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20篇 您的检索式：作者名="Haydont"

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	题名	作者	年代	出处	被引量
1	Maintenance of radiation-induced intestinal fibrosis:Cellular and molecular features显示文摘Recent advances in cell and molecular radiobiology clearly showed that tissue response to radiation injury cannot be restricted to a simple cell-killing process, but depends upon continuous and integrated pathogenic processes, involving cell differentiation and crosstalk between the various cellular components of the tissue within the extracellular matrix. Thus, the prior concept of primary cell target in which a single-cell type (whatever it’s epithelial or endothelial cells) dictates the whole tissue response to radiation injury has to be replaced by the occurrence of coordinated multicellular response that may either lead to tissue recovery or to sequel development. In this context, the present review will focus on the maintenance of the radiation-induced wound healing and fi brogenic signals triggered by and through the microenvironment toward the mesenchymal cell compartment, and will highlight how sequential and sustained modifi cations in cell phenotypes will in cascade modify cell-to-cell interactions and tissue composition.	Valérie Haydont Marie-Catherine Vozenin-Brotons	2007	World Journal of Gastroenterology2007，13，19：	4
2	Induction of CTGF by TGF-β1 in normal and radiation enteritis human smooth muscle cells: Smad/Rho balance and therapeutic perspectives显示文摘	Valérie Haydont Denis Mathé Céline Bourgier Jalil Abdelali Jocelyne Aigueperse Jean Bourhis Marie-Catherine Vozenin-Brotons	2005	Radiotherapy and Oncology2005，，2：	2
3	Induction of CTGF by TGF-betal in normal and radiation enteritis human smooth muscle cells: Smad/Rho balance and therapeutic perspectives 显示文摘	Vale'rie Haydont Denis Mathe' Ce'line Bourgier	2005	Radiotherapy and Oncology2005，76，2：	1
4	Pravastatin in- hibits the Rho/CCN2/extracellular matrix cascade in hu- man fibrosis explants and improves radiation-induced in- testinal fibrosis in rats显示文摘	Haydont V Bourgier C Pocard M	2007	Clin Cancer Res2007，13，181：	1
5	Induction of CTGF by TGF-β1 in normal and radiation enteritis human smooth muscle cells: Smad/Rho balance and therapeutic perspectives显示文摘	HAYDONT V MATHE D BOURGIER C	2005	Radiother Oncol2005，76，2：	1
6	Inhibition of Rho kinase modulates radiation induced fibrogenic phenotype in intestinal smooth muscle cells through alteration of the cytoskeleton and connective tissue growth factor expres-sion显示文摘	Bourgier C Haydont V Milliat F	2005	Gut2005，54，3：	1
7	Pravastatin Inhibits the Rho/CCN2/extracellular matrix cascade in human fibrosis explants and improves radiation-induced intestinal frbrosis in rats显示文摘	Haydont V Bourgier C Pocard M	2007	Clin CancerRes2007，13，181：	1
8	Specific signals involved in the long-term maintenance of radiation-induced fibrogenic differentiation: a role for CCN2 and low concentration of TGF-betal 显示文摘	Haydont V Riser B L Aigueperse J	2008	American Journal of Physiology-Cell Physiology2008，294，：	1
9	Induction of CTGF by TGF-betal in normal and radiation enteritis human smooth muscle cells:Smad/Rho balance and therapeutic perspectives显示文摘	Haydont V Mathé D Bourgier C		0，，：	1
10	Inhibition of Rho kinase modulates radiation induced fibrogenic phenotype in intestinal smooth muscle cells through alteration of the cytoskeleton and connective tissue growth factor expression显示文摘	Bourgier C Haydont V Milliat F	2005	Gut2005，54，3：	1
11	Pravastatin inhibits the Rho/CCN2/extracellular matrix cascade in human fibrosis explants and improves radiation-induced intestinal fibrosis in rats显示文摘	Haydont V Bourgier C Pocard M	2007	Clin Cancer Res2007，13，181：	1
12	Successful mitigation of delayed intestinal radiation injury using pravastatin is not associated with acute injury improvement or tumor protection 显示文摘	Haydont V Gilliot 0 Rivera S	2007	Int J Radiat Oncol BioI Phys2007，68，5：	1
13	Rho/ROCK pathway as a molecular target for modulation of intestinal radiation-induced toxicity显示文摘	Haydont V Bourgier C Vozenin M	2007	Br J Radiol2007，9，80：	1
14	Specific signals involved in the long-term maintenance of radiation-induced fibrogenic differentiation: a role for CCN2 and low concentration of TGF-betal 显示文摘	Haydont V Riser BL Aigueperse J	2008	Am J Physiol Cell Physiol2008，294，6：	1
15	Rho/ROCK pathway as a molecular target for modulation of intestinal radiation-induced toxicity 显示文摘	Haydont V Bourgier C Vozenin MC	2007	Br J Radiol2007，80，1：	1
16	Inhibition of Rho kinase modulates radiation induced fibrogenic phenotype in intestinal smooth muscle cells through alteration of the cytoskeleton and connective tissue growth factor expression显示文摘	Bourgier C Haydont V Milliat F		0，，03：	1
17	Induction of CTGF by TGF-beta1 in normal and radiation enteritis human smooth muscle cells:Smad/Rho balance and therapeutic perspectives显示文摘	MATHE D BOURGIER C	2005	Radiother Oncol2005，76，2：	1
18	Specific signalsinvolved in the long-term maintenance of radiation-in-duced fibrogenic differentiation:a role for CCN2 and lowconcentration of TGF-beta 1显示文摘	Haydont V Riser BL Aigueperse J		American Journal of Physiology-Cell Physiology0，，：	1
19	137 Treatment of radiation enteritis with pravastatin: role of the rho pathway on CTGF expression显示文摘	Haydont V Bourgier C Fransois A	2006	Radiotherapy and Oncology2006，78，1：	1
20	对Rho激酶的抑制可通过改变细胞骨架及结缔组织生长因子的表达,调节小肠平滑肌细胞的辐射诱发纤维化显型	Bourgier C. Haydont V. Milliat F. M.- C.Vozenin- Brotons 陈云茹	2005	世界核心医学期刊文摘(胃肠病学分册)2005，0，7：	0

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