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15篇 您的检索式:作者名="Proby"
    题名 作者 年代 出处 被引量
1Wnt5a is strongly expressed at the leading edge in non-melanoma skin cancer, forming active gradients, while canonical Wnt signaling is repressed显示文摘Pourreyron C Reilly L Proby C 2012PloS One2012,7,31:1
2Human papillomavims and the development of non-melanoma skin cancer显示文摘HARWOOD C A MCGREGOR J M PROBY C M 1999Clin Pathol1999,52,:1
3Effects of epidermal growth factor receptor and insulin-like growth factor- I receptor inhibition on proliferation and intracellular signaling in cutaneous SCCHN: Potential for dual inhibition as a therapeutic modality显示文摘Clayburgh Daniel R Gross Neil D Proby Charlotte etal 2013Head and Neck-Journal for the Sciences and Specialties of the Head and Neck2013,,:1
4Clinicopathologic features of skin cancer in organ transplant recipients:a retrospective case-control series显示文摘Harwood CA Proby CM McGregor JM 2006J Am Acad Dermatol2006,54,2:1
5Wnt5a is strongly ex pressed at the leading edge in non-melanoma skin cancer forming active gradients, while canonical Wnt signalling is re pressed显示文摘Pourreyron C Reilly L Proby C etal 2012PLoS One2012,7,31:1
6Development of chimeric molecules for recognition and targeting of antigen-specific B cells in pemphigus vulgaris 显示文摘 Ota T Suzuki H 2000Br J Dermatol2000,142,2:1
7Wnt5a Is strongly expressed at the leading edge in non-melanoma skin canc?er, forming active gradients, while canonical wnt signalling is repressed显示文摘Pourreyron C Reilly L Proby Cv 2012PLoS One2012,7,31:1
8Development of chimericmolecules for recognition and targeting of antigen-specific Bcells in pemphigus vulgaris显示文摘Proby CM Ota T Suzuki H 2000Br J Dermatol2000,142,2:1
9Human pap-illomavirus and the development of non-melanoma skin cancer显示文摘Harwood CA McGregor JM Proby CM* 1999J Clin Pathol1999,52,4:1
10Human papillomavirus and the development of non-melanoma skin cancer显示文摘Harwood C A McGregor J M Proby C M 1999J Clin Pathol1999,52,:1
11Wnt5a is strongly expressed at the leading edge in Non-Melanoma Skin Cancer,forming active gradients,while Canonical Wnt Signalling is repressed显示文摘Pourreyron C Reilly L Proby C 0,,02:1
12Human papillomavirus and the development of non-melanoma skin cancer显示文摘Harwood CA McGregor JM Proby CM 1999J Clin Pathol1999,52,4:1
13Effects of epidermal growth factor receptor and insulin-like growth factor 1 receptor inhibition on proliferation and intracellular signaling in cutaneous SCCHN: potential for dual inhibition as a therapeutic modality显示文摘Clayburgh DR Gross ND Proby C 2013Head Neck2013,35,1:1
14Wnt5a is strongly expressed at the leading edge in non-melanoma skin cancer, forming active gradients, while canonical Wnt signaling is repressed 显示文摘POURREYRON C REILLY L PROBY C 2012PloS One2012,7,31:1
15器官移植受者皮肤癌的临床病理特征:一项回顾性病例对照研究显示文摘Background: Non- melanoma skin cancers (NMSCs) are increased in organ transplant recipients, but transplant and immunocompetent squamous and basal cell carcinomas (SCCs, BCCs) have not been compared previously in a single- center study. Objective: To compare clinicopathologic features of transplant and immunocompetent NMSCs. Methods: Consecutive transplant NMSCs (60 SCCs, 100 BCCs) and immunocompetent NMSCs (40 SCCs, 125 BCCs) presenting between 1995- 1997. Results: Transplant patients were 15 years younger at time of NMSC diagnosis compared with immunocompetent individuals, and transplant tumors were often more multiple and extra cephalic. Spindle cell morphology was more common in transplant SCCs, a superficial component was more common in transplant BCCs, and histologic features of HPV infection were overrepresented in transplant tumors. Outcome was worse for transplant SCCs but not transplant BCCs. Limitations: Histologic features required to identify HPV infection have not been validated. Conclusions: These findings have direct implications for clinical care. The increased frequency and distribution of transplant NMSCs underscore the importance of whole- body surveillance. Transplant SCCs, particularly those with diffuse spindle cell change, may require more aggressive management, whereas transplant BCCs do not. Finally, our data support differences in the pathogenesis of transplant NMSC, which may influence future preventive and therapeutic strategies.Harwood C.A. Proby C.M. McGregor J.M. 刘艳 2006世界核心医学期刊文摘(皮肤病学分册)2006,0,4:0
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