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| 1 | A new adaptive switching median filter显示文摘 | AKKOUL S LEDEE R LECONGE R | 2010 | IEEE Signal Process Letters2010，17，7： | 1 |
| 2 | A new adaptive switching median filter显示文摘 | Akkoul S Ledee R Leconge R | | 0，，6： | 1 |
| 3 | A new adaptive switching median filter 显示文摘 | Akkoul S Ledee R Leconge R | 2010 | IEEE Signal Processing Let- ters2010，17，6： | 1 |
| 4 | A new adaptive switching median filter显示文摘 | Akkoul S Ledee R Leconge R | 2010 | Signal Processing Letters IEEE2010，17，6： | 1 |
| 5 | A new adaptive switching median filter显示文摘 | Akkoul S Ledee R Leconge R | 2010 | Signal Processing Letters2010，17，6： | 1 |
| 6 | A new adaptive switching median filter显示文摘 | Ledee R Leconge R Harba R | | 0，，06： | 1 |
| 7 | A new adaptive switching median filter显示文摘 | Akkoul S Ledee R Leconge R | 2010 | Signal Processing Letters IEEE2010，17，6： | 1 |
| 8 | A new adaptive switching median filter显示文摘 | AKKOUL S LECONGE R HARBA R | | 0，，： | 1 |
| 9 | Chronic administration of triclosan leads to liver fibrosis through hepcidin-ferroportin axis-mediated iron overload显示文摘Triclosan(TCS)has been manufactured as an antibacterial compound for half a century.Currently,it is widely used in various personal care products;however,its potential adverse effects raise a lot of attention.Here,we create a long-term oral administration mouse model and identify the corresponding hepatotoxicity of TCS.We discover that daily intragastric administration of 10 mg/kg TCS to mice for 12 weeks results in severe hepatic fibrosis.Further study displays that hepatic iron increased 18%,23%and 29%upon oral TCS treatment for4,8 and 12 weeks,respectively.Accompanied by hepatic iron variation,splenic and duodenal iron are increased,which indicates systemic iron disorder.Not only excessive iron accumulated in the liver,abnormal hepatic malondialdehyde,prostaglandin synthase 2 and glutathione peroxidase 4 are pointed to ferroptosis.Additional study uncovers that hepcidin expression increases 7%,10%,4%in serum and 2.4-,4.8-,and 2.3-fold on transcriptional levels upon TCS exposure for 4,8 and 12 weeks,individually.Taken together,the mice in the TCS-treated group show disordered systemic iron homeostasis via the upregulated hepatic hepcidin-ferroportin axis.Meanwhile,both hepatic iron overload(systemic level)and hepatocyte ferroptosis(cellular level)are accused of TCS-induced liver fibrosis.Ferriprox■,an iron scavenger,significantly ameliorates TCS-induced liver fibrosis.In summary,this study confirms the impact of TCS on liver fibrosis;a critical signal pathway is also displayed.The significance of the current study is to prompt us to reevaluate the“pros and cons”of TCS applications. | Jing Liu Lecong Zhang Fang Xu Ping Zhang Yang Song | 2024 | Journal of Environmental Sciences2024，，3： | 0 |
| 10 | 2004年春季广东四大家具展统计与分析显示文摘以下数据通过第13届广州家具博览会(下简称广州展)、第11届国际名家具(东莞)展览会(下简称东莞展}、第14届深圳国际家具展(下简称深圳展)、第7届龙家具精品展览会(下简称龙江展)展会会刊上参展企业名录(不包括广告、媒体类企业)统计。 | Lecong Furniture Promotion Center | 2004 | 家具2004，25，2： | 0 |
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