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17篇 您的检索式:作者名="Mogle"
    题名 作者 年代 出处 被引量
1Evolving experiencewith prevention and treatment of splenic artery syndrome afterorthotopic liver transplantation 显示文摘Mogl MT Niissler NC Presser SJ 2010Transpl Int2010,23,8:1
2Older liver graft transplantation,cholestasis and synthetic graft function 显示文摘Borchert D Glanemann M Mogl M 2005Transpl Int2005,18,6:1
3Evolving experience with prevention and treatment of splenic artery syndrome after orthotopic liver transplantation显示文摘Mogl MT Nussler NC Presser SJ 2010Transplant Int2010,23,8:1
4Emphusem-atous Pyelonephritis显示文摘Michaeli J Mogle P Perlberg S 0,,02:1
5Older liver graft transplantation,cholestasis and synthetic graft function显示文摘Borchert D Glanemann M Mogl M 2005Transpl Int2005,18,6:1
6Adult liver transplantation using liver grafts from donors over 70 years of age 显示文摘Borchert DH Glanemann M Mogl M 2005Transplant Proc2005,37,2:1
7Evolving experience with pervention and treatment of splenic artery syndrome after orthotopic liver transplantion显示文摘Mogl MT Nussler NC Presser SJ 2010Transpl Int2010,23,8:1
8Multidetector computed tomography for preoperative assessment of hepatic vasculature and prediction of splenic artery steal syndrome in patients with liver cirrhosis before transplantation显示文摘Christian Grieser Timm Denecke Ingo G. Steffen Maria Avgenaki Vera Fr?hling Martina Mogl Dirk Schnapauff Lukas Lehmkuhl Lars Stelter Florian Streitparth Jan Langrehr Jan-Holger Rothe Bernd Hamm Enrique Lopez H?nninen 2010European Radiology2010,,1:1
9Evolving experience with prevention and treatment of splenic artery syndrome after orthotopic liver transplantation显示文摘Mogl MT Nussler NC Presser SJ 2010Transplant Int2010,23,8:1
10Older liver graft transplantation,cholestasis and synthetic graft function显示文摘Borchert D Glanemann M Mogl M 2005Transpl Int2005,18,6:1
11Respiratory failure and multiple fractures in vitamin D-dependent rickets 显示文摘Alpan G Mogle P Patz D 1985Acta Paediatr Scand1985,74,2:1
12General Method for Ultrashort Light Pulse Chirp Measurement显示文摘K Naganuma K Mogl H Y 1989IEEE Journal of Quantum Electronics1989,25,6:1
13Evolving experience with prevention and treatment of splenic artery syndrome after orthotopic liver transplantation显示文摘Mogl MT Nussler NC Presser S J 2010Transplant Int2010,23,8:1
14The normal width of the adult hip joint:the relationship to age,sex and obesity显示文摘Pogrund H Bloom R Mogle P 1983Skeletal Radiol1983,10,1:1
15Safety and efficacy of new integrated bipolar and ultrasonic scissors compared to conventional laparoscopic 5-mm sealing and cutting instru- ments显示文摘Seehofer D Mogl M Boas-Knoop S 2012Surg Endosc2012,26,:1
16Extraskeletal soft-tissue chondro- sarcoma显示文摘Amir D Amir G Mogle P 1985Clin Orthop1985,198,:1
17An unhappy triad:Hemochromatosis, porphyria cutanea tarda and hepatocellular carcinoma-A case report显示文摘Liver fibrosis and cirrhosis are predisposing factors for the development of hepatocellular carcinoma (HCC). Hemosiderosis has also been described to trigger carcinogenesis. A significant iron overload, as found in hereditary hemochromatosis (HHC), is a risk factor for HCC and may also promote the symptoms of porphyria cutanea tarda (PCT). A 68-year old male patient presented to our clinic with a suspected HCC, elevated alpha-fetoprotein but normal liver function tests. He reported a 25 year-old history of vitiligo upon exposure to sunlight. The patient underwent an extended left hemihepatectomy, and the recovery was uneventful, with the exception of a persistent hyperbilirubinemia. Perfusion problems and extrahepatic cholestasis were ruled out by CT-scan with angiography and MR-cholangiopancreatography. However, MRI showed an iron overload. Histology confirmed the HCC (pT3, pN0, G3, R0) and revealed a portal fibrosis and hemosiderosis. Based on the skin lesions we suspected a PCT that was confirmed by laboratory tests showing elevated porphyrin, uroporphyrin, coproporphyrin and porphobilinogen. Concurrently, molecular diagnostics revealed homozygosity for the C282Y mutation within the hemochromatosis HFE gene. After phlebotomy and normalization of liver function tests the patient was discharged. This is the first case ever showing the unusual combination of HCC in a fibrotic liver with HHC and PCT. This diagnosis not only warrants oncological follow-up but also symptomatic therapy to normalize iron metabolism and thereby improve liver function and alleviate the symptoms of HHC and PCT. Thus progression of fibrosis may be prevented and liver regeneration supported.Martina T Mogl Andreas Pascher Sabine J Presser Michael Schwabe Peter Neuhaus Natascha C Nuessler 2007World Journal of Gastroenterology2007,13,13:0
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