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| 1 | Cretaceous source rocks and associated oil and gas resources in the world and China: A review显示文摘The Cretaceous is one of the most important stratigraphic intervals for hydrocarbon source rocks. This article summarizes the distribution, formation, and development characteristics of Cretaceous source rocks and associated oil and gas resources in the world and China, aiming at improving the understanding of this hydrocarbon enrichment and at broadening domestic exploration. Outside China, these rocks are generally formed in marine or transgressive environments during both the Upper and Lower Cretaceous. The majority of Cretaceous source rocks are located in the Persian Gulf, Mediterranean, and Gulf Coast of the USA. Kerogen types within these source rocks have distinct spatial distribution characteristics, with high-latitude Boreal Realm, Tethyan Realm and South Gondwana Realm source rocks containing type III, II, II-III kerogens, respectively. Cretaceous source rocks in China can be mainly divided into four zones: Eastern, Central, Northwest, and Qinghai–Tibet Plateau zones. The majority of Chinese source rocks formed in the Early Cretaceous, whereas the most productive source rocks are developed in the Upper Cretaceous, such as those within the Songliao Basin. Most of these basins are formed in lacustrine environments, although some may have been influenced by transgressive events. Cretaceous source rocks are formed in four distinctive ways: 1) during Oceanic Anoxic Events and associated global sea-level rises, 2) in Black Sea–type retention basins, 3) during transgression and 4) during periods of significant terrestrial input. Formation of these source rocks is controlled by four factors: paleoclimate, paleotopography, transgression, and Oceanic Anoxic Events. These four major controlling factors indicate that China's hydrocarbon exploration within the Cretaceous should focus on two key areas with extremely low exploration levels, the Qinghai–Tibet Plateau and the southeastern coast of China. | Yang Ruofei Wang Yuce Cao Jian | 2014 | Petroleum Science2014，11，3： | 5 |
| 2 | Mobile emergency (surgical) hospital: Development and application in medical relief of "4.20" Lushan earthquake in Sichuan Province, China显示文摘 | Bin Cheng Ruofei Shi Dingyuan Du Ping Hu Jun Feng Guangbin Huang Anning Cai Wei Yin Ronggang Yang | 2015 | Chinese Journal of Traumatology2015，18，1： | 5 |
| 3 | Inhibition of the lncRNA DANCR attenuates cardiomyocyte injury induced by oxygen-glucose deprivation via the miR-19a-3p/MAPK1 axis显示文摘Long noncoding RNAs(lncRNAs)have been considered as crucial regulators of acute myocardial infarction(AMI).In this study,to analyze the effect of differentiation antagonizing nonprotein coding RNA(DANCR)of lncRNA on cardiomyocyte damage in AMI,cardiomyocyte injury was induced by oxygen-glucose deprivation(OGD).Cell counting kit-8(CCK-8)assay and flow cytometry were used to assess cell viability and apoptosis,respectively.Quantitative real-time PCR was used to measure the expression levels of DANCR and miR-19a-3p.Bioinformatics analysis and luciferase gene reporter assay were utilized to explore the relationship among DANCR,miR-19a-3p,and mitogen-activated protein kinase 1(MAPK1).CCK-8 and TUNEL assays were used to explore the effects of DANCR alone or plus miR-19a-3p on the viability and apoptosis of OGD/R-exposed HL-1 cells.Western blot analysis was used to detect changes in the MAPK1/ERK1/2 pathway in HL-1 cells.We found that DANCR expression and miR-19a-3p level are negatively correlated as DANCR expression is increased,while miR-19a-3p level is decreased in AMI patients’serum and OGD/R-exposed HL-1 cells.DANCR knockdown increased miR-19a-3p level,and miR-19a-3p inhibition increased DANCR expression.Moreover,DANCR directly binds to miR-19a-3p.DANCR knockdown reduced viability but induced apoptosis in OGD/R-exposed HL-1 cells,while miR-19a-3p inhibition weakens these effects.Furthermore,MAPK1 is a target of miR-19a-3p.miR-19a-3p overexpression decreases MAPK1 and ERK1/2 in HL-1 cells,while miR-19a-3p inhibition increases MAPK1 and ERK1/2 in HL-1 cells.Moreover,DANCR knockdown reduces myocardium apoptosis in mice with the left anterior descending artery ligated.DANCR knockdown effectively restores myocardial cell apoptosis by regulating the miR-19a-3p/MAPK1/ERK1/2 axis. | Yang Ruan Hong Li Xiaojing Cao Shuai Meng Ruofei Jia Lianmei Pu Hao Fu Zening Jin | 2021 | Acta Biochimica et Biophysica Sinica2021，53，10： | 4 |
| 4 | Quality Evaluation of Spatial Point-Cloud Data Collected by Vehicle-Borne Laser Scanner 显示文摘 | Feng Jiaxiang Zhong Ruofei Yang Yong | 2008 | International Workshop on Education Technology and Training & 2008 International Workshop on Geoscience and Remote Sensing2008，，： | 1 |
| 5 | H3K14me3 genomic distributions and its regulation by KDM4 family demethylases显示文摘 | Bin Zhao Wenqi Xu Bowen Rong Guoyu Chen Xuanjia Ye Ruofei Dai Wenjinq Li Jiajia Chen Jiajun Cai Lei Song Zhao-Qing Luo Rong Zeng Yang Shi Jing-Dong J. Han Fei Lan | 2018 | Cell Research2018，28，11： | 1 |
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