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| 1 | Use of a land-surface-transfer scheme(LSX) in a global climate model (GENRSIS):The response to doubling stomatal resistance显示文摘 | Pollard D Thompson S L | 1995 | Global Planet Change1995，，10： | 2 |
| 2 | Niche-independent symmetrical self-renewal of a mammalian tissue stem cell显示文摘 | Conti L Pollard SM Gorba T | 2005 | Plos Biol2005，3，9： | 1 |
| 3 | ANXA7 expression represents hormone-relevant tumor suppression in different cancers显示文摘 | Srivastava M Torosyan Y Raffeld M Eidelman O Pollard H B Bubendorf L | 2007 | Int J Cancer2007，121，： | 1 |
| 4 | Comparison of 3 techniques for ureteroneocystostomy in cats显示文摘 | Mehl M L Kyles A E Pollard R | 2005 | Vet Surg2005，34，： | 1 |
| 5 | Adherent neural stem (NS) cells from fetal and adult forebrain 显示文摘 | Pollard SM Conti L Sun Y | 2006 | Cereb Cortex2006，16，1： | 1 |
| 6 | How to constrain 3-D fault continuity and linkage using reflection seismic data: A geomechanical approach 显示文摘 | Maerteri L Pollard D D Karpuz R | 2000 | AAPG Bulletin2000，84，： | 1 |
| 7 | Long-term tripotent differentiation capacity of human neural stem (NS) cells in adherent culture显示文摘 | Sun Y Pollard S Conti L | 2008 | Mol Cell Neurosei2008，38，2： | 1 |
| 8 | Molecular mechanisms controlling actin filament dynamics in nonmuscle cells显示文摘 | POLLARD TD BLANCHOIN L MULLINS RD | 2000 | Annu Rev Biophys Biomol Struct2000，29，： | 1 |
| 9 | Niche-independent symmetrical self-renewal of a mammalian tissue stem cell显示文摘 | Conti L Pollard SM Gorba T | 2005 | PLoS Biol2005，3，9： | 1 |
| 10 | 查看详情显示文摘 | Pollard J A Zhang D S Downing J A Knorr F J McHale J L J | | 0，，： | 1 |
| 11 | Niche-independent symmetrical self-renewal of a mammalian tissue stem cell 显示文摘 | Conti L Pollard SM Gorba T | 2005 | PLoS Biol2005，3，： | 1 |
| 12 | Quantification of branched-chain amino acids in blood spots and plasma by liquid chroma- tography tandem mass spectrometry for the diagnosis of maple syrup urine disease 显示文摘 | Sowell J Pollard L Wood T | 2011 | J Sep Sci2011，34，6： | 1 |
| 13 | The age pattern of Mortality 显示文摘 | Heligman L Pollard J H | 1980 | Journal of the Institute of Actuaries1980，，107： | 1 |
| 14 | Cyclosporine A in resistant chronic inflammatory demyelinating polyradiculoneuropathy 显示文摘 | Barnett MH Pollard JD Davies L | 1998 | Muscle Nerve1998，21，4： | 1 |
| 15 | IBem3D,a three-dimensional iterative boundary element method using angular dislocations for modeling geologic structures显示文摘 | MAERTEN F MAERTEN L POLLARD D D | 2014 | Computers&Geosciences2014，，72： | 1 |
| 16 | Not all arrestins are created equal: Therapeutic implications of the functional diversity of the β-arrestins in the heart显示文摘The two ubiquitous, outside the retina, G protein-coupled receptor(GPCR)adapter proteins, β-arrestin-1 and-2(also known as arrestin-2 and-3,respectively), have three major functions in cells: GPCR desensitization, i.e.,receptor decoupling from G-proteins; GPCR internalization via clathrin-coated pits; and signal transduction independently of or in parallel to G-proteins. Bothβ-arrestins are expressed in the heart and regulate a large number of cardiac GPCRs. The latter constitute the single most commonly targeted receptor class by Food and Drug Administration-approved cardiovascular drugs, with about onethird of all currently used in the clinic medications affecting GPCR function.Since β-arrestin-1 and-2 play important roles in signaling and function of several GPCRs, in particular of adrenergic receptors and angiotensin II type 1 receptors,in cardiac myocytes, they have been a major focus of cardiac biology research in recent years. Perhaps the most significant realization coming out of their studies is that these two GPCR adapter proteins, initially thought of as functionally interchangeable, actually exert diametrically opposite effects in the mammalian myocardium. Specifically, the most abundant of the two β-arrestin-1 exerts overall detrimental effects on the heart, such as negative inotropy and promotion of adverse remodeling post-myocardial infarction(MI). In contrast, β-arrestin-2 is overall beneficial for the myocardium, as it has anti-apoptotic and antiinflammatory effects that result in attenuation of post-MI adverse remodeling,while promoting cardiac contractile function. Thus, design of novel cardiac GPCRligands that preferentially activate β-arrestin-2 over β-arrestin-1 has the potential of generating novel cardiovascular therapeutics for heart failure and other heart diseases. | Anastasios Lymperopoulos Shelby L Wertz Celina M Pollard Victoria L Desimine Jennifer Maning Katie A McCrink | 2019 | World Journal of Cardiology2019，11，2： | 1 |
| 17 | Com- parison of 3 Techniques for Ureteroneocystostomy in Cats显示文摘 | Margo L Mehi Andrew E Kyles Pollard Pachel | 2005 | Vet Surg2005，34，： | 1 |
| 18 | Zebrafish mutants identify an essential role for laminins in notochord formation 显示文摘 | Parsons MJ Pollard SM Saude L | 2002 | Development2002，129，： | 1 |
| 19 | Lupin flours asadditives: dough mixing, breadmaking, emulsifying, andfoaming 显示文摘 | Pollard N J Stoddard F L Popineau Y | 2002 | Cereal chemistry2002，79，5： | 1 |
| 20 | Beat-to-beat repolarization variability in ventricular myocytes and its suppression by electrical coupling显示文摘 | Zaniboni M Pollard AE Yang L | 2000 | Am J Physiol2000，27，： | 1 |
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