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| 1 | Risk of ventricular arrhythmia in patients with myocardial infarction and non-obstructive coronary arteries and normal ejection fraction显示文摘AIM To assess the arrhythmic determinants and prognosis of patients presenting with myocardial infarction and nonobstructive coronary arteries(MINOCA)with normal ejection fraction(EF).METHODS This is an observational analysis of 131 MINOCA patients with normal EF.Three cardiac magnetic resonance(CMR)diagnosis classes were recognized according to the late gadolinium enhancement(LGE)pattern:Myocardial infarction(MI)(n=34),myocarditis(n=47),and'no LGE'(n=50).Ventricular events occurring during hospitalization were recorded and the entire population was followed-up at 1 year.RESULTS Ventricular arrhythmia was observed in 18(13.8%)patients during hospitalization.The'no LGE'patients experienced fewer ventricular events than the MI and myocarditis patients[4.0%vs 26.5%and 14.9%,respectively(P=0.013)].There was no significant difference between the MI and myocarditis groups.On multivariate analysis,LGE transmural extent[OR=1.52(1.08-2.15),P=0.017]and ST-segment elevation[OR=4.65(1.61-13.40),P=0.004]were independent predictors of ventricular arrhythmic events,irrespective of the diagnosis class.Finally,no patient experienced sudden cardiac death or ventricular arrhythmia recurrence at 1-year.CONCLUSION MINOCA patients with normal EF presented no 1-year cardiovascular events,irrespective of the CMR diagnosis class.LGE transmural extent and ST segment elevation at admission are risk markers of ventricular arrhythmia during hospitalization. | Loic Bière Marjorie Niro Hervé Pouliquen Jean-Baptiste Gourraud Fabrice Prunier Alain Furber Vincent Probst | 2017 | World Journal of Cardiology2017，9，3： | 3 |
| 2 | Prognostic impact of body mass index on in-hospital bleeding complications after ST-segment elevation myocardial infarction显示文摘BACKGROUND ST-elevation myocardial infarction(STEMI)remains a major cause of mortality despite early revascularization and optimal medical therapy.Tailoring individual management by considering patients’specificities may help in improving post-STEMI survival.AIM To evaluate whether in-hospital bleeding complications may be involved in post STEMI prognosis among overweight patients.METHODS We prospectively included 2070 patients with a STEMI between January 2005 and December 2012 in the French observational cohort,“Registre d’Infarctus Maine-Anjou”.Bleeding Academic Research Consortium(BARC)in-hospital bleeding complications were recorded.RESULTS Of 705 patients(35.3%)were presented as being of normal weight,defined as a body mass index(BMI)<25 kg/m^2,877(43.9%)had a BMI between 25 and 30 kg/m^2and 416(20.8%)had a BMI≥30 kg/m^2.One-year cardiovascular mortality was lower for BMI≥25 kg/m^2(5.3%and 7.1%)patients than for normal weight patients(10.8%)(P=0.001).We found an interaction between the effect of BARC 3 on mortality and BMI groups.While a BARC 3 was related to a higher 1-year mortality in general(HR:2.58,95%CI:1.44-4.64,P≤0.001),prognosis was even worse in normal weight patients(HR:2.97,95%CI:1.61-5.5,P<0.001)than for patients with a BMI≥25 kg/m^2(HR:1.94,95%CI:1.02-3.69,P=0.041).CONCLUSION Normal weight patients presented higher rates of in-hospital bleeding complications and lower survival after a STEMI.Excess mortality might be due to greater vulnerability to bleeding amongst normal weight patients. | Delphine Ingremeau Sylvain Grall Florine Valliet Laurent Desprets Fabrice Prunier Alain Furber Loïc Bière | 2020 | World Journal of Cardiology2020，12，1： | 2 |
| 3 | Delayed erythropoietin therapy reduces post-MI cardiac remodeling only at a dose that mobilizes endothelial progenitor cells 显示文摘 | Prunier Fabrice Pfister Otmar Hadri Lahouria | 2007 | Am J Physiol Heart Circ Physiol2007，292，1： | 1 |
| 4 | Mechanism for mutational inactivation of the tumor suppressor Smad2显示文摘 | Prunier G Ferrand N Frottier B | 2001 | Mol Cell Biol2001，21，： | 1 |
| 5 | The oncoprotein Ski acts as an antagonist of transforming gowth factor-beta signaling by suppressing Smad 2 phosphorylation显示文摘 | Prunier C Pessah M Ferrand N | 2003 | J Biol Chem2003，278，26： | 1 |
| 6 | Measure-menLs of rolordynamic forces on an artificial heart pvimpimpeller显示文摘 | Suzuki T Prunieres R Horiguchi H | 2007 | Journal of Fluids Engineering2007，129，11： | 1 |
| 7 | Coronary blood flow assessment after successful angioplasty for acute myocardial infarction predicts the risk of long I term cardiac events 显示文摘 | Furber AP Prunier F Nguyen HC | 2004 | Circulation2004，110，23： | 1 |
| 8 | Livest Prod Sci 显示文摘 | Dourmad J Y Etienne M Prunier A | 1994 | 40 : 87-971994，40，： | 1 |
| 9 | c-Jun inhibits transforming growth factor beta-mediated transcription by repressing Smad 3 transcriptional activity显示文摘 | Dennler S Prunier C Ferrand N | | 0，，： | 1 |
| 10 | Health, welfare and production problems in organic suckling piglets显示文摘 | PRUNIER A LUBAC S MEJER H | 2014 | Or- ganic Agriculture2014，4，2： | 1 |
| 11 | Coronary blood flow assessment after successful angioplasty for acute myocardial infarction predicts the risk of long-term cardiac events 显示文摘 | Furber AP Prunier F Nguyen HC | 2004 | Circulation2004，110，23： | 1 |
| 12 | Mechanism for mutational inactivation of the tumor suppressor Sma2显示文摘 | Prunier C Ferrand N Frottier B | 2001 | Mol Cell Biol2001，21，10： | 1 |
| 13 | The oncoprotein e-ski functions as a direct antagonist of the transtorming growth factor-{heta} type I receptor 显示文摘 | Ferrand N Atfi A Prunier C | 2010 | Cancer Res2010，70，21： | 1 |
| 14 | Mechanism for mutational inactivation of the tumor suppressor Smad2显示文摘 | Prunier C Ferran3 N Frottier B | 2001 | Mol Cell Biol2001，21，10： | 1 |
| 15 | Delayed erythropoietin therapy reduces Post-MI cardiac remodeling only ate dose that mobilizes endothelial progenitor cells显示文摘 | Prunier F Pfister O Hadri L | 2007 | Am J Physiol Heart Circ Physio2007，292，1： | 1 |
| 16 | Prevention of ventricular arrhythmias with sarcoplasmic reticulum Ca2 + ATPase pump overexpression in a porcine model of ischemia reperfusion显示文摘 | Prunier F Kawase Y Gianni D | 2008 | Circulation2008，118，： | 1 |
| 17 | Delayed erythropoietin therapy reduces post-MI cardiac remodeling only at a dose that mobilizes endothelial progenitor cells显示文摘 | Prunier F Pfister O Hadri L | 2007 | Am J Physiol Heart Circ Physiol2007，292，1： | 1 |
| 18 | Quantitative analysis of striatal dopamine D2 receptors with ^123I-iodo-2 isuride SPECT in degenerative extra1pyramidal diseases显示文摘 | Prunier C Tranquart F Cottier JP | 2001 | Nucl Med Commun2001，22，： | 1 |
| 19 | Influence of high ambient temperature on performance of reproductive sows显示文摘 | PRUNIER A DE BRAGAN A M M LE DIVIDICH J | 1997 | Livestock Production Science1997，52，2： | 1 |
| 20 | Wnt signaling: physiology and pathology显示文摘 | Prunier C Hocevar B A Howe P H | 2004 | Growth Factors2004，22，3： | 1 |
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