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| 1 | Hepatitis E virus infec- tion as a cause of graft hepatitis in liver transplant recipients 显示文摘 | Pischke S Suneetha PV Baechlein C | 2010 | Liver Transpl2010，16，1： | 1 |
| 2 | Dual function of the NK cell receptor 2B4(CD244)in the regulation of HCV-specific CD8+ T cells显示文摘 | SchlaphoffV Lunemaun S Suneetha PV | | 0，，： | 1 |
| 3 | Dual function of the NK cell receptor 2B4 (CD244) in the regulation of HCV-specific CD8 T cells显示文摘 | Schlaphoff V Lunemann S Suneetha PV | 2011 | PLoS Pathog2011，7，10： | 1 |
| 4 | Hepatitis E virus infection as a cause of graft hepatitis in liver transplant recipients 显示文摘 | Pischke S Suneetha PV Baechlein C | 2010 | Liver Transpl2010，16，1： | 1 |
| 5 | Dual function of the NK cell receptor 2B4 (CD244) in the regu- lation of HCV - specific CD8^ + T cells 显示文摘 | Schlaphoff V Lunemann S Suneetha PV | 2011 | PLoS Pathog2011，7，10： | 1 |
| 6 | Dual function of the NK cell receptor 2B4(CD244) in the regulation of HCV-specific CD8+ T cells显示文摘 | Schlaphoff V Lunemann S Suneetha PV | 2011 | PLoS Pathog2011，7，10： | 1 |
| 7 | Hepatitis E virus (HEV)-specific T-cell responses are associated with contzol of HEV infection显示文摘 | Suneetha PV Pischke S Schlaphoff V | 2012 | Hepatology2012，55，3： | 1 |
| 8 | Dual function of the NK cell receptor 21M (CD244) in the regulation of HCV-speclfieCD8 + T cells 显示文摘 | Schlaphoff V Lunemann S Suneetha PV | 2011 | PLoS Pathog2011，7，10： | 1 |
| 9 | Association between vitamin D receptor,CCR5,TNF-α and TNF-β gene polymorphisms and HBV infection and severity of liver disease显示文摘 | Suneetha PV Sarin SK Goyal A | 2006 | Int J Hepatol2006，44，5： | 1 |
| 10 | Association between vitamin D receptor,CCR5,TNF-α and TNF-β gene polymorphisms and HBV infection and severity of liver disease显示文摘 | Suneetha PV Sarin SK Goyal A | 2006 | J Clin Heptol2006，44，5： | 1 |
| 11 | Du- al function of the NK cell receptor 284 (CD244) in the regu- lation of HCV - specific CD81 T cells 显示文摘 | SCHLAPHOFF V LUNEMANN S SUNEETHA PV | 2011 | PLoS Pathog2011，7，10： | 1 |
| 12 | Association between vitamin D receptor,CCRS,TNF-a and TNF-b gene polymorphisms and HBV infection and severity of liver disease显示文摘 | Suneetha PV Satin SK Geyal A | 2006 | J Hepatol2006，44，5： | 1 |
| 13 | CCR5A32 mutation does not influence the susceptibility to HCV infection, severity of liver disease and response to therapy in patients with chronic hepatitis C显示文摘AIM: To study whether CCR5A32 mutation was associated with viral infection and severity of liver disease. METHODS: Two hundred and fifty two histologically proven, chronic HCV patients (mean age: 41±14 years; M/F: 164/88) were genotyped. PCR based genotyping of 32 bp deletion at the CCR5 locus was done. Four-hundred and eight matched healthy controls were studied to assess susceptibility to HCV infection. To assess correlation of immune gene polymorphism with severity of HCV related liver disease, patients with chronic HCV infection were divided into those with a fibrosis score of≤2 (mild) or > 2 (severe) and histological activity index (HAI) of≤5 or > 5. For correlation between CCR5A32 mutations and response to therapy, 129 patients who completed therapy were evaluated. RESULTS: The majority (89.4%) of the patients were infected with genotype 3. The frequency of homozygous CCR5A32 mutants was comparable to HCV patients as compared to the healthy controls (0.7% vs 0%, P = 0.1). Further more, the frequency of CCR5A32 mutation was comparable in patients with mild or severe liver disease. (P=NS). There was also no association observed with response to therapy and CCR5A32 mutation. CONCLUSION: CCR5A32 mutation does not have a role in disease susceptibility, severity or response to therapy in patients with chronic hepatitis C infection. | Ankur Goyal PV Suneetha GT Kumar Deepak K Shukla Naveen Arora Shiv K Sarin | 2006 | World Journal of Gastroenterology2006，12，29： | 0 |
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