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1系统综述与网状Meta分析的PRISMA扩展声明显示文摘PRISMA声明旨在提高系统综述和META分析报告的完整性,该声明已经广泛用于指导系统综述和META分析的报告和发表。原始的PRISMA声明是针对两种干预措施比较的传统的系统综述与META分析而制定的,然而,随着多种干预措施比较的系统综述的发展,实施和报告这一类系统综述面临较大挑战。此时,针对网状META分析的PRISMA扩展声明应运而生,旨在提高网状META分析系统综述的报告质量。PRISMA扩展声明是由专家们通过DELPHI调查、面对面讨论和共识大会而最终确立的。PRISMA扩展声明是在原始PRISMA声明的报告清单的基础上经过修改,最终确定了32个条目,每个条目均与网状META分析报告的内容直接相关。本文对网状META分析的PRISMA扩展声明进行了阐述,对报告清单各条目进行了举例说明,并详细说明了在原始PRISMA声明的基础上新增和修改各条目的理由。此外,PRISMA扩展声明强调了在网状META分析的实际操作中需要重点关注的信息。本文的目标读者包括网状META分析的作者与读者,以及期刊杂志的编辑与同行评审。李志霞 杨俊 叶欣 周凌波 杨智荣 孙凤 詹思延 Brian Hutton Georgia Salanti Deborah M.Caldwell Anna Chaimani Christopher H.Schmid Chris Cameron John P.A.Ioannidis Sharon Straus Kristian Thorlund Jeroen P.Jansen Cynthia Mulrow Ferrán Catalá-López Peter C.Gozsche Kay Dickersin Isabelle Boutron Douglas G.Altman David Moher 2016中国循证心血管医学杂志2016,8,6:11
2Zinc Fingers, TAL Effectors, or Cas9-Based DNA Binding Proteins: What's Best for Targeting Desired Genome Loci?显示文摘ZINC FINGER-AND TAL EFFECTORBASED DNA BINDING PROTEINS Programmable DNA binding proteins (PDPs) that selectively bind user-defined nucleotide sequences can navigate protein domains to any desired target within the genome.Different functional domains can be fused to PDPs to achieve (1) activation or repression of transcription,(2) nicking or cleavage of DNA,or (3) modification or removal of epigenetic marks at the targeted genome spot.Given their broad applicability,the development of highly specific,easy-to-use PDPs has been a holy grail of biotechnology since the determination of the 3-D structure of zinc fingers (ZFs) bound to DNA in 1991 (Pavletich and Pabo,1991).Annett Strauβ Thomas Lahaye 2013Molecular Plant2013,6,5:6
3Prolonged QTc Interval and Risk of Sudden Cardiac Death in a Population of Older Adults显示文摘Sabine M.J.M. Straus Jan A. Kors Marie L. De Bruin Cornelis S. van der Hooft Albert Hofman Jan Heeringa Jaap W. Deckers J. Herre Kingma Miriam C.J.M. Sturkenboom Bruno H. Ch. Stricker Jacqueline C.M. Witteman 2006Journal of the American College of Cardiology2006,,2:4
4老年人群中QTc间期延长与心脏性猝死风险之间的关系显示文摘OBJECTIVES: This study sought to investigate whether prolongation of the heart rate-corrected QT(QTc) interval is a risk factor for sudden cardiac death in the general population. BACKGROUND: In developed countries, sudden cardiac death is a major cause of cardiovascular mortality. Prolongation of the QTc interval has been associated with ventricular arrhythmias, but in most population-based studies no consistent association was found between QTc prolongation and total or cardiovascular mortality. Only very few of these studies specifically addressed sudden cardiac death. METHODS: This study was conducted as part of the Rotterdam Study, a prospective population- based cohort study that comprises 3,105 men and 4,878 women aged 55 years and older. The QTc interval on the electrocardiogram was determined during the baseline visit(1990 to 1993) and the first follow-up examination(1993 to 1995). The association between a prolonged QTc interval and sudden cardiac death was estimated using Cox proportional hazards analysis. RESULTS: During an average follow-up period of 6.7 years(standard deviation, 2.3 years) 125 patients died of sudden cardiac death. An abnormally prolonged QTc interval(>450 ms in men, >470 ms in women) was associated with a threefold increased risk of sudden cardiac death(hazard ratio, 2.5; 95% confidence interval, 1.3 to 4.7), after adjustment for age, gender, body mass index, hypertension, cholesterol/high-density lipoprotein ratio, diabetes mellitus, myocardial infarction, heart failure, and heart rate. In patients with an age below the median of 68 years, the corresponding relative risk was 8.0(95% confidence interval 2.1 to 31.3). CONCLUSIONS: Abnormal QTc prolongation on the electrocardiogram should be viewed as an independent risk factor for sudden cardiac death.Straus S.M.J.M Kors J.A De Bruin M.L. B.H.Ch. Stricker 王亭忠 2006世界核心医学期刊文摘(心脏病学分册)2006,2,5:3
5The Chronic Fatigue Syndrome:A Comprehensive Approach to Difinition and Study显示文摘Fukuda K Straus SE Hickie I 1994Ann Intem Med1994,121,12:1
6How do I perform a lumbar puncture and analyze the results to diagnose bacterial meningitis? 显示文摘Straus SE Thorpe KE Holroyd-Leduc J 2006JAMA2006,296,16:1
7The Chronic fatigue syndrome:a comprehensive approach to it sdefinition and study显示文摘Fukuda K Straus SE Hickl 1994Ann Intern Med1994,121,12:1
8The development of lymphomas in families with autoimmune lymphoproliferative syndrome with germline Fas mutations and defective lymphocyte apoptosis显示文摘Straus SE Jaffe ES Puck JM 2001Blood2001,98,1:1
9The Chronic Fatigue Syndrome: A Comprehensive Approach to I)ifinition and Study 显示文摘Fukuda K Straus SE Hickie I 1994Ann Intern Med1994,121,12:1
10Chest X-ray screeningimproves outcome in lung cancer,a reappraisal of randomized trialson lung cancer screening显示文摘Straus GM Gleason RE Sugarbaker DJ 2005Chest2005,,6:1
11Addition chains of vectors (problem 5125)显示文摘E G Straus 1964American Mathematical Monthly1964,70,:1
1215-deoxy- elta12,14-prostaglandinJ2 inhibits multiple steps in the NF-kappaB signaling pathway显示文摘Straus DS Pascual G li M at al 2000Proc Natl Acad Sci USA2000,97,:1
13Assessment and management of falls in older people 显示文摘Kwan E Straus SE 2014CMAJ2014,186,16:1
14Immunophenotypic profiles in families with autoimmune lymphoproliferative syndrome显示文摘Bleesing JJ Brown MR Straus SE 2001Blood2001,98,8:1
15The Chronic Fatigue Syndrome:A Comprehensive Approsch to Its Difinition andstudy显示文摘Fukuda K Straus S Hickie I 1994Ann Intem Med1994,121,12:1
16The chronic fatigue syndrom: a comprehensive approach to its definition and study 显示文摘Fukuda K Straus S E Hicki I 1994Ann Intern Med1994,121,12:1
17The Chronic Fatigue Syndrome:A Comprehensive Approach to Definition and Study显示文摘 Straus SE Hickie I 1994Ann Intern Med1994,121,12:1
18Cyclopentenone prostaglandins : new insights on biological activities and cellular targets 显示文摘Straus DS Glass CK 2001Med Res Rev2001,21,3:1
1915-deoxy-delta 12,14-prostaglandin J2 inhibits multiple steps in the NF-kappa B signaling pathway显示文摘Straus DS Pascual G Li G 2002Proc NatI Acad Sci USA2002,97,9:1
20Specific decrease in liver insulin like growth factor-Ⅰ and brain insulin-like growth factor-Ⅱgene expression in energy-restricted rats 显示文摘Straus DS Takemoto CD 1991J Nutr1991,121,8:1
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