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28篇 您的检索式：作者名="ABRAMS SI"

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	题名	作者	年代	出处	被引量
1	External beam radiation of tumors alters phenotype of tumor cells to render them susceptible to vaccine-mediated T-cell killing 显示文摘	Chakraborty M Abrams SI Coleman CN	2004	Cancer Res2004，1564，12：	1
2	Differential role of Fas/Fas ligand interactions in cytolysis of primary and metastatic colon carcinoma cell lines by human antigen-specific CD8 + CTL显示文摘	Bergmann-Leitner ES Abrams SI	2000	J Immuno12000，164，9：	1
3	Bortezomib and depsipeptide sensitize tumors to tumor necrosis factor-related apoptosis-inducing ligand:a novel method to potentiate natural killer cell tumor cytotoxicity显示文摘	LUNDQVIST A ABRAMS SI SCHRUMP DS	2006	Cancer Res2006，66，14：	1
4	How widespread are the symptoms of an overactive bladder and how are they managed? A population-based prevalence study显示文摘	Milsim SI Abrams P Cardozo L	2001	Br J Urol Int2001，87，：	1
5	Alterations in Fas expression are characteristic of, but not solely responsible for enhanced metastatic competence显示文摘	Liu K Scott I Abrams SI	2003	Immunol2003，170，12：	1
6	A phase I vaccine trial with peptides reflecting ras oncogene mutations of solid tumors显示文摘	Khleif SN Abrams SI Hamilton JM	1999	J lmmunother1999，22，2：	1
7	Positive and negative consequences of Fas/Fas lig and interactions in the anlitumor response显示文摘	Abrams SI	2005	Front Biosci2005，10，：	1
8	Irradiation of tumor cells up-regulates Fas and enhances CTL lyric activity and CTL adoptive immunotherapy 显示文摘	Chakraborty M Abrams SI Camphausen K	2003	J Immunol2003，170，12：	1
9	External beamradiation of tumor altersphenotype of tumor cells to render them susceptible to vaccinemediated T cell killing显示文摘	ChakrabortyM Abrams SI Coleman CN	2004	Cancer Res2004，64，12：	1
10	How tumours escape mass de- struction显示文摘	STEWART TJ ABRAMS SI	2008	Oncogene2008，27，45：	1
11	External beam radiation of tumors alters phenotype of tumor cells to render them susceptible to vaccine-mediated T-cell killing 显示文摘	Chakraborty M Abrams SI I Coleman CN	2004	Cancer Res2004，64，12：	1
12	Cooperative disengagement of Fas and intercellular adhesion molecule-1 function in neoplastic cells confers enhanced colonization efficiency显示文摘	Liu K Caldwell SA Abrams SI	2005	Cancer Res2005，65，3：	1
13	Altered immune function during long-term host- tumor interactions can be modulated to retard autochthonousneoplastic growth 显示文摘	Stewart TJ Abrams SI	2007	J Immunol2007，179，5：	1
14	显示文摘	Hodge JW Mclaughlin JP Abrams SI	1995	Cancer Res1995，55，：	1
15	A phase I vaccine trial with peptides reflecting rasoncogene mutations of solid tumors 显示文摘	Khleif SN Abrams SI Hamilton JM	1999	J Immunother1999，22，2：	1
16	Targeting CXCL12/CXCR4 signaling with oncolytic virotherapy disrupts tumor vasculature and inhibits breast cancer metastases显示文摘	Gil M Seshadri M Komorowski MP Abrams SI Kozbor D	2013	Proc Natl Acad Sci USA2013，110，14：	1
17	Treatment of human colon carcinoma cell lines with anti-neoplastic agents enhances their lytic sensitivity to antigen-specific CD8^+ cytotoxic T lymphocytes显示文摘	Bergmann-Leitner ES Abrams SI	2001	Cancer Immunol Immunother2001，50，9：	1
18	External beam radiation of tumor alters phenotype of tumor cells to render them susceptible to vaccine-mediated T-cell killing显示文摘	Chakraborty M Abrams SI Coleman CN	2004	Cancer Res2004，64，12：	1
19	Treatment of human colon carcinoma cell lines with anti-neoplastic agents enhances their lytic sensitivity to antigen-specific CD8+ cytotoxic T lymphocytes显示文摘	Bergmann ES Abrams SI	2001	Cancer Immunol Immunother2001，50，9：	1
20	Exposure of human primary colon carcinoma cells to anti-Fas interactions influences the emergence of pre-existing Fas-resistant metastatic subpopulations显示文摘	Liu K McDuffie E Abrams SI	2003	J Immunol2003，171，8：	1

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