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| 1 | Transfer Learning for Pedestrian Etection 显示文摘 | CAO Xianbin WANG Zhong YAN Pingkun | 2013 | Neurocomputing2013，100，： | 1 |
| 2 | Segmentation of volumetric MRA image by using capillary active contour 显示文摘 | Yan Pingkun Kassim A | 2006 | Medical Image Analysis2006，10，3： | 1 |
| 3 | Transfer learning for pedestrian detection显示文摘 | Cao Xianbin Wang Zhong Yan Pingkun | 2013 | Neurocomputing2013，100，1： | 1 |
| 4 | Discrete deformable model guided by partial active shape model tbr TRUS image segmentation 显示文摘 | Pingkun Yan Sheng Xu Baris Turbey etc | 2010 | IEEE Transactions on Biomedical Engineering2010，57，5： | 1 |
| 5 | Net-FLICS:fast quantitative wide-field fluorescence lifetime imaging with compressed sensing–a deep learning approach显示文摘Macroscopic fluorescence lifetime imaging(MFLI)via compressed sensed(CS)measurements enables efficient and accurate quantification of molecular interactions in vivo over a large field of view(FOV).However,the current dataprocessing workflow is slow,complex and performs poorly under photon-starved conditions.In this paper,we propose Net-FLICS,a novel image reconstruction method based on a convolutional neural network(CNN),to directly reconstruct the intensity and lifetime images from raw time-resolved CS data.By carefully designing a large simulated dataset,Net-FLICS is successfully trained and achieves outstanding reconstruction performance on both in vitro and in vivo experimental data and even superior results at low photon count levels for lifetime quantification. | Ruoyang Yao Marien Ochoa Pingkun Yan Xavier Intes | 2019 | Light(Science & Applications)2019，8，1： | 1 |
| 6 | Single-image super- resolution via local learning 显示文摘 | Tang Yi Yan Pingkun Li Xuelong | 2011 | International Journal of Machine Learning and Cybernetics2011，2，1： | 1 |
| 7 | Segmentation of volumetric MRA images by using capillary active contour 显示文摘 | Yan Pingkun Kassim AA | 2006 | Medical Image Analysis2006，10，3： | 1 |
| 8 | Vehi- cle Detection and Tracking in Airborne Videos by Multi-motion Layer Analysis显示文摘 | Cao Xianbin Lan Jinhe Yan Pingkun | 2011 | Machine Vision and Applications2011，3，： | 1 |
| 9 | Transfer learning for pedestrian detection显示文摘 | Cao Xianbin Wang Zhong Yan Pingkun | 2013 | Neurocomputing2013，100，： | 1 |
| 10 | Multi-functions of exonuclease 1 in DNA damage response and cancer susceptibility显示文摘Exonuclease 1(EXO1)can catalyze nucleotide chain excision with its conserved N-terminal domain of 5′ to 3′ exonuclease activity,enabling it to influence diverse biological processes facing the challenges of genotoxic environmental factors such as ionizing radiation.This nuclease activity enables EXO1 to maintain replication forks and telomeres length,to facilitate post-replication DNA repair and to process the end resection step of homologous recombination of DNA double-strand breaks-induced by ionizing radiation.When DNA replication is disrupted or blocked,EXO1 can cleave the broken DNA ends to form 3’ssDNA,leading to repair pathways activation.Excess EXO1-mediated nucleotide excision,however,can introduce an abundance of single-stranded DNA that can cause mutation and recombination via micro-homology-mediated end joining or single-strand annealing mechanisms,contributing to a loss of genetic information.EXO1 activity must therefore be carefully regulated within healthy cells.The mutations and dysregulations of EXO1 can increase the sensitivity of cells to radiation injury and risk of oncogenic transformation,limit the adoption of specific treatments in a range of human diseases.As such,EXO1 represents a promising target for the treatment and prevention of cancer.In the present review,we delineate the structural properties and functional characteristics of EXO1,discuss the relationship between this exonuclease and cancer susceptibility as well as the second cancers related to radiotherapy. | Shuang Yan Shanshan Gao Pingkun Zhou | 2021 | Radiation Medicine and Protection2021，2，4： | 0 |
| 11 | Ubiquitination and degradation of SIK2 by DNA-PKcs deficiency promote radiation-induced mitotic catastrophe显示文摘Salt-inducible kinase 2 (SIK2) is a member of the AMP-activated serine/threonine kinase family. It has been reported that inhibition of SIK2 can enhance the cytotoxicity of paclitaxel,1 promote premitotic apoptosis, and lead to cell cycle arrest in the metaphase.2 Thus, targeting SIK2 may be a therapeutic strategy for cancers drug and radiotherapy resistance. Mitotic catastrophe is a type of abnormal mitosis leading to cell death characterized by the multipolar spindle and multinucleation, which was first discovered during an ionizing radiation (IR)-induced cell damage.3 However, the mechanism of mitotic catastrophe is not well understood. The present study aimed to assess the effect of the knockdown of SIK2 on IR-induced mitotic catastrophe. | Jiaojiao Zhu Ying Zhang Ziyan Yan Jianxiao Wang Ping Wang Xinxin Liang Yuhao Liu Xingkun Ao Maoxiang Zhu Pingkun Zhou Yongqing Gu | 2023 | Genes & Diseases2023，10，2： | 0 |
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