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| 1 | The effect of erythropoietin to the proliferation of cancer cell lines显示文摘对客观调查了红细胞生成素(EPO ) 是否将支持人的癌症房间线 MCF-7, Panc-1 和 HepG2 的增长以便决定 EPO 是否将结果更坏的预后到有恶性病的病人。我们由 MTT 试金检验了禁止的生长或 EPO 的宣传效果到 MCF-7, Panc-1 和 HepG2 的方法。然后,我们检验了与 EPO 并且没有由 FCM 的 EPO 有教养的那些癌症房间线的房间周期。结果在那里显示出的结果不是禁止的重要生长或到 MCF-7, Panc-1 和 HepG2 的 EPO 的宣传效果。与 EPO 有教养的那些癌症房间线的房间周期也有不重要对没有 EPO 有教养的那些癌症房间线的不同。EPO 没有提升,这被结束的结论完成到癌症房间线的增长,并且也许不结果更坏的预后到有恶性病的病人。 | Wenshan He Tao Huang Jinghua Ren Jinghui Zhang | 2008 | The Chinese-German Journal of Clinical Oncology2008，7，6： | 3 |
| 2 | Impacts of introduced Spartina alterniflora along an elevation gradient at the Jiuduansha Shoals in the Yangtze Estuary, suburban Shanghai, China显示文摘 | HE Wenshan FEAGIN R LU Jianjian | 2007 | Ecological Engineering2007，29，3： | 1 |
| 3 | Multifunctional oxygen-enriching nano-theranostics for cancer-specific magnetic resonance imaging and enhanced photodynamic/photothermal therapy显示文摘The combination of photodynamic therapy(PDT)and photothermal therapy(PTT)has attracted much interest in recent years,but non-specific distribution of photosensitizers and intrinsic tumor hypoxic microenvironment have continued to limit its therapeutic efficiency.We herein report a nano-theranostic system,denoted as Ce6-CuS/MSN@PDA@MnO2-FA NPs,which combines PDT,PTT,magnetic resonance(MR)imaging with hypoxia-relieving and tumor-targeting functionalities.Central to this design is the use of mussel-inspired polydopamine(PDA)coating to encapsulate the chlorin e6(Ce6)and copper sulfide nanoparticles(CuS NPs)loaded mesoporous silica nanoparticle(MSN)core.The PDA coating not only acts as pH sensitive gatekeeper to prevent the premature release of Ce6 under non-acidic tumor microenvironment(TME),but also facilitates post-functionalization so that hypoxia-relieving MnO2 nano-sheets and tumor-targeting ligand folic acid-PEG-thiol(FA-PEG-SH)can be decorated on the outer part of the drug system.In vitro and in vivo measurements clearly demonstrated that all these functionalities worked synergistically as expected.The system,having a low dark cytotoxicity,can be effectively internalized by 4T1 cells and decrease the cell viability to 2%upon 660 nm/808 nm laser irradiation.Tumors in 4T1 tumor-bearing mice can almost be completely destroyed in 2 weeks via combined PDT/PTT.Together with the TME-sensitive MR imaging performance demonstrated,Ce6-CuS/MSN@PDA@MnO2-FA NPs represent a multifunctional prototype which holds great potential to be developed into clinical theranostics. | Li Zhang Zhe Yang Jinghua Ren Li Ba Wenshan He Chun-Yuen Wong | 2020 | Nano Research2020，13，5： | 1 |
| 4 | Ecosystem service values and restoration in the urban Sanyang wetland of Wenzhou, China显示文摘 | Chunfu Tong Rusty A. Feagin Jianjian Lu Xiufeng Zhang Xiaojun Zhu Wei Wang Wenshan He | 2006 | Ecological Engineering2006，，： | 1 |
| 5 | The Expression of Cycl in E and Its Relationship with the Prognosis in Patients with Breast Cancer 显示文摘 | He Wenshan Huang tao Wang Haijiu | 2004 | J Huazhong Univ Sci Tech ( Health Sci)2004，33，： | 1 |
| 6 | Expression of Cyclin E and Its Relationship with the Prognosis of Patients with Breast Cancer显示文摘客观:在有病人的预后的乳癌纸巾和它与乳癌的关系调查 cyclin E 的表示。方法:表示 ofcyclin E, HER-2/neu, nm23-H1 和肌动朊被免疫在 80 乳癌纸巾和 18 良性的胸肿瘤纸巾检测组织化学的方法。在 cyclin E 和留下的基因或有乳癌的病人的临床的数据之间的关系被分析。结果:过去在恶意的纸巾的 cyclin E 的表示率在良性瘤纸巾显然比那高(P <
0.01 ) 。过去在疾病的以后的舞台的 cyclin E 的表示在疾病的早舞台比那高(P <
0.05 ) 。cyclin E 在的表示嗯积极纸巾是比那低的在嗯否定纸巾(P <
0.05 ) 。在 PR 积极纸巾 andPR negative 纸巾的 cyclin E 的表示没有有效差量(P >
0.05 ) 。cyclin E inHER-2/neu 的表示积极纸巾在 HER-2/neu 否定纸巾比那高(P <
0.05 ) 。并且 cyclin E 在的表示嗯, PR 和 HER-2/neu 所有积极纸巾高得多(P <
0.01 ) 。在在 nm23-H1 之间的 cyclin E 的表示没有有效差量积极纸巾和 nm23-H1 negative 纸巾(P >
0.05 ) 。在积极的肌动朊和连续分发纸巾的 cyclin E 的表示在肌动朊 negative 或不连续的分发纸巾(P< 0.05 ) 是比那低的。结论:cyclin E 的表示与乳癌有强壮的关联到病人的预后。 | HE Wenshan HUANG Tao WANG Haijiu | 2006 | The Chinese-German Journal of Clinical Oncology2006，5，4： | 1 |
| 7 | Minimum component high frequency Gm-C wavelet filters based on Maclaurin series and multiple loop feedback显示文摘 | ZHAO Wenshan SUN Yichuang HE Yigang | 2010 | Electronics Letters2010，46，1： | 1 |
| 8 | Outlook for Hydrogen Fuel Cell Ships显示文摘Hydrogen fuel cell is a power generation device that converts.the chemical energy of oxygen and hydrogen into electric energy.The product is waler,which has the characteristics of high eficiency,no pollution and low equipment operation noise.The adoption of hydrogen fuel cell propulsion scheme is one of the important measures to meel the requirements of the new environmental porolection regulations. | Huang He Xu Wenshan Li Mingmin | 2022 | 船舶经济贸易2022，，4： | 0 |
| 9 | CONVERGENCE RATE FOR ITERATES OF q-BERNSTEIN POLYNOMIALS显示文摘最近, q-Bernstein 多项式被很多个作者强烈地调查了。他们为 q ≠ 1, q-Bernstein 多项式许多有趣的性质拥有的结果表演。在这篇论文,集中率为两个 q-Bernstein 多项式重申;他们的布尔和被估计。而且,浸透 { B n (·, q n )} 什么时候 n →∞;B n 的集中率(f, q; x ) 什么时候 f ∈ C [n1 ][0,1 ] , q →∞也被介绍。 | Xueyan Xiang Qian He Wenshan Yang | 2007 | Analysis in Theory and Applications2007，23，3： | 0 |
| 10 | Cyclin E RNAi对乳腺癌细胞株生长抑制作用的实验研究(英文)显示文摘Objective:The aim of the study was to investigate the biological character change of breast cancer cell line by inhibiting the Cyclin E expressing level.Methods:Human breast cancer cell line MCF-7 was transfected by Cyclin E siRNA vector pEGFP/CCNE2.SiRNA-induced silencing of Cyclin E was determined at RNA level and protein level,respectively.The proliferation of MCF-7 and its sensitivity to chemical therapy were measured by CCK-8 assay,while cell cycle was measured by flow cytometry(FCM).Results:The plasmid could reduce the expression of Cyclin E.The proliferation ability of MCF-7 was decreased and the sensitivity to chemical therapy was enhanced according to the inhibition of Cyclin E expression.The transfected MCF-7 was arrested at G1 phase of cell cycle.Conclusion:The inhibition of Cyclin E can decrease the breast cancer cell's growth,increase its sensation to chemical therapy and slow down its cell cycle.The Cyclin E siRNA may provide us a practical tool for further study on the gene therapy to breast cancers. | Xueqin Li Na Shen Wenshan He Tao Huang | 2011 | The Chinese-German Journal of Clinical Oncology2011，10，9： | 0 |
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