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| 1 | Endoscopic management of esophageal stenosis in children:New and traditional treatments显示文摘Post-esophageal atresia anastomotic strictures and postcorrosive esophagitis are the most frequent types of cicatricial esophageal stricture. Congenital esophageal stenosis has been reported to be a rare but typical disease in children; other pediatric conditions are peptic, eosinophilic esophagitis and dystrophic recessive epidermolysis bullosa strictures. The conservative treatment of esophageal stenosis and strictures(ES) rather than surgery is a well-known strategy for children. Before planning esophageal dilation, the esophageal morphology should be assessed in detail for its length, aspect, number and level, and different conservative strategies should be chosen accordingly. Endoscopic dilators and techniques that involve different adjuvant treatment strategies have been reported and depend on the stricture's etiology, the availability of different tools and the operator's experience and preferences. Balloon and semirigid dilators are the most frequently used tools. No high-quality studies have reported on the differences in the efficacies and rates of complications associated with these two types of dilators. There is no consensus in the literature regarding the frequency of dilations or the diameter that should be achieved. The use of adjuvant treatments has been reported in cases of recalcitrant stenosis or strictures with evidence of dysphagic symptoms. Corticosteroids(either systemically or locally injected), the local application of mitomycin C, diathermy and laser ES sectioning have been reported. Some authors have suggested that stenting can reduce both the number of dilations and the treatment length. In many cases, this strategy is effective when either metallic or plastic stents are utilized. Treatment complications, such esophageal perforations, can be conservatively managed, considering surgery only in cases with severe pleural cavity involvement. In cases of stricture relapse,even if such relapses occur following the execution of well-conducted conservative strategies, surgical stricture resection and anastomosis or esophageal substitution are the only remaining options. | Luigi Dall rsquo Oglio Tamara Caldaro Francesca Foschia Simona Faraci Giovanni Federici di Abriola Francesca Rea Erminia Romeo Filippo Torroni Giulia Angelino Paola De Angelis | 2016 | World Journal of Gastrointestinal Endoscopy2016,8,4: | 16 |
| 2 | ConservativeapproachinPeutz-JeghersSyndrome:Single-balloon enteroscopyandsmallbowelpolypectomy显示文摘AIM: To assess the usefulness of the balloon assisted enteroscopy in preventing surgical intervention in pa-tients with Peutz-Jeghers syndrome (PJS) having a small bowel large polyps. METHODS: Seven consecutive asymptomatic pts(age 15-38 years) with PJS have been collected; six under-went polypectomy using single balloon enteroscopy(Olympus SIF Q180) with antegrade approach using push and pull technique. SBE system consists of the SIF-Q180 enteroscope, an overtube balloon control unit(OBCU Olympus Balloon Control Unit) and a dispos-able silicone splinting tube with balloon(ST-SB1). All procedures were performed under general anesthesia. Previously all pts received wireless capsule endos-copy(WCE). Prophylactic polypectomy was reservedmainly in pts who had polyps > 15 mm in diameter. The balloon is inflated and deflated by a balloon control unit with a safety pressure setting range from-6.0 kPa to +5.4 kPa. Informed consent has been obtained from pts or parents for each procedure.RESULTS: Six pts underwent polypectomy of small bowel polyps; in 5 pts a large polyp > 15 mm(range 20-50 mm in diameter) was resected; in 1 patient with WCE negative, SBE was performed for previous surgi-cal resection of gastrointestinal stromal tumors. In 2 pts endoscopic clips were placed due to a polypectomy. No surgical complication have been reported. SBE with resection of small bowel large polyps in PJS pts was useful to avoid gastrointestinal bleeding and emergency laparotomy due to intestinal intussuscep-tions. No gastrointestinal tumors were found in sub-sequent enteroscopic surveillance in all seven pts. In order surveillance, all pts received WCE, upper en-doscopy, ileocolonoscopy every 2 years. No pts had extraintestinal malignant lesions. SBE was performed when WCE was positive for significant polyps(> 15 mm).CONCLUSION: The effective of prophylactic polyp-ectomy of small bowel large polyps(> 15 mm) could be the first line treatment for conservative approach in management of PJS patients. | Torroni F Romeo E Rea F De Angelis P Foschia F Faraci S Federici di Abriola G Contini AC Caldaro T Dall’Oglio L | 2014 | World Journal of Gastrointestinal Endoscopy2014,6,7: | 7 |
| 3 | Hyperhomocysteinemia, Oxidative Stress, and Cerebral Vascular Dysfunction显示文摘 | Frank M. Faraci Steven R. Lentz | 2004 | Stroke: Journal of the American Heart Association2004,,2: | 4 |
| 4 | Arthrogryposis, renal dysfunction and cholestasis syndrome: Report of five patients from three Italian families显示文摘 | M. Rocco F. Callea B. Pollice M. Faraci F. Campiani C. Borrone | 1995 | European Journal of Pediatrics1995,,10: | 2 |
| 5 | 显示文摘 | Ram M K Adami M Faraci P | 2000 | Polymer2000,41,: | 1 |
| 6 | Cerebral vascular dysfunction during hypercholesterolemia显示文摘 | Kitayama J Faraci MR Lentz SR | 2007 | Stroke2007,38,7: | 1 |
| 7 | Severe neurologic complications after hematopoietic stem cell transplantation in children显示文摘 | Faraci M Lanino E Dini G | 2002 | Neurology2002,2459,12: | 1 |
| 8 | Multiplicity theorems for discrete boundary value problems显示文摘 | Faraci F Iannizzotto A | 2007 | Aequationes Math2007,74,: | 1 |
| 9 | Nitric oxide and cerebral circulation显示文摘 | Faraci FM Brian JE | | 0,,03: | 1 |
| 10 | Gene transfer of calcitonin gene-related peptide to cerebral arteries显示文摘 | Toyoda K Faraci FM Russo AF | 2000 | Am J Physiol Heart Circ Physio12000,278,2: | 1 |
| 11 | Gene transfer of calcitonin gene-related peptide prevents vasoconstriction after subarachnoid hemorrhage显示文摘 | Toyoda K Faraci FM Watanabe Y | 2000 | Circ Res2000,87,9: | 1 |
| 12 | Hyperhomocysteinemia:a million way to lose control 显示文摘 | Faraci FM | 2003 | Arterioscl Thromb Vasc Biol2003,23,: | 1 |
| 13 | Subcloning, expression, and purifi-cation of the enterobactin biosynthetic enzyme 2, 3-dihydroxybenzo-ate-AMP ligase: demonstration of enzyme-bound ( 2, 3-dihydroxybe-nzoyl ) adenylate product 显示文摘 | Rusnak F Faraci WS Walsh CT | 1989 | Biochemistry1989,28,17: | 1 |
| 14 | Dilatation of the basilar artery in response to selective activation of endothelin B receptors in vivo 显示文摘 | KITAZONO T HEISTAD DD FARACI FM | 1995 | J Pharmco Exp Ther1995,273,1: | 1 |
| 15 | hyperhomocysteinnemia,oxidative stress,andcerebral dysfunction显示文摘 | Faraci FM Lentz SR | | 0,,03: | 1 |
| 16 | Hyperhomocysteinemia, oxidative stress, and cerebral vascular dysfunction 显示文摘 | Faraci FM Lentz SR | 2004 | Stroke2004,35,2: | 1 |
| 17 | Cerebral vascular dysfunction in TallyHo mice:a new model of Type II diabetes显示文摘 | Didion S P Lynch C M Faraci F M | 2007 | Am J Physiol Heart Circ Physiol2007,292,3: | 1 |
| 18 | Subarachnoid hemorrhage:what happens to the cerebral arteries?显示文摘 | Sobey CG Faraci FM | 1998 | Clin Exp Pharmacol Physiol1998,25,11: | 1 |
| 19 | Functional activity of Ca^2+-dependent K+ channels is increased in basilar artery during chronic hypertension 显示文摘 | PATERNO R HEISTAD DD FARACI FM | 1997 | ^ Am J Physiol1997,272,32: | 1 |
| 20 | Nitri coxide and the cerebralcirculation显示文摘 | FARACI FM BRIAN JE JR | 1994 | Stroke1994,25,: | 1 |