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49篇 您的检索式:作者名="Figg M"
    题名 作者 年代 出处 被引量
1Sirtuin 6抑制平滑肌细胞衰老,减少动脉粥样硬化显示文摘血管平滑肌细胞(vascular smooth muscle cell, VSMC)衰老促进动脉粥样硬化及斑块不稳定性,部分是通过脂质介导的DNA氧化损伤和端粒功能障碍起作用。Sirtuin 6(SIRT6)是一种核去乙酰化酶,参与DNA损伤应答信号转导、炎症反应和代谢。然而,其在调节VSMC衰老和动脉粥样硬化中的作用尚不清楚。陈嘉睿(译) 练桂丽(审校) Grootaert M Finigan A Figg N Uryga AK Bennett M 2021中华高血压杂志2021,29,3:21
2Hyporinducibility of hypoxia responsive genes without p53/p212 dependent checkpoint in aggressive prostate cancer 显示文摘Salnikow K Costa M Figg WD 2000Cancer Res2000,60,20:1
3Hyperinducibility of hypoxia- responsive genes without p53/p21-dependent checkpoint in aggressive prostate cancer 显示文摘Salnikow K Costa M Figg WD 2000Cancer Res2000,60,20:1
4Drosophila Kruppel protein is a transcriptional repressor显示文摘LICHT J D GROSSEL M J FIGGE J 1990Nature1990,346,:1
5Effect of high concentrations dietary vitamin E and meat stability of broiler chicks at 7 weeps of age显示文摘Barto I Figg M 1992Brit Poult Sci1992,33,:1
6Vascular smooth muscle cell apoptosis induces interleukin-1-directed inflammation:effects of hyperlip-idemia-mediated inhibition of phagocytosis显示文摘Clarke M C Talib S Figg N L 2010Circ Res2010,106,2:1
7Hyperinducibility of hypoxia-responsive genes without p53/p21-dependent checkpoint in aggressive prostate cancer显示文摘Salnikow K Costa M Figg WD Cancer Res0,60,20:1
8Hyperinducibility of hypoxiaresponsive genes without p53/p21-dependent checkpoint in aggressive prostate cancer显示文摘Salnikow K Costa M Figg WD 2000Cancer Res2000,60,20:1
9Vascular smooth muscle cell apop-tosis induces interleukin-1 directed inflammation:effects of hyperlipi-demia mediated inhibition of phagocytosis显示文摘Clarke M C Talib S Figg N L 2010Circ Res2010,106,2:1
10Angiogenesis inhibitors: current strategies and future prospects 显示文摘COOK K M FIGG W D 2010CA Cancer J Clin2010,60,4:1
11Micellar paclitaxel improves severe psoriasis in a prospective phase Ⅱ pilot study显示文摘Ehrlich A Booher S Becerra Y Borris D L Figg W D Turner M L 0,,:1
12Thalidomide显示文摘FRANKS M E MACPHERSON G R FIGG W D 2004Lancet2004,263,9423:1
13Apoptosis of vascular smooth muscle ceils induces features of plaque vulnerability in atheroscle- rosis 显示文摘Clarke M Figg N Maguire J 2006Nature Med2006,12,9:1
14Neurite outgrowth triggered by the cell adhesion molecule L1 requires activation and inactivation of the cytoskeletal protein cofilin显示文摘FIGGE C LOERS G SCHACHNER M 2012Mol Cell Neurosci2012,49,2:1
15Apoptosis of vas- cular smooth muscle ceils induces features of plaque vulnerability in atherosclerosis显示文摘CLARKE M C FIGG N MAGUIRE J J 2006Nat Med2006,12,9:1
16Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis显示文摘CLARKE M C FIGG N MAGUIRE J J 0,,09:1
17Chronic apoptosis of vascular smooth muscle cells accelerates atherosclerosis and promotes calcification and medial degeneration显示文摘CLARKE M C LITTLEWOOD T D FIGG N 0,,12:1
18Thalidomide an antiangiogenic agent with clinical activity in cancer显示文摘NG SS BROWN M FIGG WD 2002Biomed Pharmacother2002,56,4:1
19Smooth muscle cell apoptosis promotes vessel remodeling and repair via activation of cell migration,proliferation,and collagen synthesis显示文摘YU H CLARKE M C FIGG N 0,,11:1
20A doub- le-blind randomized crossover study of oral thalidomide versus placebo for androgen dependent prostate cancer treated with intermittent androgen ablation显示文摘FIGG W D HUSSAIN M H GULLEY J L 2009J Urol2009,181,3:1
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