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12篇 您的检索式:作者名="Rodolphe S"
    题名 作者 年代 出处 被引量
1Effects of total enteral nutrition supplemented with a multi-fibre mix on faecal short chain fatty acids and microbiota显示文摘Schneider S M Fernand G P Rodolphe A 2006Clinic Nutrition2006,25,:1
2The advocacy coalition framework:application to the policy process for the development of forest certification in Sweden显示文摘Chris E Rodolphe S 2001Journal of European Public Policy2001,,8:1
3Effects of total enteral nutrition supplemented with a multi-fibre mix on faecalshort-chain fatty acids and microbiota显示文摘SCHNEIDER S M FERNAND G P RODOLPHE A 2006Clinical Nutrition2006,25,1:1
4FUNYBASE: a FUNgal phYlogenomic dataBASE 显示文摘Marthey S Aguileta G Rodolphe F 2008BMC Bioinformatics2008,,9:1
5Enhanced regeneration of tomato and pepper seedling explants for Agrobacterium-mediated transformation显示文摘Javier Pozueta-Romero Guy Houlné Luis Ca?as Rodolphe Schantz Jesús Chamarro 2001Plant Cell Tissue and Organ Culture2001,,2:1
6Effects of total enteral nutrition supplemented with a multi-fibre mix on faecal short-chain fatty acids and microbiota显示文摘SCHNEIDER S M FERNAND G P RODOLPHE A 2006Clin Nutr2006,25,:1
7CFD analysis of industrial multi-staged stirred vessels显示文摘Marion A G Rodolphe S Catherine X 2006Chemical Engineering Process2006,45,1:1
8Effects of total enteral nutrition supplemented with a multifibre mix on fae- cal short chain fatty acids and microbiota显示文摘SCHNEIDER S M FERNAAND G P RODOLPHE A 2006Clinical Nutrition2006,25,1:1
9Rapid detection of HIV-1 by reverse-transcription,loop-mediated iso- thermal amplification(RT-LAMP) 显示文摘Curtis K A Rodolph D L Owen S M 2008J Virol Meth- ods2008,2,:1
10Gullans discovery of molecular mechanisms of neuroprotection using cell-based bioassays and oligonucleotide arrays显示文摘SATINDER S S YOSHIDA T A RODOLPHE C 2002Physiol Genomics2002,11,1:1
11Effects of total enteral nutrition supplemented with amulti-fibre mix on faecal short-chain fatty acids and mi-crobiota显示文摘SCHNEIDER S M FERNAND G P RODOLPHE A etal 2006Clin Nutr2006,25,:1
12Pathological evolution of hepatitis C virus-“Healthy carriers”显示文摘AIM: To determine factors associated with fibrosis progression in hepatitis C virus (HCV)-infected patients without signifi cant initial pathological lesions. METHODS: Seventy six untreated HCV-infected pa-tients with initially normal liver as defi ned by a Knodell score≤3,with 2 liver biopsies and detectable HCV-RNA were included. Markers of fibrosis progression were assessed. RESULTS: Median duration of infection and time between paired biopsies was 13 (95% CI: 1-28) and 4 (95% CI: 2-16) years respectively. Alanine-transaminase (ALT) activity was normal in 43.4% of cases. 50% demonstrated progression of the necro-in? ammation and 34% of fi brosis after a median time evolution of 4 years (95% CI: 2-16). The median dif-ference in the necro-inflammation and fibrosis score between biopsies was low,1.5 and 0.0 respectively. Univariate analysis showed there was no difference between fibrosis activity or evolution according to genotype or viral load. A higher fibrosis progression (P = 0.03) was observed in patients with body mass index (BMI) > 25. Fibrosis progression correlated with the time interval between biopsies (P = 0.01). A sig-nifi cant progression of activity (1.7 vs 0.4,P < 0.05) or fi brosis (0.9 vs 0.0,P < 0.01) was observed in patients with elevated ALT. There was a signifi cant correlation between activity progression and fi brosis progression (P = 0.003). Multivariate analysis demonstrated that fi brosis progression was associated with elevated ALT,BMI > 25 and the time interval between 2 biopsies. CONCLUSION: There is no fibrosis progression in 66% of patients without signifi cant initial histopatho-logical lesion. Fibrosis progression is associated with elevated ALT and BMI > 25.Rodolphe Sobesky Pascal Lebray Bertrand Nalpas Anas Vallet-Pichard Hélène Fontaine Jean-Luc Lagneau Stanislas Pol 2008World Journal of Gastroenterology2008,14,24:0
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