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| 1 | Prognostic impact of body mass index on in-hospital bleeding complications after ST-segment elevation myocardial infarction显示文摘BACKGROUND ST-elevation myocardial infarction(STEMI)remains a major cause of mortality despite early revascularization and optimal medical therapy.Tailoring individual management by considering patients’specificities may help in improving post-STEMI survival.AIM To evaluate whether in-hospital bleeding complications may be involved in post STEMI prognosis among overweight patients.METHODS We prospectively included 2070 patients with a STEMI between January 2005 and December 2012 in the French observational cohort,“Registre d’Infarctus Maine-Anjou”.Bleeding Academic Research Consortium(BARC)in-hospital bleeding complications were recorded.RESULTS Of 705 patients(35.3%)were presented as being of normal weight,defined as a body mass index(BMI)<25 kg/m^2,877(43.9%)had a BMI between 25 and 30 kg/m^2and 416(20.8%)had a BMI≥30 kg/m^2.One-year cardiovascular mortality was lower for BMI≥25 kg/m^2(5.3%and 7.1%)patients than for normal weight patients(10.8%)(P=0.001).We found an interaction between the effect of BARC 3 on mortality and BMI groups.While a BARC 3 was related to a higher 1-year mortality in general(HR:2.58,95%CI:1.44-4.64,P≤0.001),prognosis was even worse in normal weight patients(HR:2.97,95%CI:1.61-5.5,P<0.001)than for patients with a BMI≥25 kg/m^2(HR:1.94,95%CI:1.02-3.69,P=0.041).CONCLUSION Normal weight patients presented higher rates of in-hospital bleeding complications and lower survival after a STEMI.Excess mortality might be due to greater vulnerability to bleeding amongst normal weight patients. | Delphine Ingremeau Sylvain Grall Florine Valliet Laurent Desprets Fabrice Prunier Alain Furber Loïc Bière | 2020 | World Journal of Cardiology2020，12，1： | 2 |
| 2 | The oncoprotein Ski acts as an antagonist of transforming gowth factor-beta signaling by suppressing Smad 2 phosphorylation显示文摘 | Prunier C Pessah M Ferrand N | 2003 | J Biol Chem2003，278，26： | 1 |
| 3 | c-Jun inhibits transforming growth factor beta-mediated transcription by repressing Smad 3 transcriptional activity显示文摘 | Dennler S Prunier C Ferrand N | | 0，，： | 1 |
| 4 | Mechanism for mutational inactivation of the tumor suppressor Sma2显示文摘 | Prunier C Ferrand N Frottier B | 2001 | Mol Cell Biol2001，21，10： | 1 |
| 5 | The oncoprotein e-ski functions as a direct antagonist of the transtorming growth factor-{heta} type I receptor 显示文摘 | Ferrand N Atfi A Prunier C | 2010 | Cancer Res2010，70，21： | 1 |
| 6 | Mechanism for mutational inactivation of the tumor suppressor Smad2显示文摘 | Prunier C Ferran3 N Frottier B | 2001 | Mol Cell Biol2001，21，10： | 1 |
| 7 | Quantitative analysis of striatal dopamine D2 receptors with ^123I-iodo-2 isuride SPECT in degenerative extra1pyramidal diseases显示文摘 | Prunier C Tranquart F Cottier JP | 2001 | Nucl Med Commun2001，22，： | 1 |
| 8 | Wnt signaling: physiology and pathology显示文摘 | Prunier C Hocevar B A Howe P H | 2004 | Growth Factors2004，22，3： | 1 |
| 9 | Quantitative analysis of striatal dopamine D2 receptors with 123 I-iodolisuride SPECT in degenerative extrapyramidal diseases显示文摘 | Prunier C Tranquart F Cottier P | 2001 | Nucl Med Commun2001，22，11： | 1 |
| 10 | c-Jun inhibits transforming growth factor beta-mediated transcription by repressing Smad3 transcriptional activity显示文摘 | Dennler S Prunier C Ferrand N | 2000 | J Biol Chem2000，275，28： | 1 |
| 11 | Mechanism for mutational in activation of the tumor suppressor Smad2 显示文摘 | Prunier C Ferrand N Frottier B | 2001 | Mol Cell Biol2001，21，10： | 1 |
| 12 | Quantitative analysis of striatal dopamine D2 receptors with^123I - iodolisuride SPECT in degenerative extrapyramidal disease显示文摘 | Prunier C Tranquart F Cottier P | 2001 | Nuel Med Commun2001，22，12： | 1 |
| 13 | Behavioural patterns and progesterone,cortisol and prolactin levels around parturition in European(LargeWhite)and Chinese(Meishan)sows显示文摘 | MEUNIER-SALA譈N M C GORT F PRUNIER A | 1991 | Appl Anim Behav Sci1991，31，12： | 1 |
| 14 | High ambient temperatures: How they affect sow lactation performance 显示文摘 | Earner C Prunier A | 2002 | Pig News and Information2002，23，4： | 1 |
| 15 | A comparison be- tween physical properties of carbon black-polymer and carbon nanotubes显示文摘 | Adohi B J P Mdarhri A Prunier C | 2010 | Polymer Composites J Appl Phys2010，108，7： | 1 |
| 16 | The RIPOST-MI study, assessing remote ischemic perconditioning alone or in com- bination with local ischemic postconditioning in ST-segment eleva- tion myocardial infarction 显示文摘 | Prunier F Angoulvant D Saint Etienne C | 2014 | Basic Res Cardiol2014，109，2： | 1 |
| 17 | Disabled-2 (Dab2) mediates transforming growth factor beta (TGFbeta)-stimulated fibronectin synthesis through TGFbeta-activated kinase 1 and activation of the JNK pathway显示文摘 | Hocevar BA Prunier C Howe PH | 2005 | J Biol Chem2005，80，25： | 1 |
| 18 | Wnt signaling: physiology and pathology 显示文摘 | Prunier C Hocervar BA Howe PH | 2004 | Growth Factors2004，22，3： | 1 |
| 19 | The oncoprotein c-Ski functions as a direct antagonist of the transforming growth factor-β type I receptor显示文摘 | Ferrand N Atfi A Prunier C | 2010 | Cancer Res2010，70，21： | 1 |
| 20 | Quantification of dopamine transporter by 123I-PE2I SPECT and the noninvasive Logan graphical method in Parkinson's disease显示文摘 | Prunier C Payoux P Guilloteau D | 2003 | J Nucl Med2003，44，5： | 1 |
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