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15篇 您的检索式:作者名="Pruschy"
    题名 作者 年代 出处 被引量
1Apoptosis modulating agents in combination with radiotherapy current status andoutlook 显示文摘Belka C Jendrossek V Pruschy M 2004Int J Radiat Oncol BM Phys2004,58,2:1
2Key targets for the execution of radiation-induced tumor cell apoptosis: the role of p53 and caspases显示文摘Pruschy M Rocha S Zaugg K 2001Int J Radiat Oncol Biol Phys2001,49,2:1
3Metabolism of tumors under treatment: Mapping of metabolites with quantitative bioluminescence显示文摘Broggini-Tenzer A Vuong V Pruschy M 2011Radiother Oncol2011,99,:1
4PKC412-a protein kinase inhibitor with a broad therapeutic potential显示文摘Fabbro D Ruetz S Bodis S Pruschy M Csermak K Man A 2000Anticancer Drug Des2000,15,1:1
5Regulation of VEGF-expression by patupilone and ionizing radiation in lung adenocarcinoma cells显示文摘Carla Rohrer Bley Katrin Orlowski Polina Furmanova Paul M.J. McSheehy Martin Pruschy 2011Lung Cancer2011,,3:1
6Apopsis-modulating agents in combination with radiotherapy-current status and outlook显示文摘Belka C Jendrossek V Pruschy M 2004Int J Radiat Oncol Biol Phys2004,58,2:1
7Potentiation of DNA-damage-induced cytotoxicity by G2 checkpoint abrogators 显示文摘Tenzer A Pruschy M 2003Curr Med Chem Anti-Cancer Agents2003,3,1:1
8Apopsis modulating agents in combination with radiotherapy current status and outlook显示文摘Belka C Jendrossek V Pruschy M 0,,02:1
9Key targets for the execution of radiation-induced tumor cell apoptosis: the role of p53and caspases显示文摘PRUSCHY M ROCHA S ZAUGG K 2001Int J Radiat Oncol Biol Phys2001,49,2:1
10Key targets for the execution of radiation-induced tumor cell apoptosis: the role of p53 and caspases 显示文摘 Rocha S Zaugg K Tenzer A Hess C Fisher DE Glanzmann C Bodis S 2001Int J Radiat Oncol Biol Phys2001,49,2:1
11MVP and vauhs:a role in the radiation response显示文摘Lara PC Pruschy M Zimmermann M 2011Radiat Oncol2011,6,:1
12Apoptosis-modulating agents in combination with radiotherapy-current status and outlook显示文摘Belka C Jendrossek V Pruschy M 2004Int J Radiat Oncol Biol Phys2004,58,2:1
13Apopsis modulating agents in combination with radiotherapy current status and outlook显示文摘Belka C Jendrossek V Pruschy M 2004Int J Radiat Oncol Biol Phys2004,58,2:1
14Potentiation of DNA-damage-induced cytotoxicity by G2 checkpoint abrogators 显示文摘Tenzer A Pruschy M 2003Curt Med Chem Anti-Canc Agents2003,3,1:1
15Impact of small molecule-mediated inhibition of ammonia detoxification on lung malignancies and liver metabolism显示文摘Dear Editor,Metabolically induced cancer heterogeneity creates a large source of novel potential targets towards an enhanced therapeutic window alone and in combination with classic chemo-and radiotherapy[1,2].This is of particular interest for non-small cell lung cancer(NSCLC),which accounts for more than 80%of all lung tumor types characterized by limited responses to current treatment options[3–5].Georgios Makris Semih Kayhan Marvin Kreuzer Véronique Rüfenacht Erica Faccin Jarl Underhaug Carmen Diez-Fernandez Philip A.Knobel Martin Poms Nadine Gougeard Vicente Rubio Aurora Martinez Martin Pruschy Johannes Häberle 2023Cancer Communications2023,43,4:0
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