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198篇 您的检索式:作者名="Yan SD"
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1Belowground carbon balance and carbon accumulation rate in the successional series of monsoon evergreen broad-leaved forest显示文摘The balance, accumulation rate and temporal dynamics of belowground carbon in the successional series of monsoon evergreen broadleaved forest are obtained in this paper, based on long-term observations to the soil organic matter, input and standing biomass of litter and coarse woody debris, and dissolved organic carbon carried in the hydrological process of subtropical climax forest ecosystem—monsoon evergreen broad-leaved forest, and its two successional forests of natural restoration—coniferous and broad-leaved mixed forest and Pinus massoniana forest, as well as data of root biomass obtained once every five years and respiration measurement of soil, litter and coarse woody debris respiration for 1 year. The major results include: the belowground carbon pools of monsoon evergreen broad-leaved forest, coniferous and broad-leaved mixed forest, and Pinus massoniana forest are 23191 ± 2538 g·m?2, 16889 ± 1936 g·m?2 and 12680 ± 1854 g·m?2, respec- tively, in 2002. Mean annual carbon accumulation rates of the three forest types during the 24a from 1978 to 2002 are 383 ± 97 g·m?2·a?1, 193 ± 85 g·m?2·a?1 and 213 ± 86 g·m?2·a?1, respectively. The belowground carbon pools in the three forest types keep increasing during the observation period, suggesting that belowground carbon pools are carbon sinks to the atmosphere. There are seasonal variations, namely, they are strong carbon sources from April to June, weak carbon sources from July to September; while they are strong carbon sinks from October to November, weak carbon sinks from December to March.ZHOU Guoyi1, ZHOU Cunyu1, LIU Shuguang2, TANG Xuli1, OUYANG Xuejun1, ZHANG Deqiang1, LIU Shizhong1, LIU Juxiu1, YAN Junhua1, ZHOU Chuanyan1, LUO Yan1, GUAN Lili1 & LIU Yan1 1. Dinghushan Forest Ecosystem Research Station, Chinese Academy of Sciences, Guangzhou 510650, China 2. SAIC, EROS Data Center, U.S. Geological Survey, Sioux Falls, SD 57198, USA 2006Science China Earth Sciences2006,49,3:36
2RAGE and amyloid-beta peptide neurotoxity in Alzheimer′s disease显示文摘Yan SD Chen X Fu J 1996Nature1996,382,6593:1
3Pathogenic role of mitochondrial amyloid-β peptide显示文摘Chen JX Yan SD 2007Expert Rev Neurother2007,7,11:1
4Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis 显示文摘Schmidt AM Yan SD Wautier JL 1999Circulation research1999,84,5:1
5Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress 显示文摘Yan SD Chen X Schmidi AM 1994Proc Natl Acad Sci USA1994,91,16:1
6RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease显示文摘Yan SD Chen X Fu J 1996Nature1996,382,:1
7Cyclic GMP phosphodiesterases and regulation of smooth muscle function显示文摘Rybalkin SD Yan C Bomfeldt KE 2003Circ Res2003,93,4:1
8RAGE and amyloid-beta peptide neurotox- ieity in Alzheimer's disease 显示文摘Yan SD Chen X Fu J 1996Nature1996,382,:1
9Role of ERAB/L-3-hydroxyacylcoenzyme A dehydrogenase type Ⅱ activity in A beta-induced cytotoxicity显示文摘Yan SD Shi Y Zhu A 1999Biol Chem1999,274,4:1
10The biology of the receptor for advanced glycation end products and its ligands显示文摘Schmidt AM Yan SD Yan SF 2000Biochim Biophys Acta2000,1498,23:1
11Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis显示文摘Schmidt AM Yan SD Wautier JL 1999Circ Res1999,84,:1
12Engineering Streptomyces tenebrarius to synthesize single component显示文摘Hong WR Yan SD 2012Letters in Applied Microbiology2012,55,1:1
13Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress 显示文摘Yan SD Chen X Schmidt AM 1994Proc Natl Acad Sci USA1994,91,16:1
14Activation of receptor for advanced glycation end products:a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis显示文摘Schmidt AM Yan SD Wautier JL 1999Circ Res1999,84,5:1
15Enhanced cellular oxidant stress by the interaction of advanced glycafion end products with their receptors/binding proteins 显示文摘Yan SD Schmidt AM Anderson GM 1994J Biol Chem1994,269,:1
16The biology of the receptor for dvanced glycation end products and its ligands 显示文摘Schmit AM Yan SD Yan SF 2000Biochem Biophys Acta2000,1498,23:1
17RAGE and amyloid-β peptides neurotoxicity in Alzheimer's disease显示文摘 Chen X Fu J 1996Nature1996,382,:1
18Mitochondrial Abeta:a potential cause of metabolic dysfunction in Alzheimer's disease显示文摘Chen X Yan SD 2006IUBMB Life2006,58,12:1
19Advanced glycation end products and RAGE:a common thread in aging,diabetes,neurodegeneration,and inflammation显示文摘Ramasamy R Vannucci SJ Yan SD 2005Glycobiology2005,15,7:1
20The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses显示文摘Schmidt AM Yan SD Yan SF 2001J Clin Invest2001,108,7:1
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