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6篇 您的检索式:作者名="Rashidi I"
    题名 作者 年代 出处 被引量
1Focal adhesion kinase (FAK) involvement in human endometrial remod- eling during the menstrual cycle 显示文摘Orazizadeh M Rashidi I Saremi J 2009Iran Biomed J2009,13,2:1
2A decision treebased approach for determining low bone mineral density in inflammatory bowel disease using WEKA software显示文摘 Rashidi M I 2007Eur J Gastroenterol Hepatol2007,19,12:1
3Adenomyosis among samples from hysterectomy due to abnormal uterine bleeding in Ahwaz, southern Iran显示文摘Mobarakeh MD Maghsudi A Rashidi I 2012Adv Biomed Res2012,,:1
4Performance Investigation of a Combined MCFC System 显示文摘Rashidi R Berg P Dincer I 2009International Journal of Hydrogen Energy2009,34,10:1
5Hepatoprotective Activity of Capparis spinosa Root Bark Against CC1 4 Induced Hepatic Damage in Mice显示文摘Aghel N Rashidi I Mombeini A 0,,04:1
6The influence of β_2AR polymorphisms as a predictor of successful short acting β_2-agonist nebulization during asthmatic exacerbation显示文摘Aim:In this study,we would like to determine associations between β2-Adrenergic Receptor(β2AR)polymorphisms at codon 16 and 27 and the response to short acting β2-agonist during asthmatic exacerbation.Methods:This was a prospective cross-sectional study of one year duration.One hundred and thirty two asthmatic patients were recruited.Five mls of venous blood was taken for DNA extraction and then genotyped for the β2AR polymorphisms using multiplex PCR.Patient's clinical responses to β2-agonist nebulization were then compared to their genotype to determine the association.Results:We found that there was no association between β2AR polymorphisms at both codon 16 and 27 with response towards short acting β2-agonist,P=0.315 and P=0.706 respectively.Conclusion:We suggested that β2AR polymorphisms at both codon 16 and 27 had no influent on the response to short acting β2-agonist.Nasir M Nor Hidayah AB Rashidi A Rusli I 2010海南医学院学报2010,16,7:0
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