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| 1 | 微小RNA-1和微小RNA-29b对急性心肌梗死后不良心室重塑的诊断和预后价值显示文摘已知微小RNA(microRNAs,miRs)在急性心肌梗死(acute myocardial infarction,AMI)临床前期模型中可发挥纤维化和抗纤维化作用。为了评估miR-1,miR-21,miR-29b和miR-92a能否作为患者发生AMI后不良心室重塑(adverse ventricular remodeling,AVR)的循环生物标志物, | 刘莉 叶鹏 Grabmaier U Clauss S Gross L Klier I Franz WM Steinbeck G Wakili R Theiss HD Brenner C | 2017 | 中华高血压杂志2017，25，7： | 17 |
| 2 | Epstein-Barr病毒相关疾病的IgG/Z抗体检测显示文摘血清中Epstein-Barr病毒(EBV)的IgG/Z抗体对鼻咽癌和传染性单核细胞增多症是较特异的,其阳性率分别为85.7%和84%。50%的伯基特淋巴瘤病人亦有IgG/Z抗体。正常人则没有,但有IgG/VCA等抗体,表明IgG/Z抗体在初次感染后是容易消失的。Z抗原与EBV复制相关,故IgG/Z抗体对鼻咽癌不仅有诊断意义,还可能有预后的意义。 | 曾毅 Jean-claude Nicolas Guy Schwaab Guy de The Bernard Clausse Thomas Tursz Irene Joab | 1992 | 病毒学报1992，8，3： | 11 |
| 3 | Temporomandibular joint damage in K/BxN arthritic mice显示文摘Rheumatoid arthritis(RA) is an autoimmune disease affecting 1% of the world population and is characterized by chronic inflammation of the joints sometimes accompanied by extra-articular manifestations. K/Bx N mice, originally described in 1996 as a model of polyarthritis, exhibit knee joint alterations. The aim of this study was to describe temporomandibular joint(TMJ)inflammation and damage in these mice. We used relevant imaging modalities, such as micro-magnetic resonance imaging(μMRI)and micro-computed tomography(μCT), as well as histology and immunofluorescence techniques to detect TMJ alterations in this mouse model. Histology and immunofluorescence for Col-I, Col-II, and aggrecan showed cartilage damage in the TMJ of K/Bx N animals, which was also evidenced by μCT but was less pronounced than that seen in the knee joints. μMRI observations suggested an increased volume of the upper articular cavity, an indicator of an inflammatory process. Fibroblast-like synoviocytes(FLSs)isolated from the TMJ of K/Bx N mice secreted inflammatory cytokines(IL-6 and IL-1β) and expressed degradative mediators such as matrix metalloproteinases(MMPs). K/Bx N mice represent an attractive model for describing and investigating spontaneous damage to the TMJ, a painful disorder in humans with an etiology that is still poorly understood. | Sabine Kuchler-Bopp Alexandre Mariotte Marion Strub Chrystelle Po Aurore De Cauwer Georg Schulz Xavier Van Bellinghen Florence Fioretti Francois Clauss Philippe Georgel Nadia Benkirane-Jessel Fabien Bornert | 2020 | International Journal of Oral Science2020，12，1： | 6 |
| 4 | Dysfunctional stem and progenitor cells impair fracture healing with age显示文摘Successful fracture healing requires the simultaneous regeneration of both the bone and vasculature;mesenchymal stem cells (MSCs) are directed to replace the bone tissue, while endothelial progenitor cells (EPCs) form the new vasculature that supplies blood to the fracture site. In the elderly, the healing process is slowed, partly due to decreased regenerative function of these stem and progenitor cells. MSCs from older individuals are impaired with regard to cell number, proliferative capacity, ability to migrate, and osteochondrogenic differentiation potential. The proliferation, migration and function of EPCs are also compromised with advanced age. Although the reasons for cellular dysfunction with age are complex and multidimensional, reduced expression of growth factors, accumulation of oxidative damage from reactive oxygen species, and altered signaling of the Sirtuin-1 pathway are contributing factors to aging at the cellular level of both MSCs and EPCs. Because of these geriatric-specific issues, effective treatment for fracture repair may require new therapeutic techniques to restore cellular function. Some suggested directions for potential treatments include cellular therapies, pharmacological agents, treatments targeting age-related molecular mechanisms, and physical therapeutics. Advanced age is the primary risk factor for a fracture, due to the low bone mass and inferior bone quality associated with aging;a better understanding of the dysfunctional behavior of the aging cell will provide a foundation for new treatments to decrease healing time and reduce the development of complications during the extended recovery from fracture healing in the elderly. | Diane R Wagner Sonali Karnik Zachary J Gunderson Jeffery J Nielsen Alanna Fennimore Hunter J Promer Jonathan W Lowery M Terry Loghmani Philip S Low Todd O McKinley Melissa A Kacena Matthias Clauss Jiliang Li | 2019 | World Journal of Stem Cells2019，11，6： | 3 |
| 5 | 高速电镀钯-镍合金工艺显示文摘由于金用作电子连接接触表面镀层,成本高,电镀钯-镍合金已成为最具吸引力的接触材料的替代品。然而,传统氨系电镀钯-镍工艺和新的无氨电镀钯-镍工艺有许多环保特征未能达到最佳。与早期电镀钯-镍合金相比,低氨高速电镀钯-镍合金工艺具有技术性能卓越,物料消耗降低和环境状况改善等优势。 | CLAUSS M GUEBEY J SCHWAGER F | 2009 | 电镀与环保2009，29，6： | 3 |
| 6 | 德国社会文化背景下的园林与种植艺术发展史显示文摘植物种植艺术和景观设计有着长达数百年的变迁史,而位于中欧的德国由于受到各种风格的影响更是如此。影响植物种植艺术与景观设计的不仅仅是外部因素,社会内部的艺术形态发展对其同样有很大的影响。文章从园林的起源开始阐述了不同时期植物和建筑的风格在德国社会文化背景下的发展。德国植物景观除了深受国外风格影响外,其种植和设计方式也深刻反映了当时的社会政治环境。 | 张德顺 曹玮 Inga Clauss 樊艺青 曹伯桢 | 2017 | 中国城市林业2017，15，5： | 2 |
| 7 | Stiff macromolecules with aliphatic side-chains-side-chian mobility, conformation, and organization from 2D solid-state NMR spectroscopy显示文摘 | Clauss J Schmidtrohr K Adam A | 1992 | Macromolecules1992，25，20： | 1 |
| 8 | The evolution of a genetic locus encoding small serine proteinase inhibitors 显示文摘 | Clauss A Lilja H Lundwall A | 2005 | Biochem Biophys Res Commun2005，333，2： | 1 |
| 9 | 显示文摘 | Corinna Clauss Marcus Schwarz | 2010 | Carbon2010，48，： | 1 |
| 10 | Phenotype and course of hutchinson-gilford progeria syndrome显示文摘 | Merideth M A Gordon L B Clauss S | 2008 | N Engl J Med2008，358，6： | 1 |
| 11 | Orodental phenotype and genotype findings in all subtypes of hypophosphatasia显示文摘 | Reibel A Manière MC Clauss F | 2009 | Orphanet J Rare Dis2009，4，： | 1 |
| 12 | Expression and modulation of homeobox genes from cluser B in endothelial cells显示文摘 | Belotti D Clausse N Flagiello D | 1998 | Lab Invest1998，78，10： | 1 |
| 13 | Prospective randomized study comparing docetaxel,estramustine,and prednisone with docetaxel and prednisone in metastatic hormone-refractory prostate cancer显示文摘 | Machiels JP Mazzeo F Clausse M | 2008 | J Clin Oncol2008，26，32： | 1 |
| 14 | Ectomycorrhizal symbiosis affects functional diversity of rhizosphere fluorescent pseudomonads显示文摘 | Frey-Klett P Chavatte M Clausse M L | 2005 | New Phytologist2005，165，1： | 1 |
| 15 | Vascular permeability factor: a tumor derived polypeptide that induces endothelial cell and monocyte procoagulant activity, and promotes monocyte migration 显示文摘 | Clauss M Gerlach M Gerlach H | 1990 | J Exp Med1990，172，6： | 1 |
| 16 | Molecular biology of the VEGF and the VEGF receptor family 显示文摘 | Clauss M | 2000 | Semin Thromb Hemost2000，26，5： | 1 |
| 17 | , Comparative in- vestigations on digestion in grazing (Ceratotherium simum ) and browsing ( Diceros bicornis ) rhinoceroses 显示文摘 | STEUER P CLAUSS M S' DEKUM K H | 2010 | Comparative Bio- chemistry and Physiology-Part A: Molecular & Integrative Physi- ology2010，156，4： | 1 |
| 18 | Hydrodynamic shape optimization of large offshore structures显示文摘 | G F Clauss L Birk | 1996 | Applied Ocean Research1996，，： | 1 |
| 19 | Seed germination in desert annuals:An empirical test of adaptive bet hedging显示文摘 | Clauss M Venable D | | 0，，： | 1 |
| 20 | Structural and functional differences in various divisions of the rabbit colon 显示文摘 | Snipes RL Clauss W Weber A | 1982 | Cell Tissue Res1982，225，2： | 1 |
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