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67篇 您的检索式:作者名="LINDSTEN T"
    题名 作者 年代 出处 被引量
1The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues显示文摘Lindsten T Ross AJ King A 2000Mol Ce112000,6,:1
2The combined functions of pro- apoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues 显示文摘Lindsten T Ross A J King A 2000Mol Cell2000,6,6:1
3The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues 显示文摘LINDSTEN T ROSS A J KING A 2000Mol Cell2000,6,6:1
4BH3-only proteins that bind pro-survival Bcl-2 family members fail to in- duce apoptosis in the absence of Bax and Bak 显示文摘Zong WX Lindsten T Ross AJ 2001Genes Dev2001,15,12:1
5The combined functions of proapoptotic Bcl-2 family members bakand bax are essential for normal development of multiple tissues显示文摘Lindsten T Ross AJ King A 2000Mol Cell2000,6,:1
6Bax is the principal proapoptotic Bcl-2 -related proteinduring differentiation of the oligodendroglial lineage显示文摘Itoh T Itoh A Lindsten T 2004J Neurochem2004,90,1:1
7Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasis显示文摘Rathmell JC Lindsten T Zong WX 2002Nat Immunol2002,3,10:1
8BH3-only proteins that bind pro survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak显示文摘Zong W X Lindsten T Ross AJ 2001Genes Dev2001,15,12:1
9The combined functions of proapop- totie Bcl -2 family members Bax are essential for normal development of multiple tissues 显示文摘Lindsten T Ross A J King A 2006Mol Cell2006,,6:1
10Characterization of CTLA-4 structure and expression on human T cells 显示文摘Lindsten T Lee KP Harris ES 1993J Immunol1993,151,7:1
11Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis显示文摘Zong W X Li C Hatzivassiliou G Lindsten T Yu Q C Yuan J Thompson C B 2003J Cell Biol2003,162,1:1
12Therapeutic targets in cancer cell metabolism and autophagy显示文摘Cheong H Lu C Lindsten T 2012Nat Biotechnol2012,30,7:1
13Bcl-x, a bcl-2-related gene that functions as a dominant regulator ofapoptotic cell death 显示文摘Boise LH Gonzalez-Garcia M Postema CE Ding L Lindsten T Turka LA Mao X NunezG Thompson CB 1993Cell1993,74,4:1
14tBID, a membrane-targeted death ligand, oligonerizes BAK to release cytochrone c 显示文摘Wei MC Lindsten T Mootha VK 2000Genes Dev2000,14,16:1
15BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak显示文摘 LINDSTEN T ROSS A J 2001Genes Dev2001,15,12:1
16tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c显示文摘Wei MC Lindsten T Mootha VK 2000Genes Dev2000,14,:1
17Bax and Bak indenpendently promote cytochrome C release from mitochondria 显示文摘Degenhardt K Sundararajan R Lindsten T 2002J Biol Chem2002,277,14:1
18The combined functions of proapoptotie Bcl-2 family members Bak and Bax are essential for normal development of multiple tissues显示文摘Lindsten T Ross AJ King A 2000Mol Cell2000,6,:1
19The combined functions of proapop- tic Bel- 2 familymembers Bak and Bax areessential for mormal de- velopment of multiple tissues 显示文摘Lindsten T Ross AT Ki A 2000Molec cell2000,6,:1
20The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues 显示文摘Lindsten T Ross AJ King A 2000Mol Cell2000,6,:1
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