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| 1 | Interferon-gamma sensitizes osteosarcoma cells to fas-induced apoptosis by up-regulating fas receptors and caspase-8显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 2 | : Interferon-gamma sensitizesosteosarcoma cells to fas-induced apoptosis by up-regulating fasreceptors and caspase-8 显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 3 | Systemic levels following PGE1 inhalation in neonatal hypoxemic respiratory failure 显示文摘 | Sood B G Glibetic M Aranda J V | 2006 | Acta Paediat2006，95，： | 1 |
| 4 | Interferon-gamma sensitizes osteosareoma cells to Fas-induced apoptosis by up-regulating Fas receptors and caspase-8 显示文摘 | INABA H GLIBETIC M BUCK S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 5 | PGEJTXB2 irnbalance in neonatal hypoxemic respiratory failure显示文摘 | Sood BG Delaney-Black V Glibetic M et aI | 2007 | Acta Paediatrica2007，96，5： | 1 |
| 6 | Effect of glutamine on the induction of nitrate reductase显示文摘 | Li X Z Larson D E Glibetic M | 1995 | Physiologia Plantarum1995，93，： | 1 |
| 7 | Interferon-gamma sensitizes osteosarcoma cells to Fas-induced apoptosis by up-regulating Fas receptors and caspase-8 显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 8 | : Interferon-gamma sensitizesosteosarcoma cells to fas-induced apoptosis by up-regulating fasreceptors and caspase-8 显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 9 | Interferon-gamma sensitizes osteosarcoma cells to Fas-induced apoptosis by up-regulating Fas receptors and caspase-8显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 10 | : Interferon-gamma sensitizesosteosarcoma cells to fas-induced apoptosis by up-regulating fasreceptors and caspase-8 显示文摘 | Inaba H Glibetic M Buck S | 2004 | Pediatr Blood Cancer2004，43，7： | 1 |
| 11 | PGE(2) /TXB2 imbalance in neonatal hypoxemic respiratory failure显示文摘 | Sood BG Delaney-Black V Glibetic M | | 0，，05： | 1 |
| 12 | Systemic levels following PGEI inhalation in neonatal hypoxemic respiratory failure显示文摘 | SOOD BG GLIBETIC M ARANDA iV | 2006 | Acta Paediat2006，95，9： | 1 |
| 13 | Heat stress protects against ung injury in the neutropenic, endotoxemic rat显示文摘 | Heidemann SM Glibetic M | 2005 | Inflam- aation2005，29，1： | 1 |
| 14 | Preparatory training attenuates drastic response of the insulin-like growth factor binding protein 1 at the point of maximal oxygen consumption in handball players显示文摘Background:Intensive exercise changes physiological need for glucose and several biochemical pathways responsible for its metabolism response.Among them are those which involve insulin,insulin-like growth factor(IGF-1),and IGF-binding proteins(IGFBPs).Different types and degrees of exercise,as well as an athlete's fitness,may induce a range of responses regarding concentrations and time needed for the alteration.The idea of the work was to find out whether and how insulin/IGF axis responds to additional physical activity in the already trained subjects and if so,is the adaptation potentially beneficial from the aspect of metabolic control.Methods:The effect of 4-week intensive training on campus(preparatory training) on the levels of insulin,IGF-1,and IGFBPs during maximal progressive exercise test(MPET) on a treadmill was compared to the results obtained during MPET conducted after a regular training season of a female elite handball team(n = 17,age:17 ± 1 years,height:171 ± 8 cm,weight:65 ± 8 kg,body mass index:22 ± 1 kg/m^2 at the beginning of the study;there were no significant changes at the end).Serum samples were obtained from players immediately before the test(basal),at the end of the test after reaching the point of maximal oxygen consumption(VO_(2max)),and after recovery.Results:The concentration of insulin decreased at VO_(2max),but remained higher in players after preparatory training(12.2 ± 2.5 m U/L vs.8.9 ± 4.4 m U/L,p = 0.049).The level of IGFBP-1 decreased in players at VO_(2max) in either case of training,but it remained much higher in tests performed after the preparatory regime than before(p = 0.029).Concentrations of IGF-1,IGFBP-2,-3,and-4 did not change significantly.Conclusion:The inverse relation between insulin and IGFBP-1 was lost during MPET,as these 2 molecules changed in the same direction.The results obtained suggest less severe stress-induced depression of insulin and IGFBP-1 after preparatory training.But another metabolic mechanism cannot be excluded,and that is potentially impaired insulin sensitivity resulting in higher level of IGFBP-1. | Olgica Nedic Milos Sunderic Goran Miljus Zoran Valdevit Vladimir Jakovljevic Marija Glibetic Vesna Vucic | 2017 | Journal of Sport and Health Science2017，6，3： | 0 |
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