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| 1 | 中国地理人的解决研究的概述和进步显示文摘增加的中国都市化和工业化在地理,建筑学,并且城市的计划的领域里推动了更大的注意到人的解决和人陆地的关系的学习。我们使用了 bibliometric 方法和统计软件在 16 本中国地理杂志在人的解决上考察 180 篇文章。我们发现中国地理人的解决研究被下列描绘:( 1 )大多数研究在在媒介的模式放大的宏规模,城市的解决区别和理想集中于人的解决扩展,估价指示物,为城市、农村的解决的模型,理论探索和单个因素的、人的解决和复杂、地理存活率的计划惯例,解决环境的全面评估,并且计划社区单位在微规模,社区解决;关于推进人的解决的社会文化的调查和警告。(2 ) 没有进步在综合并且集成方法系统被取得了。PSR 当模特儿, DPSIR 模型被用于指向机制,当标准解决评估系统被问询表调查由物理与经济的指示物组成时。在另一方面,基于 GIS 的空间聚类是最近的年里的一个经常的焦点。在瓷器都市化和工业化的上下文以内在人的解决和理论系统上开创研究将在中国城市和区域的可持续性上提供指导。下列五个方面要求更大的注意:(1 ) 关于人的解决的自然适用性研究,和人的解决的调查要求在城市的环境,相应估价指示物,系统,和进化有关生态地合适的解决反映不同要求的范围,并且居民的影响社会经济的属性。(2 ) 空间时间的评估和可持续性在各种各样的规模在城市、农村的人的解决上研究,集中于在象在城市之间的城市簇和比较那样的各种各样的规模的进化和空间区别,在城市和社区以内。(3 ) 为人的解决进化研究的理论和技术的开发包括察觉技术和方法,数据采矿测量,并且预报并且地区性、城市的人的解决进化进程,机制和模式的模拟。(4 ) 在为有效管理和开发集中于优化,模式,和政策的人的解决的控制上研究。(5 ) 估计人的解决系统服务值并且建立合适的人的解决系统,包括社会、经济、文化、生态的服务值。 | MA Renfeng WANG Tengfei ZHANG wenzhong YU Jianhui WANG Dai CHEN Li JIANG Yanpeng FENG Gequn | 2016 | Journal of Geographical Sciences2016，26，8： | 6 |
| 2 | Effect of cyclovirobuxine D on human growth-associated protein 43 and neurocan expression in ischemic brain tissue of stroke-prone renovascular hypertensive rats显示文摘BACKGROUND: Experimental data indicate that human growth-associated protein 43 mRNA expression coincides with axonal growth during nerve ganglion development; while neurocan, secreted from astrocytes, can inhibit sprouting and elongation of the axonal growth cone. OBJECTIVE: To verify regulatory effects of cyclovirobuxine D (CVB-D) extracted from Chinese box branchlet on human growth-associated protein 43 (GAP-43), and neurocan expression in brain tissue of stroke-prone renovascular hypertensive (RHRSP) rats, at different time points after cerebral ische-mia/reperfusion. DESIGN: Randomized grouping design and controlled animal study. SETTING: This study was performed at the Center of Guangdong Hospital of Traditional Chinese Medicine (a national key laboratory) from March 2003 to September 2006. MATERIALS: 100 healthy male Sprague-Dawley rats, aged 2-3 months and weighing 90-120 g, were se- lected for this study. CVB-D was provided by Nanjing Xiaoying Pharmaceutical Factory (Batch number: 307701). METHODS: The initial tip of renal arteries was clamped bilaterally for 10 weeks to establish the RHRSP model. 100 RHRSP rats were randomly divided into 4 groups: naive group (n = 10), sham surgery group (n = 10), CVB-D group (n = 40), and lesion group (n = 40). Rats in the naive group did not undergo any treatment, and cervical vessels of rats in the sham surgery group were exposed, but not blocked. The right middle cerebral artery of rats in the CVB-D group and lesion group were occluded to establish cerebral ischemia. Rats in the CVB-D group were intraperitoneally injected with CVB-D (6.48 mg/kg) every day and with saline (1.5 mL/injection) twice a day. Rats in the lesion group were intraperitoneally injected with saline (2 mL/injection). MAIN OUTCOME MEASURES: Immunohistochemistry was applied to detect GAP-43 and neurocan expression in the ischemic penumbra region of CVB-D and lesion brains at 2 hours post-cerebral ischemia and at 1, 7, 14, and 30 days post-perfusion (n = 10 at each time point). Similarly, GAP-43 and neurocan expres- sion was detected in the right hemisphere of na?ve and sham-operated animals. The results were expressed as positive cells. RESULTS: A total of 100 rats were included in the final analysis. The number of GAP-43 positive cells in- creased in the CVB-D group 1, 7, 14, and 30 days post-cerebral ischemia/perfusion compared to the lesion group, as indicated by a significant difference between the CVB-D and lesion group (P < 0.05-0.01). The number of neurocan-positive cells decreased in the CVB-D group on the first day compared to the model group; however, there was no significant difference between the two groups (P > 0.05). On post-ischemia days 7, 14, and 30, the number of neurocan-positive cells in the CVB-D group was significantly less than in the lesion group (P < 0.05). Both, GAP-43 and neurocan expression was not detectable in brains of na?ve and sham-operated animals. CONCLUSION: CVB-D treatment up-regulated GAP-43 expression and down-regulated neurocan expression in the ischemic region of RHRSP rats. | Feng Tan Wei Gu Saiying Wan Haike Wu Jinliang Wang Jingbo Sun Jiamao Cheng Xin Zhang Renfeng Huang | 2008 | Neural Regeneration Research2008，3，4： | 1 |
| 3 | Evidence-based medicine (Translate)显示文摘 | RENFENG FENG YIXUN TAO | 2002 | Shanghai Journal of Medical Science2002，17，： | 1 |
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