维普中文期刊产品整合服务
13篇 您的检索式:作者名="Andreas Bringmann"
    题名 作者 年代 出处 被引量
1Different modes of foveal regeneration after closure of full-thickness macular holes by(re)vitrectomy and autologous platelet concentrate显示文摘AIM: To describe using spectral-domain optical coherence tomography the regeneration of the foveal morphology after pars plana(re)vitrectomy surgery and gas tamponade combined with injection of autologous platelet concentrate to treat full-thickness macular holes, and to describe different anatomical outcome. METHODS: A retrospective case series of 8 eyes of 8 patients was described. RESULTS: In all cases investigated, the plateletassisted closure of macular holes was associated with a rapid resolution of cystic cavities in the foveal walls. In two patients, there was a regular regeneration of the foveal morphology after hole closure;the regenerated central fovea had a regular structure with a foveola and photoreceptors. In three other patients, there was an irregular regeneration of the fovea;a foveola was not formed, photoreceptor cells were absent from the foveal center, and the center was composed of Müller and retinal pigment epithelial(RPE) cells. The foveal regeneration after hole closure may proceed with or without a temporary detachment of the foveal center from the RPE, and with or without a direct contact between the central outer nuclear layer(ONL) and the RPE. Contacts between the ONL and RPE were observed only in patients with an irregular foveal regeneration after hole closure.CONCLUSION: The data show that there are different modes of foveal regeneration after closure of macular holes with(re)vitrectomy and platelet concentrate. It is suggested that the regular regeneration of the foveal morphology proceeds by Müller cell-mediated tissue movements without cell proliferation, whereas the irregular foveal regeneration proceeds in part by proliferation of Müller and RPE cells.Andreas Bringmann Claudia Jochmann Jan Darius Unterlauft Renate Wiedemann Matus Rehak Peter Wiedemann 2020International Journal of Ophthalmology(English edition)2020,13,1:3
2Role of retinal glial cells in neurotransmitter uptake and metabolism显示文摘Andreas Bringmann Thomas Pannicke Bernd Biedermann Mike Francke Ianors Iandiev Jens Grosche Peter Wiedemann Jan Albrecht Andreas Reichenbach 2008Neurochemistry International2008,,3:2
3Müller cells in the healthy and diseased retina显示文摘Andreas Bringmann Thomas Pannicke Jens Grosche Mike Francke Peter Wiedemann Serguei N. Skatchkov Neville N. Osborne Andreas Reichenbach 2006Progress in Retinal and Eye Research2006,,4:2
4Involvement of Müller glial cells in epiretinal membrane formation显示文摘Andreas Bringmann Peter Wiedemann 2009Graefe’s Archive for Clinical and Experimental Ophthalmology2009,,7:1
5Müller cells as players in retinal degeneration and edema显示文摘Andreas Reichenbach Antje Wurm Thomas Pannicke Ianors Iandiev Peter Wiedemann Andreas Bringmann 2007Graefe’s Archive for Clinical and Experimental Ophthalmology2007,,5:1
6Cellular signaling and factors involved in Müller cell gliosis: Neuroprotective and detrimental effects显示文摘Andreas Bringmann Ianors Iandiev Thomas Pannicke Antje Wurm Margrit Hollborn Peter Wiedemann Neville N. Osborne Andreas Reichenbach 2009Progress in Retinal and Eye Research2009,,6:1
7Müller cells as players in retinal degeneration and edema显示文摘Andreas Reichenbach Antje Wurm Thomas Pannicke Ianors Iandiev Peter Wiedemann Andreas Bringmann 2007Graefe’s Archive for Clinical and Experimental Ophthalmology2007,,5:1
8Nerve growth factor inhibits osmotic swelling of rat retinal glial (Müller) and bipolar cells by inducing glial cytokine release显示文摘Tarcyane Barata Garcia Thomas Pannicke Stefanie Vogler Benjamin‐Andreas Berk Antje Grosche Peter Wiedemann Johannes Seeger Andreas Reichenbach Anderson Manoel Herculano Andreas Bringmann 2014J. Neurochem2014,,3:1
9Müller cells as players in retinal degeneration and edema显示文摘Andreas Reichenbach Antje Wurm Thomas Pannicke Ianors Iandiev Peter Wiedemann Andreas Bringmann 2007Graefe’s Archive for Clinical and Experimental Ophthalmology2007,,5:1
10Convergence in the Biosynthesis of Acetogenic Natural Products from Plants,Fungi and Bacteria显示文摘Gerhard Bringmann Andreas Irmer Doris Feineis 2009Phytochemistry2009,70,:1
111-Trichloromethyl-1,2,3,4-tetrahydro-β-carboline (TaClo) and related derivatives: chemistry and biochemical effects on catecholamine biosynthesis显示文摘Gerhard Bringmann Doris Feineis Ralf God Karl Peters Eva-Maria Peters Joachim Scholz Franz Riederer Andreas Moser 2002Bioorganic & Medicinal Chemistry2002,,7:1
12Muller cells in the healthy and diseased retina 显示文摘Andreas Bringmann Thomas Pannicke Jens Grosche 2006Progress in Retinal and Eye Research2006,,:1
13Foveal regeneration after resolution of cystoid macular edema without and with internal limiting membrane detachment:presumed role of glial cells for foveal structure stabilization显示文摘AIM: To document with spectral-domain optical coherence tomography the morphological regeneration of the fovea after resolution of cystoid macular edema(CME) without and with internal limiting membrane(ILM) detachment and to discuss the presumed role of the glial scaffold for foveal structure stabilization. METHODS: A retrospective case series of 38 eyes of 35 patients is described. Of these, 17 eyes of 16 patients displayed foveal regeneration after resolution of CME, and 6 eyes of 6 patients displayed CME with ILM detachment. Eleven eyes of 9 patients displayed other kinds of foveal and retinal disorders associated with ILM detachment. RESULTS: The pattern of edematous cyst distribution, with or without a large cyst in the foveola and preferred location of cysts in the inner nuclear layer or Henle fiber layer(HFL), may vary between different eyes with CME or in one eye during different CME episodes. Large cysts in the foveola may be associated with a tractional elevation of the inner foveal layers and the formation of a foveoschisis in the HFL. Edematous cysts are usually not formed in the ganglion cell layer. Eyes with CME and ILM detachment display a schisis between the detached ILM and nerve fiber layer(NFL) which is traversed by Müller cell trunks. ILM detachment was also found in single eyes with myopic traction maculopathy, macular pucker, full-thickness macular holes, outer lamellar holes, and glaucomatous parapapillary retinoschisis, and in 3 eyes with Müller cell sheen dystrophy(MCSD). As observed in eyes with MCSD, cellophane maculopathy, and macular pucker, respectively, fundus light reflections can be caused by different highly reflective membranes or layers: the thickened and tightened ILM which may or may not be detached from the NFL, the NFL, or idiopathic epiretinal membranes. In eyes with short single or multiple CME episodes, the central fovea regenerated either completely, which included the disappearance of irregularities of the photoreceptor layer lines and the reformation of a fovea externa, or with remaining irregularities of the photoreceptor layer lines. CONCLUSION: The examples of a complete regeneration of the foveal morphology after transient CME show that the fovea may withstand even large tractional deformations and has a conspicuous capacity of structural regeneration as long as no cell degeneration occurs. It is suggested that the regenerative capacity depends on the integrity of the threedimensional glial scaffold for foveal structure stabilization composed of Müller cell and astrocyte processes. The glial scaffold may also maintain the retinal structure after loss of most retinal neurons as in late-stage MCSD.Andreas Bringmann Martin Karol Jan Darius Unterlauft Thomas Barth Renate Wiedemann Leon Kohen Matus Rehak Peter Wiedemann 2021International Journal of Ophthalmology(English edition)2021,14,6:0
返回顶部 每页显示:
共1页 首页 上一页 第1页 下一页 末页 /1 跳转

网站首页 | 关于我们 | 联系我们 | 产品服务 | 客服中心 | 广告服务 | 版权声明 | 网站联盟 | 友情链接 | 售卡网点

版权所有© 渝B2-20050021-1 渝公网安备 50019002500403号 违法和不良信息举报中心

互联网出版许可证 新出网证(渝)字10号 全国400电话 - 免长途话费