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26篇 您的检索式:作者名="CHECLER F"
    题名 作者 年代 出处 被引量
1Processing of the beta-amyloid precursor protein and its regulation in Alzheimer's disease显示文摘Checler F 1995J Neurochem1995,65,4:1
2Processing of the beta-amyloid precursor protein and its regu- lation in Alzheimer's disease 显示文摘Checler F 1995J Neurochem1995,65,4:1
3Amyloid precursor protein,presenilins,and alpha-Synuclein:molecular pathogenesis and pharmacological applications in Alzheimer′s disease显示文摘Suh YH Checler F 0,,03:1
4P53 in neurodegenerative diseases and brain cancers 显示文摘CHECLER F COSTA C A D 2014Pharmacol Ther2014,142,1:1
5Amyloid precursor protein,presenilins,and -synuclein:molecular pathogenesis and pharmacological applications in Alzheimer's disease显示文摘Suh YH Checler F 0,,03:1
6Processing of the beta-amyloid precursor protein and its regulation in Alzheimer disease 显示文摘Checler F 1995J Neurechem1995,65,4:1
7Alzheimer's disease: The amyloid cascade hypothesis-20 years on显示文摘Checler F Turner T 2012Neurochem2012,120,1:1
8BACE1 is at the crossroad of a toxic vicious cycle involving cellular stress andβ-amyloid production in Alzheimer’s disease显示文摘Chami L Checler F 2012Mol Neurodegener2012,7,1:1
9Alzheimer’s and prion diseases: distinctpathologies, common proteolytic denominators显示文摘Checler F Vincent B 2002Trends Neurosci2002,25,61:1
10p53 in neurodegenerative diseases and brain cancers显示文摘Checler F Alves da Costa C 2014Pharmacol Ther2014,142,1:1
11Combined pharmacological, mutational and cell biology approaches indicate that p53-dependent caspase 3 activation triggered by cellular prion is dependent on its endocytosis显示文摘Sunyach C Checler F 2005J Neurochem2005,92,6:1
12Jurkat T cells express a functional neutral endopeptidase activity (CALLA) involved in T cell activation显示文摘Mari B Checler F Ponzio G 1992EMBO J1992,11,11:1
13Amyloid precursor protein, presenilins, and α-Synuclein:molecular pathogenesis and pharmacological applications in Alzheimer's disease显示文摘Suh YH Checler F 2002Pharmacol Rev2002,54,3:1
14Apoptosis in Parkinson/s disease: is p53 the missing link between genetic and sporadic Parkinsonism? 显示文摘Alves da Costa C Checler F 2011Cell Siqnal2011,23,96:1
15APP epsilon,the esepsilon- secretase-derived N-terminal product of the beat-amyloid precursor protein, behaves as a type I protein and undergoes alpha-, beta-, and gamma-secretase cleavages显示文摘Lefranc-Jullien S Sunyach C Checler F 2006J Neurochem2006,97,:1
16P53 is regulated by and regulates members of the gamma-secretase com- plex 显示文摘Checler F Dunys J Alves da Costa C 2010Neurodegener Dis2010,1,7:1
17Apoptosis in Parkinson's disease:is p53 the missing link between genetic and sporadic Parkinsonism显示文摘Alves da Costa C Checler F 0,,06:1
18Processing of the beta-amyloid precursor protein and its regulation in Alzheimer's disease 显示文摘Checler F 1995J Neurochem1995,65,4:1
19Alzheimer's and prion diseases:distinct pathologies,common proteolytic denominators显示文摘Checler F Vincent B 2002Trends Neurosci2002,25,:1
20Wild-type but not Parkinson's disease-related ala-53 right-arrow thr mutant alpha-syn protects neuronal cells from apoptotic stimuli 显示文摘da Costa CA Ancolio K Checler F 2000J Biol Chem2000,275,24:1
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