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21篇 您的检索式:作者名="Ferguson DO"
    题名 作者 年代 出处 被引量
1The nonhomologous end-joining pathway of DNA repair for genomic stability and the suppression of translocations显示文摘Ferguson DO 2000Proc Natl Acad Sci USA2000,97,:1
2DNA double strand break repair and chromosomal translocation:lessons from animal models 显示文摘Ferguson DO Alt FW 2001Oncogene2001,20,40:1
3Unrepaired DNA breaks in p53-deficient cells lead to oncogenic gene amplification subsequent to translocations 显示文摘Zhu C Mills KD Ferguson DO 2002Cell2002,109,7:1
4Unrepaired DNA breaks in p53 deficient cells lead to oncogenic gene amplification subsequent to translocations显示文摘Zhu C Mills KD Ferguson DO 2002Cell2002,109,7:1
5The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocadons显示文摘Ferguson DO Dekiguchi JM Chang S 2000Proc Natl Acad Sci U S A2000,97,12:1
6Dysregulated TCL1 requires the germinal center and genome instability for mature B-cell transformation 显示文摘Shen RR Ferguson DO Renard M 2006Blood2006,108,6:1
7Histone H2AX:a dosage-dependent suppressor of oncogenic translocations and tumors显示文摘Bassing CH Suh H Ferguson DO 2003Cell2003,114,:1
8Proapoptotic BID is required for myeloid homeostasis and tumor suppression显示文摘Zinkel SS Ong CC Ferguson DO 2003Genes Dev2003,17,:1
9DNA double strand and chromosomal translocation:Lessons from animal models显示文摘Ferguson DO Alt FW 2001Oncogene2001,20,40:1
10The role of DNA breaks in genomic instability and tumorigenesis显示文摘 FERGUSON DO ALT F W 2003Immunol Rev2003,194,:1
11Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching显示文摘Wang JH Ferguson DO Xie W 2008J Exp Med2008,205,13:1
12Multiple roles for MRE11 at uncapped telomeres 显示文摘Deng YB Guo XL Ferguson DO 2009Nature2009,460,7257:1
13DNA double strand break repair chromosome translocation: lessons from animal models显示文摘 Alt FW 2000Oncogene2000,20,:1
14Multiple roles for MREll at uncapped telomeres显示文摘Deng YB Guo XL Ferguson DO 2009Nature2009,460,7257:1
15The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations显示文摘Ferguson DO Sekiguchi JM Chang S 2000Proc Natl Acad Sci USA2000,97,12:1
16Proapoptotic BID is required for myeloid homeostasis and tumor suppression显示文摘Zinkel SS Ong CC Ferguson DO 2003Genes Dev2003,17,2:1
17Unrepaired DNA breaks in p53 -deficient cells lead to oncogenih gene amplification subsequent to translocations 显示文摘Zhu C Mills KD Ferguson DO etal 2002Cell2002,109,7:1
18Histone H2AX:A dosage-dependent suppressor of oncogenic translocationsand tumors 显示文摘Bassing CH Suh H Ferguson DO 2003Cell2003,114,3:1
19The interplay between nonhomologous end-joining and cell cycle checkpoint factors in development, genomic stability, and tumorigenesis 显示文摘Ferguson DO Sekiguchi JM Frank KM 2000Cold Spring Harb Symp Quant Biol2000,65,:1
20Proapoptotic BID is required for myeloid homeostasis and tumor suppression显示文摘 Ong CC Ferguson DO 2003Genes Dev2003,17,:1
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