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29篇 您的检索式：作者名="Fonseca MI"

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	题名	作者	年代	出处	被引量
1	Complement C3 and C4 expression in Clq sufficient and deficient mouse models of Alzheimer's disease显示文摘	Zhou J Fonseca MI Pisalyaput K et aI	2008	J Neurochem2008，106，5：	1
2	Complement C3 and C4 expression in Clq sufficient and deficient mouse models of Alzheimer's disease显示文摘	Zhou J Fonseca MI Pisalyaput K		0，，05：	1
3	Adipose tissue as an endocrine organ: from theory to practice显示文摘	Fonseca - Alaniz MH Takada J Alonso - Vale MI	2007	J Pediatr ( Rio J)2007，83，5：	1
4	Prognostic factors associated with in-hospital mortality in patients with spontaneous bacterialperitonitis显示文摘	Musskopf MI Fonseca FP Gass J		0，，06：	1
5	Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of alzheimer's disease显示文摘	Fonseca MI Ager RR Chu SH		0，，02：	1
6	Microglial C5aR (CD88) expression correlates with amyloid-β deposition in murine models of Alzheimer's disease显示文摘	Ager RR Fonseca MI Chu SH		0，，02：	1
7	Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease显示文摘	Fonseca MI Chu SH Berci AM		0，，01：	1
8	Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease显示文摘	Zhou J Fonseca MI Pisalyaput K		0，，05：	1
9	Distribution of serotonin 2A ,2C and 3 receptor mRNA in spinal cord and medulla oblongata显示文摘	Fonseca MI Ni YG Dunning DD Miledi R	2001	Brain Res Mol Brain Res2001，89，：	1
10	Structral and functional evidence for microglial expression of C1q receptor that enhances phagocytosis显示文摘	Webster SD Park M Fonseca MI	2000	J Leukoc Biol2000，67，1：	1
11	Absence of C1 q leads to less neuropathology in transgenic mouse models of Alzheimer's disease显示文摘	Fonseca MI Zhou J Botto M		0，，29：	1
12	Complement activa- tion fragment C5a receptors, CD88 and C5L2, are associated with neurofibrillary pathology显示文摘	Fonseca MI McGuire SO Counts SE	2013	J Neuroinflammation2013，10，：	1
13	Complement C3 and C4 expression in C1q suffcient and deficient mouse models of Alzheimer's disease显示文摘	Zhou J Fonseca MI Pisalyaput K		0，，：	1
14	Absence of Clq leads to less neu- rophathology in transgenic mouse models of Alzheimer's disease 显示文摘	Fonseca MI Zhou J Botto M	2004	J Neurosci2004，24，29：	1
15	Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of Alzheimer's disease显示文摘	Fonseca MI Ager RR Chu SH	2009	J Immunol2009，183，2：	1
16	Complement activation frag- ment C5a receptors, CD88 and C5L2, are associated with neurofibrillary pathology显示文摘	Fonseca MI McGuire SO Counts SE	2013	J Neuroinflammation2013，10，：	1
17	Complement C3 and C4 expression in Clq sufficient and deficient mouse modelss of Alzheimer's Disease显示文摘	Zhou J Fonseca MI Pisalyaput K	2008	J Neurochem2008，106，：	1
18	Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease显示文摘	Zhou J Fonseca MI Pisalyaput K	2008	J Neurochem2008，106，5：	1
19	Neuronal localization of C1q in preclinical Alzheimer's disease显示文摘	Fonseca MI Kawas CH Troncoso JC	2004	Neurobiol Dis2004，15，1：	1
20	Structural and functional evidence for microglial expression of ClqR(P),the Clq receptor that enhances phagocytosis显示文摘	Webster SD Park M Fonseca MI	2000	J LeukocBiol2000，67，1：	1

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