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41篇 您的检索式:作者名="Prodam"
    题名 作者 年代 出处 被引量
1GH therapy in adult GH deficieney:a review of treatment schedules and the evidence for low starting doses 显示文摘Gaseo V Prodam F Grottoli S 2013Eur J Endocrinol2013,168,3:1
2Hypopituitarism following brair injury:when does it occur and how best to test? 显示文摘Gasco V Prodam F Pagano L 2012Pituitary2012,15,1:1
3Ghrelin: a link between eating disorders, obesity and reproduction显示文摘Gottero C Broglio F Prodam F 2004Nutr Neurosci2004,7,56:1
4Ghrelin: from somatotrope secretion to new perspectives in the regulation of peripheral metabolic functions 显示文摘Broglio F Prodam F Riganti F 2006Front Horm Res2006,35,:1
5Ghrelin: endocrine,metabolic and cardiovascular actions显示文摘Broglio F Prodam F Me E 2005J Endocrinol Invest2005,28,5:1
6Quality of life,mood disturbances and psychological parameters in adult pa-tients with GH deficiency显示文摘Prodam F Caputo M Belcastro S 2012Panminerva Med2012,54,4:1
7GH therapy in adult GH deficiency:a review of treatment schedules and the evidence for low starting doses显示文摘GASCO V PRODAM F GROTTOLI S 2013Eur J Endocrinol2013,168,3:1
8Ghrelin:a link between eating disorders,obesity and reproduction显示文摘 Broglio F Prodam F 2004Nutr Neurosci2004,7,56:1
9Obesity modifies expression profiles of metabolic markers in superficial and deep subcutaneous abdominal adipose tissue depots显示文摘Walker GE Marzullo P Prodam F 2014Endocrine2014,46,1:1
10Update on epidemiology,etiology,and diagnosis of adult growth hormone deficiency显示文摘Prodam F Pagano L Corneli G 2008JEndocrinol Invest2008,31,9:1
11Ghrelin:from somatotrope secretion to new perspectives in the regulation of peripheral metabolic functions显示文摘Broglio F Prodam F Riganti F 2006Front Horm Res2006,35,:1
12Hypopituitarism followingbrain injury : when does it occur and how best to test 显示文摘Gasco V Prodam F Pagano L 2012Pituita-ry2012,15,1:1
13GH therapy in adult GHdeficiency : a review of treatment schedules and the evidence forlow starting doses 显示文摘Gasco V Prodam F Grottoli S 2013Eur J Endocrinol2013,168,3:1
14Obesity modifies expression profiles of metabolic markers in superficial and deep subcutaneous ab- dominal adipose tissue depots 显示文摘Walker GE Marzulo P Prodam F 2014Endocrine2014,46,1:1
15Hypopituitarism following brain injury: when does it occur and how best to test? 显示文摘Gasco V Prodam F Pagano L 2012Pituitary2012,15,1:1
16Heterozygous mutation of HESX1 causing hypopituitarism and multiple anatomical malformations without features of septo-optic dysplasia 显示文摘Corneli G Vivenza D Prodam F 2008J Endocrinol Invest2008,31,8:1
17Effects of growth hormone(GH) therapy withdrawal on glucose metabolism in not confirmedGH deficient adolescents at final height 显示文摘Prodam F Savastio S Genoni G 2014PLoS One2014,,87:1
18Ghrelin:from somatotrope secretion to new perspectives in the regulation of peripheral metabolic functions显示文摘Broglio F Prodam F Riganti F 2006Front Horm Res2006,35,2:1
19GH therapy in adult GH de ciency:A review of treatment schedules and the evidence for low starting doses显示文摘VALENTINA GASCO FLAVIA PRODAM SILVIA GROT- TOLI 2013Eur J Endocrinol2013,168,3:1
20Diet as a strategy for type 1 diabetes prevention显示文摘The increasing incidence of type 1 diabetes mellitus(T1DM)in children represents one of the major worldwide health concerns.It is widely thought that T1DM is caused by the autoimmune destruction of insulin-producingβcells in the pancreas due to an aberrant T-cell immune response.To date,there are no effective therapeutic strategies to prevent T1DM,as its clinical symptoms become apparent only when mostβcells have already been destroyed.Furthermore,clinical trials for the prevention of T1DM in children at risk have all been unsuccessful to date.Intriguingly,recent studies have implicated alterations in the gut microbial flora(that is,microbiota)as a potential etiological factor for T1DM,raising the possibility that interfering with the microbes populating our body could represent a promising approach to prevent or manage T1DM.New evidence strongly supporting this hypothesis comes from a milestone study by Mariño et al.,1 which shows that a diet capable of modulating the activity of the gut microbiota can indeed inhibit the disease onset in a mouse model of T1DM.Overall,these findings pave the way for the development of food-based dietary approaches for preventing high-risk children from developing T1DM.Flavia Prodam Annalisa Chiocchetti Umberto Dianzani 2018Cellular & Molecular Immunology2018,15,1:1
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