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24篇 您的检索式:作者名="Poussin MA"
    题名 作者 年代 出处 被引量
1B7-1 costimulatory molecule is critical for the development of experimental autoimmune myasthenia gravis显示文摘Poussin MA Tuzun E Goluszko E 2003J Immunol2003,170,:1
2Suppressed clinical experimental autoimmune myasthenia gravis in bm12 mice is linked to reduced intracellular calcium mobilization and IL-10 and IFN-gamma release by acetylcholine receptor-specific T cells显示文摘Poussin MA Fuller CL Goluszko E 2003J Neuroimmunol2003,134,12:1
3Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis 显示文摘Goluszko E Deng C Poussin MA 2002J Neuroimmunol2002,122,12:1
4Suppression of experimental autoimmune myasthenia gravis in IL-IO gene- disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR 显示文摘Poussin MA Goluszko E Hughes TK 2000J Neuroimmunol2000,111,12:1
5Suppressed clinical experimental autoimmune myasthenia gravis in bin12 mice is linked to reduced intracellular calcium mobilization and IL-10 and IFN-gamma release by acetylcholine receptor specific T cells 显示文摘Poussin MA Fuller CL Goluszko E 2003J Neuroimmunol2003,134,12:1
6Role of IL-5 during primary and secondary immune response to acetylcholine receptor显示文摘Poussin MA Goluszko E Franco JU 2002J Neuroimmunol2002,125,:1
7Role of IL-5during primary and secondary immune response to acetylcholine receptor显示文摘Poussin MA Goluszko E Franco JU 2002J Neuroimmunol2002,125,12:1
8B7-1 costimulatory molecule is critical for the development of experimental auto-immune myasthenia gravis显示文摘Poussin MA Tuzun E Goluszko E 2003170(8):43892003,170,8:1
9Role of IL-5 during primary and secondary immune response to acetyl choline receptor 显示文摘Poussin MA G01uszko E Franco JU 2002J Neuroimmunol2002,125,12:1
10B7-1 costimulatory molecule is critical for the development of experimental auto-immune myasthenia gravis显示文摘Poussin MA Tuzun E Goluszko E 2003J Immunol2003,170,8:1
11B7-1 eostimulatory molecule is criticalfor the development of experimental autoimmune myasthenia gravis显示文摘Poussin MA Tuzun E Goluszko E 2003Immunology2003,170,:1
12Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis显示文摘Goluszko E Deng C Poussin MA 2002J Neuroimmunol2002,122,:1
13Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR显示文摘Poussin MA Goluszko E Hughes TK 2000J Neuroimmunol2000,111,12:1
14Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis 显示文摘 Deng C Poussin MA 2002J Neuroimmunol2002,122,1:1
15Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR显示文摘Poussin MA Goluszko E Hughes TK 2000J Neuroimmunol2000,111,:1
16Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis显示文摘Goluszko E Deng C Poussin MA 2002J Neuroimmunol2002,122,12:1
17Suppressed clinical experimental autoimmune myasthenia gravis in bml2 mice is linked to reduced intracellular calcium mobilization and IL-10 and IFN-gamma release by acetylcholine receptor-specific T cells 显示文摘Poussin MA Fuller CL Goluszko E 2003Neuroimmunol2003,134,12:1
18B7-1 costimulatory molecule is critical for the development of experimental autoimmune myasthenia gravis显示文摘Poussin MA Tuzun E Goluszko E 2003J Immunol2003,170,:1
19Role of IL-5 during primary and secondary immune response to acetylcholine receptor显示文摘Poussin MA Goluszko E Franco JU 2002J Neuroimmunol2002,125,12:1
20Role of IL-5 during primary and secondary immune response to acetylcholine receptor显示文摘Poussin MA Goluszko E Franco JU 2002J Neuroimmunol2002,125,12:1
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