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| 1 | Flt3 mutations and leukaemia显示文摘 | Kottaridis PD Gale RE Linch DC | 2003 | Br J Haematol2003，122，4： | 1 |
| 2 | Characterization of Noncovalent Complexes Formed Between Minor Groove Binding Molecules and Duplex DNA by Electrospray Ionization-Mass Spectrometry 显示文摘 | Gale DC Smith RD | 1995 | J Am Soc mass Spectrom1995，6，12： | 1 |
| 3 | PI3-kinase/Akt is constitutively active in primary acute myeloid 1 eukaemia cells and regulates survival and chemoresis-tance via NF-kappaB, Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 4 | PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 5 | p13-kinase/Akt is constitutivelyactive in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB,Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 6 | Characterization of noncovalent complexes formed between minor groove binding molecules and duplex DNA by electrospray ionization mass spectrometry显示文摘 | Gale DC Smith RD | 1995 | J Am Soc Mass Spectrom1995，6，： | 1 |
| 7 | FLT3 mutations and leukaemia显示文摘 | Kottaridis PD Gale RE Linch DC | 2003 | BrJ Haematol2003，122，4： | 1 |
| 8 | FLT3 mutations and leukemia 显示文摘 | Kottardis PD Gale RE Linch DC | 2003 | BrJ Haematol2003，122，4： | 1 |
| 9 | Flt3 mutations and leukemia显示文摘 | Kottardis PD Gale RE Linch DC | 2003 | Br J Haematol2003，122，4： | 1 |
| 10 | PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 11 | Flt3 mutations and leukemia 显示文摘 | Kottaridis PD Gale RE Linch DC | 2003 | Br J Haematol2003，122，4： | 1 |
| 12 | PI3 - kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF - kappaB, Mapkinase and p53 pathways 显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 13 | PI3-kinase/Akt is constitutively active in primary acute myeloid leukemia cells and regulates survival and chemoresistanee via NF-kappaB, MAPK and p53 pathways 显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 14 | Pallidal Stimulation for Dystonia in Pantothenate Kinaseassoeiated Neurodegeneration 显示文摘 | Shields DC Sharma N Gale JT | 2007 | Pediatr Neurol2007，37，6： | 1 |
| 15 | PI32kinase /Akt isconstitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB,Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | | 0，，： | 1 |
| 16 | Flt3 mutation and leukemia显示文摘 | Kottlaridis PD Gale RE Linch DC | 2003 | Br J Haematol2003，122，4： | 1 |
| 17 | PI3-ki- nase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘 | Grandage VL Gale RE Linch DC | 2005 | Leukemia2005，19，4： | 1 |
| 18 | 影响血压的膳食纤维类型显示文摘该研究旨在确定不同类型膳食纤维对收缩压和舒张压的影响。方法:对采用随机效应模型的随机对照试验文献进行系统回顾和荟萃分析,纳入研究标准包括:在1990年1月1日和2013年12月1日之间进行的,至少为期6周的随机对照试验,对分离出的纤维或纤维丰富的膳食对血压的影响进行分析,并与安慰剂对照组进行比较。结果:共纳入18项满足人选标准的报道纤维摄入和血压关系的研究进入荟萃分析。 | Evans CE Greenwood DC Threapleton DE Cleghorn CL Nykjaer C Woodhead CE Gale CP Burley VJ 刘莉 叶鹏 | 2015 | 中华高血压杂志2015，23，3： | 0 |
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