 1046-38 cell 显示文摘Wang Y Egan J M Raygada M et a l 1995Endocrinology1995,136,:1
13Prepubertal exposure to zearalenone or genistein reduces mammary tumorigenesis显示文摘Hilakivi C L Onojafe I Raygada M 1999Br J Cancer1999,80,:1
14Multidrug efflux pump overexpression in Staphylococcus aureus after single and multiple in vitro exposures to biocides and dyes显示文摘Huet AA Raygada JL Mendiratta K 2008Microbiology2008,154,10:1
15Managing CA-MRSA infections: current and emerging options显示文摘Raygada J L Levine D P 2009Infect Med2009,26,2:1
16自发性卵巢早衰女性生育脆性X染色体综合征儿显示文摘Objective: To inform clinicians about a reproductive risk associated with spontaneous premature ovarian failure and the fragile X mental retardation 1 gene (FMR1). Design: Case report. Setting: National Institutes of Health Clinical Center. Patient(s): A 35-year-old woman with confirmed spontaneous premature ovarian failure. Intervention(s): FMR1 genetic testing. Main Outcome Measure(s): Number of CGG trinucleotide repeats in the 5′untranslated region of FMR1. Result(s): Despite having ovarian failure the woman subsequently conceived and delivered a son with fragile X syndrome (>200 CGG repeats). She was then found to carry an FMR1 premutation (85 CGG repeats). Conclusion(s): This is a real-life manifestation of a theoretical risk; a woman conceived subsequent to the diagnosis of spontaneous premature ovarian failure and has a child who manifests mental retardation due to fragile X syndrome. Women with spontaneous premature ovarian failure are at increased risk of having an FMR1 premutation and should be informed of the availability of fragile X testing. Should an FMR1 premutation be uncovered, this will allow patients to make informed reproductive decisions and help clinicians to properly diagnose family members who may have menstrual irregularity, developmental delay, or neurologic symptoms.Corrigan E.C. Raygada M.J. Vanderhoof V.H. Nelson L.M. 李跃萍 2006世界核心医学期刊文摘(妇产科学分册)2006,0,3:0
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