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63篇 您的检索式:作者名="Scheuner D"
    题名 作者 年代 出处 被引量
1Chop deletion reduces oxidative stress,improvesβcell function,and promotes cell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,:1
2Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in mul- tiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
3The unfolded proteinresponse in nutrient sensing and differentiation显示文摘Kaufman RJ Scheuner D Schroder M 2002Nat Rev MolCell Biol2002,3,6:1
4Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer#s disease显示文摘Scheuner D Eckman C Jensen M 1996Nat Med1996,2,8:1
5Translational control is required for the unfolded protein response and in vlvo glu- cose homeostasis显示文摘Scheuner D Song B McEwen E 2001Mol Cell2001,7,6:1
6Short-term impacts of harvesting and burning disturbances on physical and chemical characteristics of forest soils in western Newfoundland, Canada 显示文摘Scheuner E T Makeschin F Wells E D 2004European Journal of Forest Research2004,123,:1
7Chop deletion reducesoxidative stress, improves beta cell function, and promotescell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J ClinInvest2008,118,10:1
8Chop deletion reduces ox- idative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008Clin Invest2008,118,10:1
9Translational control is required for the unfolded protein response and in vivo glucose homeostasis显示文摘Scheuner D Song B McEwen E 2001Mol Cell2001,7,6:1
10Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes 显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
11The unfolded protein response in nutrient sensing and differentiation 显示文摘Kaufman RJ Scheuner D Schroder M 2002Nat Rev Mol Cell Biol2002,3,6:1
12Secreted amyloid β protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease显示文摘Scheuner D Eckmen C Jensen M 1996Nature Med1996,2,86:1
13Chop deletion reducesoxidative stress, improves p cell function, and promotes cellsurvival in multiple mouse models of diabetes 显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
14Translational control is required for the unfolded protein response and in vivo glucose homeostasis显示文摘Scheuner D Song B MeEwen E 2001Mol Cell2001,7,6:1
15Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
16Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
17The unfolded protein response:a pathway that links insulin demand with β-cell failure and diabetes显示文摘Scheuner D Kaufman RJ 2008Endocr Rev2008,29,3:1
18The unfolded protein response in nutrient sensing and differentiation 显示文摘Kaufman R J Scheuner D Schroder M 2002Nat Rev Mol Cell Biol2002,3,:1
19Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes显示文摘Song B Scheuner D Ron D 2008J Clin Invest2008,118,10:1
20Potential role of PKR in double-stranded RNA-induced macrophage activation 显示文摘Maggi LB Heitmeier MR Scheuner D 2000EMBO J2000,19,14:1
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