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14篇 您的检索式:作者名="Fantauzzi"
    题名 作者 年代 出处 被引量
1Galectin-3 ablation protects mice from diet-induced NASH: A major scavenging role for galectin-3 in liver显示文摘Carla Iacobini Stefano Menini Carlo Ricci Claudia Blasetti Fantauzzi Angela Scipioni Laura Salvi Samantha Cordone Francesca Delucchi Matteo Serino Massimo Federici Flavia Pricci Giuseppe Pugliese 2010Journal of Hepatology2010,,5:2
2XPS and XAES analysis of the chemical state of copper, arsenic and sulphur in natural and synthetic enargite 显示文摘Fantauzzi M Atzei D Elsener B Lattanzi P Rossi A 2006Surface and Interface Analysis2006,38,5:1
3An XPS analytical approach for elucidating the microbially mediated enargite oxidative dissolution显示文摘Fantauzzi M Rossi G Elsener B 2009Analytical and Bioanalytical Chemistry2009,393,:1
4Galectin-3 ablation protects mice from diet-induced NASH: A major scavenging role for galectin-3 in liver显示文摘Carla Iacobini Stefano Menini Carlo Ricci Claudia Blasetti Fantauzzi Angela Scipioni Laura Salvi Samantha Cordone Francesca Delucchi Matteo Serino Massimo Federici Flavia Pricci Giuseppe Pugliese 2010Journal of Hepatology2010,,5:1
5An XPS analytical approach for elucidating the microbially mediated enargite oxidative dissolution 显示文摘Fantauzzi M Rossi G Elsener B 2009Analytical and Bioanalytical Chemistry2009,393,8:1
6The surface of enargite after exposure to acidic ferric solutions:an XPS/XAES study显示文摘Fantauzzi M Elsener B Alzei D Lattanzi P Rossi A 2007Surface and Interface Analysis2007,39,1213:1
7An XPS analytical approach for elucidating the mi-crobially mediated enargite oxidative dissolution显示文摘Fantauzzi M Rossi G Elsener B Loi G Atzei D Rossi A 2009Analytical and Bioanalyticai Chemistry2009,393,:1
8The dark and brightside of atherosclerotic calcification 显示文摘Pugliese G Iacobini C Fantauzzi C B 2015Atherosclerosis2015,238,8:1
9The dark and bright side of atherosclerotic calcification显示文摘Giuseppe Pugliese Carla Iacobini Claudia Blasetti Fantauzzi Stefano Menini 2015Atherosclerosis2015,,:1
10Some considerations on the iterative harmonic analysis convergence 显示文摘Cubne R Fantauzzi M Gagliardi F 1993IEEE Transactions on Power Delivery1993,8,2:1
11Enargite oxidation: a review 显示文摘Da Pelo S Musu E Atzei D Elsener B Fantauzzi M Rossi M 2008Earth Sci- ence Reviews2008,86,14:1
12The surface of enargite after exposure to acidic ferric so- lutions : an XPS/XAES study 显示文摘Fantauzzi M Elsener B Atzei D Lattanzi P Rossi A 2007Surface and Interface Analysis2007,39,1213:1
13XPS and XAES Analysis of Copper, Arsenic and Sulfur Chemical State in Enargites 显示文摘Fantauzzi M Atzei D Elsener B 2006Sutf lnteoCace Anal2006,38,5:1
14Microbial translocation, residual viremia and immune senescence in the pathogenesis of HIV-1 infection显示文摘The pathophysiological mechanisms that underlie the progression of human immunodeficiency virus-1(HIV-1) disease to full-blown AIDS are not well understood. Findings suggest that, during HIV-1 infection, plasma lipopolysaccharide(LPS) levels, which are used as an indicator of microbial translocation(MT), are elevated throughout the acute and chronic phases of HIV-1 disease. The translocation of bacterial products through the damaged gastrointestinal barrier into the systemic circulation has been described as a driver of immune activation. In contrast, comorbidities that are associated with HIV-1 infection have been attributed to chronic inflammation and immune system dysfunction secondary to MT or low-level HIV-1 replication in plasma and cell reservoirs. Moreover, accelerated aging is significantly associated with chronic inflammation, immune activation, and immune senescence. In this review, we aimed to investigate the role of inflammation as a pivotal marker in the pathogenesis of HIV-1 disease. We will discuss the key features of chronic inflammation and immune activation that are observed during the natural course of the disease and those features that are detected in c ART-modified infection. The review will focus on the following aspects of HIV-1 infection:(1) MT;(2) the role of residual viremia; and(3) 'immune senescence' or 'inflammaging.' Many questions remain unanswered about the potential mechanisms that are involved in HIV-1 pathogenesis. Further studies are needed to better investigate the mechanisms that underlie immune activation and their correlation with HIV-1 disease progression.Alessandra Fantauzzi Francesca Falasca Gabriella d’Ettorre Eugenio Nelson Cavallari Ombretta Turriziani Vincenzo Vullo Ivano Mezzaroma 2013World Journal of Clinical Infectious Diseases2013,3,4:0
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