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| 1 | Evaluation of hepatic cystic lesions显示文摘Hepatic cysts are increasingly found as a mere coincidence on abdominal imaging techniques, such as ultrasonography (USG), computed tomography (CT) and magnetic resonance imaging (MRI). These cysts often present a diagnostic challenge. Therefore, we performed a review of the recent literature and developed an evidence-based diagnostic algorithm to guide clinicians in characterising these lesions. Simple cysts are the most common cystic liver disease, and diagnosis is based on typical USG characteristics. Serodiagnostic tests and microbubble contrast-enhanced ultrasound (CEUS) are invaluable in differentiating complicated cysts, echinococcosis and cystadenoma/cystadenocarcinoma when USG, CT and MRI show ambiguous findings. Therefore, serodiagnostic tests and CEUS reduce the need for invasive procedures. Polycystic liver disease (PLD) is arbitrarily defined as the presence of > 20 liver cysts and can present as two distinct genetic disorders: autosomal dominant polycystic kidney disease (ADPKD) and autosomal dominant polycystic liver disease (PCLD). Although genetic testing for ADPKD and PCLD is possible, it is rarely performed because it does not affect the therapeutic management of PLD. USG screening of the liver and both kidneys combined with extensive family history taking are the cornerstone of diagnostic decision making in PLD. In conclusion, an amalgamation of these recent advances results in a diagnostic algorithm that facilitates evidence-based clinical decision making. | Marten A Lantinga Tom JG Gevers Joost PH Drenth | 2013 | World Journal of Gastroenterology2013，19，23： | 12 |
| 2 | Prevention of overuse: A view on upper gastrointestinal endoscopy显示文摘Many upper gastrointestinal(GI) endoscopies worldwide are performed for inappropriate indications. This overuse of healthcare negatively affects healthcare quality and puts pressure on endoscopy services. Dyspepsia is one of the most common inappropriate indications for upper GI endoscopy as diagnostic yield is low. Reasons for untimely referral are: unfamiliarity with dyspepsia guidelines,uncertainty about etiology of symptoms, and therapy failure. Unfiltered openaccess referrals feed upper GI endoscopy overuse. This review highlights strategies applied to diminish use of upper GI endoscopies for dyspepsia. First,we describe the impact of active guideline implementation. We found improved guideline adherence, but resistance was encountered in the process. Secondly, we show several forms of clinical assessment. While algorithm use reduced upper GI endoscopy volume, effects of referral assessment of individual patients were minor. A third strategy proposed Helicobacter pylori test and treat for all dyspeptic patients. Many upper GI endoscopies can be avoided using this strategy, but outcomes may be prevalence dependent. Lastly, empirical treatment with Proton pump inhibitors achieved symptom relief for dyspepsia and avoided upper GI endoscopies in about two thirds of patients. Changing referral behavior is complex as contributing factors are manifold. A collaboration of multiple strategies is most likely to succeed. | Judith J de Jong Marten A Lantinga Joost PH Drenth | 2019 | World Journal of Gastroenterology2019，25，2： | 3 |
| 3 | Serum S-100 and neuron specific enolase for prediction of regaining consciousness after global cerebral ischemia 显示文摘 | Martens P Raabe A Johnsson P | 1998 | Stroke1998，29，11： | 2 |
| 4 | Interactions Between Dendritic ceils and cytokine-induced kiLler cells lead to an activation of both populations显示文摘 | Marten A Ziske C Schottker B | 2001 | J Immunohherapy2001，24，： | 1 |
| 5 | Paternal leakage of mitochondrial DNA in the great tit ( Parus major) 显示文摘 | KVIST L MARTENS J NAZARENKO A A | 2003 | Mol Biol Evol2003，20，2： | 1 |
| 6 | Heterodimerization between members of the Nur subfamily of orphan nuclear receptors as a novel mechanism for gene Activation 显示文摘 | Maira M Martens C Philips A | 1999 | Mol Cell Biol1999，19，11： | 1 |
| 7 | Minimal reaidual disease in acute leukemia:preclinical studies in a relevant rat model(BNML)显示文摘 | Hagenbeek A Martens AC | 1991 | Bailli Clin Haematol1991，4，： | 1 |
| 8 | Phylogeny of long-tailed tits and allies inferred from mitochondrial and nuclear markers (Aves:Passeriformes,Aegithalidae)显示文摘 | P(a)ckert M Martens J Sun Y-H | | 0，，： | 1 |
| 9 | X-ray structure of RLI, an essential twin cassette ABC ATPase involved in ribosome biogenesis and HIV capsid assembly 显示文摘 | Kareher A Buttner K Martens B | 2005 | Structure2005，13，4： | 1 |
| 10 | DNA methyl-ation asa biomarker in breast cancer显示文摘 | Martens J W Margossian A L Schmitt M | 2009 | Future Oncol2009，5，8： | 1 |
| 11 | Validation and verification of regression in small data sets 显示文摘 | MARTENS H A DARDENNE P | 1998 | Chemometrics and Intelligent Laboratory Systems1998，44，12： | 1 |
| 12 | Crystallines microporous phosphates: A family of versatile catalysts and adsorbents 显示文摘 | MARTENS J M JACOBS P A | 1994 | Studies in Surface Science and Catalysis1994，85，： | 1 |
| 13 | Enhanced lytic activity of cy-tokine-Induced killer cell against multiple myelomacell after coculture with idio-type~pulsed dendritic cells显示文摘 | Marten A Renoth S Von-Lilienfeld- toal M | 2001 | Haematologica2001，86，10： | 1 |
| 14 | Estimation of tree canopy leaf area index by gap fraction analysis显示文摘 | Martens S N Ustin S L Rousseau R A | 1993 | Forest Ecology and Management1993，61，12： | 1 |
| 15 | The dimensions of perfectionism显示文摘 | Frost R O Marten P A Lahart C | 1990 | Cognit TherRes1990，14，5： | 1 |
| 16 | Estimation of the void structure and pore dimensions of molecular sieve zeo- lites using the hydroconversion of n-deeane 显示文摘 | Martens J A Tielen M Jacobs P A | 1984 | Zeolites1984，4，2： | 1 |
| 17 | Isomerization and hydrocracking of heptane over bimetallic bifunctional Pt- Pd/H - Beta and PtPd/USY zeolite catalysts 显示文摘 | Blomsma E Martens J A Jacobs P A | 1997 | J Catal1997，165，： | 1 |
| 18 | Management and crop residue influence soil aggregate stability显示文摘 | Marten D A | 2000 | Journal of Environmental Quality2000，29，： | 1 |
| 19 | Interactions between dendritic cells and cytokine-induced killer cells lead to an activation of both population显示文摘 | Ziske C Renoth S | 2001 | J Immunotherapy2001，24，6： | 1 |
| 20 | Interactions between dendritic cells and cytokine-induced killer cells lead to an activation of both populations显示文摘 | Marten A Ziske C Schottker B | 2001 | J Immunother2001，24，6： | 1 |
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