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19篇 您的检索式：作者名="Fluckey"

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		题名	作者	年代	出处	被引量
	1	Resistance trainingusing eccentric overload induces early adaptations in skeletal muscle size 显示文摘	Norrbrand L Fluckey JD Pozzo M	2008	Eur J Appl Physio12008，102，3：	1
	2	Insulin-facilitated increase of muscle protein synthesis after resistance exercise involves a MAP kinase pathway 显示文摘	Fluckey JD Knox M Smith L	2006	Am J Physiol Endocrinol Metad2006，290，6：	1
	3	Regulators of blood and lipoproteins? PPARγ and AMPK, induced by exercise, are correlated with lipids and lipoproteins in overweight/obese men and women显示文摘	Greene N P Fluckey J D Lambert B L	2012	Am J Physiol Endocrinol Metab2012，303，9：	1
	4	A rat resistance exercise regimen attenuates losses of musculoskeletal mass during hindlimb suspension显示文摘	Fluckey JD Dupont-Versteegden EE Montague DC		0，，04：	1
	5	Insulin-facilitated increase of muscle protein synthesis after resistance exercise involves a MAP kinase pathway显示文摘	Fluckey J D Knox M Smith L	2006	Am J Physiol Endocrinol Metad2006，290，6：	1
	6	Regula- tors of blood lipids and lipoproteins? PPARdelta and AMPK, induced by exercise, are correlated with lipids and lipoproteins in overweight/obese men and women显示文摘	GREENE N P FLUCKEY J D LAMBERT B S	2012	Am J Phys Endocri- nol Metab2012，303，10：	1
	7	A rat resistance exercise regimen attenuates losses of musculosketetal mass during hindlimb suspension 显示文摘	Fluckey JD Dupont-Versteegden EE Montague DC	2002	Acta Physiol Scand2002，176，：	1
	8	Effect of flywheel-based resistance exercise on processes contributing to muscle atrophy during unloading in adult rats 显示文摘	Dupont-Versteegden EE FLuckey JD Knox M	2006	J Appl Physiol2006，101，：	1
	9	Augraented insulin action on rates of protein synthesis following resistance exercise in rats 显示文摘	Fluckey JD Jefferson LS Vary TC Farrell PA	1996	Am J Physiol1996，270，：	1
	10	Effect of resistance exercise training on cortical and cancellous bone in mature male rats 显示文摘	Westerhnd KC Fluckey JD Gordon SE	1998	J Appl Physiol1998，84，：	1
	11	A rat resistance exercise regimen attenuates losses of musculoskeletal mass during hindlimb suspension 显示文摘	Fluckey JD Dupont-Versteegden EE Montague DC	2002	Acta Physiol Scand2002，176，4：	1
	12	Effect of resistance exercise training on cortical and cancellous bone in mature male rats显示文摘	Westerlind KC Fluckey JD Gordon SE	1998	J Appl Physiol1998，84，2：	1
	13	Skeletal muscle PGF(2)(alpha) and PGE(2) in response to eccentric resistance exercise:influence of ibuprofen acetaminophen显示文摘	Trappe TA Fluckey JD White F		0，，10：	1
	14	Insulin facilitated increase of muscle protein synthesis after resistance exercise involves a MAP kinase pathway 显示文摘	Fluckey J D Knox M Smith L	2006	Am J Physiol Endocrinol Metad2006，290，6：	1
	15	A rat resistance exercise regimen attenuates losses of musculoskeletal mass during hindlimb suspension 显示文摘	Fluckey JD Dupont - Versteegden EE Montague DC	2002	Acta Physiol Scand2002，176，9：	1
	16	Skeletal muscle PGF (2 alpha) and PGE (2) in response to eccentric resistance exer- cise: Influence of ibuprofen and acetaminophen显示文摘	Trappe TA Fluckey JD White F	2001	Journal of Clin- ical Endocrinology & Metabolism2001，86，10：	1
	17	Resistance training using eccentric overload induces early adaptations in skeletal muscle size显示文摘	Lena Norrbrand James D. Fluckey Marco Pozzo Per A. Tesch	2008	European Journal of Applied Physiology2008，，3：	1
	18	Active involvement of PKC for insulin-mediated rates of muscle protein synthesis in Zucker rats显示文摘	Fluckey JD Cortright RN Tapscott E	2004	Am J Physiol Endocrinol Metab2004，286，5：	1
	19	Culprits or consequences:Understanding the metabolic dysregulation of muscle in diabetes显示文摘The prevalence of type 2 diabetes(T2D)continues to rise despite the amount of research dedicated to finding the culprits of this debilitating disease.Skeletal muscle is arguably the most important contributor to glucose disposal making it a clear target in insulin resistance and T2D research.Within skeletal muscle there is a clear link to metabolic dysregulation during the progression of T2D but the determination of culprits vs consequences of the disease has been elusive.Emerging evidence in skeletal muscle implicates influential cross talk between a key anabolic regulatory protein,the mammalian target of rapamycin(mTOR)and its associated complexes(mTORC1 and mTORC2),and the well-described canonical signaling for insulin-stimulated glucose uptake.This new understanding of cellular signaling crosstalk has blurred the lines of what is a culprit and what is a consequence with regard to insulin resistance.Here,we briefly review the most recent understanding of insulin signaling in skeletal muscle,and how anabolic responses favoring anabolism directly impact cellular glucose disposal.This review highlights key cross-over interactions between protein and glucose regulatory pathways and the implications this may have for the design of new therapeutic targets for the control of glucoregulatory function in skeletal muscle.	Colleen L O'Reilly Selina Uranga James D Fluckey	2021	World Journal of Biological Chemistry2021，12，5：	0

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