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| 1 | Gastrointestinal complications after kidney transplantation显示文摘Gastrointestinal complications are common after renal transplantation,and they have a wide clinical spectrum,varying from diarrhoea to post-transplant inflammatory bowel disease(IBD).Chronic immunosuppression may increase the risk of post-transplant infection and medication-related injury and may also be responsible for IBD in kidney transplant re-cipients despite immunosuppression.Differentiating the various forms of post-transplant colitis is challenging,since most have similar clinical and histological features.Drug-related colitis are the most frequently encountered colitis after kidney transplantation,particularly those related to the chronic use of mycophenolate mofetil,while de novo IBDs are quite rare.This review will explore colitis after kidney transplantation,with a particular focus on different clinical and histological features,attempting to clearly identify the right treatment,thereby improving the final outcome of patients. | Rossella Gioco Daniela Corona Burcin Ekser Lidia Puzzo Gaetano Inserra Flavia Pinto Chiara Schipa Francesca Privitera Pierfrancesco Veroux Massimiliano Veroux | 2020 | World Journal of Gastroenterology2020，26，38： | 6 |
| 2 | Endobronchial tumor in children:Unusual finding in recurrent pneumonia,report of three cases显示文摘We are reporting 3 cases of pediatric endobronchialtumors presented with recurrent pneumonia. The median age of patients, at time of presentation, was 10.6 years. All patients presented with recurrent pneumonia with a mean time to occurrence, after onset of symptoms, of 14 mo. Bronchoscopy was early performed as part of diagnostic work-up and it revealed an endobronchial mass in every case. Complete surgical resection was performed in all cases, with lung preservation in two of them. Neither post-operative chemotherapy nor radiotherapy was required. The mean duration of follow-up was 7 years and all patients are still alive and disease-free. Recurrent pneumonia, in pediatrics, should raise the suspicion of an obstructing lesion, congenital malformation or systemic disease. A systematic approach is useful for organize the clinicians initial workup. Prompt diagnosis allows parenchymal-sparing surgery, which offers the best chance of cure and reduces clinical and functional complications in these patients. | Silvia Madafferi Vincenzo D Catania Antonella Accinni Renata Boldrini Alessandro Inserra | 2015 | World Journal of Clinical Pediatrics2015，4，2： | 5 |
| 3 | Arterial structure and function in inflammatory bowel disease显示文摘Inflammatory bowel disease(IBD) is the result of a combination of environmental,genetic and immunologic factors that trigger an uncontrolled immune response within the intestine,which results in inflammation among genetically predisposed individuals. Several studies have reported that the prevalence of classic cardiovascular risk factors is lower among subjects with IBD than in the general population,including obesity,dyslipidaemia,diabetes and hypertension. Therefore,given the risk profile of IBD subjects,the expected cardiovascular morbidity and mortality should be lower in these patients than in the general population. However,this is not the case because the standardized mortality ratio is not reduced and the risk of coronary heart disease is increased in patients with IBD. It is reasonable to hypothesize that other factors not considered in the classical stratification of cardiovascular risk may be involved in these subjects. Therefore,IBD may be a useful model with which to evaluate the effects of chronic low-grade inflammation in the development of cardiovascular diseases. Arterial stiffness is both a marker of subclinical target organ damage and a cardiovascular risk factor. In diseases characterized by chronic systemic inflammation,there is evidence that the inflammation affects arterial properties and induces both endothelial dysfunction and arterial stiffening. It has been reported that decreasing inflammation viaanti tumor necrosis factor alpha therapy decreases arterial stiffness and restores endothelial function in patients with chronic inflammatory disorders. Consistent with these results,several recent studies have been conducted to determine whether arterial properties are altered among patients with IBD. In this review,we discuss the evidence pertaining to arterial structure and function and present the available data regarding arterial stiffness and endothelial function in patients with IBD. | Luca Zanoli Stefania Rastelli Gaetano Inserra Pietro Castellino | 2015 | World Journal of Gastroenterology2015，21，40： | 3 |
| 4 | Increased arterial stiffness in inflammatory bowel diseases is dependent upon inflammation and reduced by immunomodulatory drugs显示文摘 | Luca Zanoli Stefania Rastelli Gaetano Inserra Paolo Lentini Enrico Valvo Emanuela Calcagno Pierre Boutouyrie Stephane Laurent Pietro Castellino | 2014 | Atherosclerosis2014，，2： | 2 |
| 5 | Inflammation and the metabolic syndrome: Role of angiotensin II and oxidative stress显示文摘 | León Ferder MD PhD Felipe Inserra MD Manuel Martínez-Maldonado MD | 2006 | Current Hypertension Reports2006，，3： | 1 |
| 6 | Angiotensin Ⅱ,mitochondria,cyto-skeletal,and extracellular matrix connections:an integrating viewpoint显示文摘 | de Cavanagh EM Ferder M Inserra F | 2009 | Am J Physiol Heart Circ Physiol2009，296，3： | 1 |
| 7 | Dietary citrus pulp reduces lipid oxidation in lamb meat 显示文摘 | INSERRA L PRIOLO A BIONDI L | 2014 | Meat Science2014，96，4： | 1 |
| 8 | Effect of temperature on infection and survival of Rotylenchulus reni formis 显示文摘 | Heald C M Inserra R N | 1988 | Journal of Nematology1988，20，： | 1 |
| 9 | Enalaprilattenuates oxidative stress in diabetic rats显示文摘 | DE CAVANAGH EM INSERRA F TOBLLI J | 2001 | Hypertension2001，38，5： | 1 |
| 10 | Functional indices {or sciatic, peroneal, and posterior tibial nerve lesions in the mouse 显示文摘 | Inserra M M Bloch D A Terris D J | 1998 | Microsurgery1998，18，2： | 1 |
| 11 | The world pandemic of vitamin D deficiency could possibly be explained by cellular in- flammatory response activity induced by the renin-angiotensin sys- tem显示文摘 | Ferder M Inserra F Manucha W | 2013 | Am J Pbysiol Cell Physiol2013，304，11： | 1 |
| 12 | inflammation and metabolic ayndrome: role of anglotensin Ⅱ and oxidative stress 显示文摘 | Ferder L Inserra F Martinez-Maldonado M | 2006 | Curr Hypertens Rep2006，，： | 1 |
| 13 | Human papillomavirus persistence and nutrients involved in the methylation pathway among a cohort of young women显示文摘 | Sedjo RL Inserra P Abrahamsen M | 2002 | Cancer Epidemiol Biomarlkers Prev2002，11，4： | 1 |
| 14 | Rotylenchulus species: identification, distribution, host ranges, and crop plant resistance显示文摘 | ROBINSON A F INSERRA R N CASWELL C E P | 1997 | Nematropica1997，27，： | 1 |
| 15 | Melanoma in a 5-year- old child with a giant congenital melanocytic naevus显示文摘 | Surrenti T Dioeiaiuti A Inserra A | 2012 | Acta Derm Venereol2012，92，6： | 1 |
| 16 | AngiotensinⅡ,mito-chondria,cytoskeletal,and extracellular matrix connections:an in-tegrating viewpoint显示文摘 | de Cavanagh E M Ferder M Inserra F | 2009 | Am J Physiol Heart Circ Physiol2009，296，3： | 1 |
| 17 | Morphological changes in cavernous tissue in spontaneously hypertensive rats显示文摘 | Toblli JE Stella I Inserra F | | 0，，6： | 1 |
| 18 | Rotylenchulus species: identification, host ranges, and crop plant resistance显示文摘 | Robinson A F Inserra R N Caswell-Chen E P | 1997 | Nematropica1997，27，2： | 1 |
| 19 | Morphological changes in cavernous tissue in spontaneously hypertensive rats显示文摘 | Stella I Inserra F | 2000 | Am J Hypertens2000，13，61： | 1 |
| 20 | Anatomy involved in the jugular foramen approach for jugul otympanic paraganglioma resection显示文摘 | Inserra ML Pfister M Jackler RK | 2004 | Neurosurg Focus2004，17，2： | 1 |
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