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| 1 | Altered motor activity,explora-tionandanx-iety in heterozygousneuregulin-1 mutant mice:impli-cations for understanding schizophrenia显示文摘 | Karl T Duffy L Scimone A | 2007 | Genes Brain Behav2007，6，7： | 1 |
| 2 | Behavioral profile of a heter-ozygous mutant mouse model for EGF-like domain neuregulin-1显示文摘 | Duffy L Cappas E Scimone A | 2008 | Behav Neurosci2008，122，4： | 1 |
| 3 | Kinetics of sunflower oil contamination with polycyclic aromatic hydro- carbons from contaminated recycled low density polyethylene film 显示文摘 | SCIMKO P KHUNOVA V SCIMON P | 1995 | Int J Food Sci Technol1995，30，6： | 1 |
| 4 | A multistep adhesion cascade for lymphoid progenitor cell homing to the thymus显示文摘 | Scimone M L Aifantis I Apostolou I | 2006 | Proc Natl Acad Sci U S A2006，103，18： | 1 |
| 5 | Efficient thymic immigration of B220+ lymphoid-restricted bone marrow cells with T precursor potential显示文摘 | Martin C H Aifantis I Scimone M L | 2003 | Nat Immunol2003，4，9： | 1 |
| 6 | Caspase-11 regulates cell migration by promoting Aipl-cofilin-mediated actin depolymerization显示文摘 | Li J Brieher WM Scimone ML | 2007 | Nat Cell Biol2007，9，3： | 1 |
| 7 | A regulatory program for excretory system regeneration in planarians显示文摘 | Scimone ML Srivastava M Bel GW | | 0，，20： | 1 |
| 8 | A genome screen of a large bipolar affetive disorder pedigree sujpports evidence for a susceptibility locus on chromosome 13q 显示文摘 | Badenhop RF Moses MJ Scimone A | 2001 | Mol Psychiatry2001，6，4： | 1 |
| 9 | The SIP-analog FTY720 differentially modulates T-cell homing via HEV: T-cell-expressed S1P1 amplifies integrin activation in peripheral lymph nodes but not in Peyer patche 显示文摘 | Halin C Scimone ML Bonasio R | 2005 | Blood2005，106，4： | 1 |
| 10 | Etoposide, l-leucovorin and fluorouracil (ELF) regimen in metastatic gastric cancer: A phase Ⅱ study 显示文摘 | Scimone A Maisano R Altavilla G Ferraro G Laudani A Pergolizzi S Zanghi M | 1999 | J Chemother1999，11，1： | 1 |
| 11 | A genome screen of 13 bipolar affective disorder pedigrees provides evidence for susceptibility loci on chromosome 3 as well as chromosomes 9,13 and 19显示文摘 | Badenhop RF Moses MJ Scimone A | 2002 | Molecular Psychiatry2002，7，6： | 1 |
| 12 | Neo- blast specialization in regeneration of the planarian Schmidtea mediterranea 显示文摘 | Scimone M L Kravarik K M Lapan S W | 2014 | Stem Cell Reports2014，3，2： | 1 |
| 13 | A multistep adhesion cascade for lymphoid progenitor cell homing to the thymus显示文摘 | Scimone M L Aifantis I Apostolou I | 2006 | Proc Natl Acad Sci USA2006，103，18： | 1 |
| 14 | Levamisole and lymphocyte responsiveness显示文摘 | Chan SH Scimons MJ | 1975 | Lancet1975，1，： | 1 |
| 15 | A genorne screen of a large bipolar affective disorder pedigree sujpports evidence for a susceptibility locus on chromosome 13q 显示文摘 | Badenhop RF Moses MJ Scimone A | 2001 | Mol Psychiatry2001，6，4： | 1 |
| 16 | Neutrophil elastase converts human immature dendritic cells into transforming growth factor-β1-secreting cells and reduces allostimulatory ability显示文摘 | Maffia PC Zittermann SE Scimone ML | 2007 | Am J Pathol2007，171，3： | 1 |
| 17 | Investigating the role of imprinted genes in pediatric sporadic brain arteriovenous malformations显示文摘Arteriovenous malformation AVM is a vascular congenital defect affecting microvasculatu re of both brain and peripheral o rga ns.Arteriovenous malformation of the brain(bAVM,OMIM#108010),in particular,affects up to 15 per100,000 persons with no sex predominance.Almost 50%of the patients manifest intracerebral hemorrhage and epileptic seizures,as main clinical symptoms. | Concetta Scimone Luigi Donato Antonina Sidoti | 2022 | Neural Regeneration Research2022，17，1： | 0 |
| 18 | Human retinal secretome:A cross-link between mesenchymal and retinal cells显示文摘In recent years,mesenchymal stem cells(MSC)have been considered the most effective source for regenerative medicine,especially due to released soluble paracrine bioactive components and extracellular vesicles.These factors,collectively called the secretome,play crucial roles in immunomodulation and in improving survival and regeneration capabilities of injured tissue.Recently,there has been a growing interest in the secretome released by retinal cytotypes,especially retinal pigment epithelium and Müller glia cells.The latter trophic factors represent the key to preserving morphofunctional integrity of the retina,regulating biological pathways involved in survival,function and responding to injury.Furthermore,these factors can play a pivotal role in onset and progression of retinal diseases after damage of cell secretory function.In this review,we delineated the importance of cross-talk between MSCs and retinal cells,focusing on common/induced secreted factors,during experimental therapy for retinal diseases.The cross-link between the MSC and retinal cell secretomes suggests that the MSC secretome can modulate the retinal cell secretome and vice versa.For example,the MSC secretome can protect retinal cells from degeneration by reducing oxidative stress,autophagy and programmed cell death.Conversely,the retinal cell secretome can influence the MSC secretome by inducing changes in MSC gene expression and phenotype. | Luigi Donato Concetta Scimone Simona Alibrandi Sergio Zaccaria Scalinci Domenico Mordà Carmela Rinaldi Rosalia D'Angelo Antonina Sidoti | 2023 | World Journal of Stem Cells2023，15，7： | 0 |
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